His scientific interests lie mostly in Immunology, Molecular biology, FOXP3, Signal transduction and Immune tolerance. His Immunology research focuses on CTLA-4, Immune dysregulation, Regulatory T cell, Interleukin 4 and Primary immunodeficiency. The study incorporates disciplines such as Transcription factor, Mutant, Cell adhesion, Cell adhesion molecule and Kinase in addition to Molecular biology.
His FOXP3 research incorporates themes from IL-2 receptor, Toll-like receptor and Interleukin 10. Talal A. Chatila interconnects Ionomycin, Receptor, Viral replication, Interleukin-4 receptor and Phosphorylation in the investigation of issues within Signal transduction. His Immune tolerance research is multidisciplinary, incorporating elements of Adoptive cell transfer and Immunotherapy.
His primary scientific interests are in Immunology, FOXP3, Cell biology, Molecular biology and Immune system. His Food allergy, Immune tolerance, Autoimmunity, Regulatory T cell and DOCK8 Deficiency investigations are all subjects of Immunology research. The various areas that Talal A. Chatila examines in his FOXP3 study include Cancer research, Toll-like receptor, IL-2 receptor, Immune dysregulation and Immunotherapy.
His studies in Cell biology integrate themes in fields like T cell, Biochemistry and CD3. His study in Molecular biology is interdisciplinary in nature, drawing from both Nuclear protein, Transcription factor, Lymphokine, Interleukin 2 and Kinase. His Signal transduction study integrates concerns from other disciplines, such as Receptor and Ionomycin.
His primary areas of study are Immunology, Regulatory T cell, Food allergy, Immune system and Transcription factor. His study in Immunology concentrates on Asthma, Inflammation, Autoimmunity, Antibody and Innate lymphoid cell. As a part of the same scientific family, Talal A. Chatila mostly works in the field of Food allergy, focusing on Microbiome and, on occasion, Disease.
His work on Immune dysregulation, Immune tolerance, Treg cell and Antigen specific as part of general Immune system research is often related to Open peer review, thus linking different fields of science. Talal A. Chatila has included themes like Reprogramming, Signal transduction, Cell biology and FOXP3 in his Transcription factor study. His FOXP3 research includes themes of Cell and Cancer research.
Talal A. Chatila mainly focuses on Immunology, Transcription factor, Immune dysregulation, FOXP3 and Food allergy. His Immunology research includes elements of Receptor, Blockade and Phenocopy. As part of the same scientific family, Talal A. Chatila usually focuses on Transcription factor, concentrating on Signal transduction and intersecting with Regulation of gene expression and FOXO1.
Talal A. Chatila works mostly in the field of Immune dysregulation, limiting it down to concerns involving Antigen and, occasionally, Cellular differentiation. His Food allergy research is multidisciplinary, incorporating perspectives in Regulatory T cell, RAR-related orphan receptor gamma, Asthma and Bacteroides. When carried out as part of a general Immune system research project, his work on Interleukin 2 and Immune tolerance is frequently linked to work in Context, therefore connecting diverse disciplines of study.
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The Toll-Like Receptor 2 Pathway Establishes Colonization by a Commensal of the Human Microbiota
June L. Round;S. Melanie Lee;Jennifer Li;Gloria Tran.
Science (2011)
JM2, encoding a fork head–related protein, is mutated in X-linked autoimmunity–allergic disregulation syndrome
Talal A. Chatila;Frank Blaeser;Nga Ho;Howard M. Lederman.
Journal of Clinical Investigation (2000)
The association of atopy with a gain-of-function mutation in the alpha subunit of the interleukin-4 receptor.
Gurjit K. Khurana Hershey;Michal F. Friedrich;Laura A. Esswein;Matthew L. Thomas.
The New England Journal of Medicine (1997)
Human Inborn Errors of Immunity: 2019 Update on the Classification from the International Union of Immunological Societies Expert Committee
Stuart G. Tangye;Waleed Al-Herz;Aziz Bousfiha;Talal Chatila.
Journal of Clinical Immunology (2020)
Primary Immunodeficiency Diseases: an Update on the Classification from the International Union of Immunological Societies Expert Committee for Primary Immunodeficiency 2015
Capucine Picard;Waleed Al-Herz;Aziz Bousfiha;Jean Laurent Casanova.
Journal of Clinical Immunology (2015)
International Union of Immunological Societies: 2017 Primary Immunodeficiency Diseases Committee Report on Inborn Errors of Immunity
Capucine Picard;H. Bobby Gaspar;Waleed Al-Herz;Aziz Bousfiha.
Journal of Clinical Immunology (2018)
Regulatory T cell development in the absence of functional Foxp3
Wen Lin;Dipica Haribhai;Lance M Relland;Nga Truong.
Nature Immunology (2007)
The 2017 IUIS Phenotypic Classification for Primary Immunodeficiencies.
Aziz Bousfiha;Leïla Jeddane;Capucine Picard;Fatima Ailal.
Journal of Clinical Immunology (2018)
Large deletions and point mutations involving the dedicator of cytokinesis 8 (DOCK8) in the autosomal-recessive form of hyper-IgE syndrome
Karin R. Engelhardt;Sean McGhee;Sabine Winkler;Atfa Sassi.
The Journal of Allergy and Clinical Immunology (2009)
A requisite role for induced regulatory T cells in tolerance based on expanding antigen receptor diversity.
Dipica Haribhai;Jason B. Williams;Shuang Jia;Derek Nickerson.
Immunity (2011)
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