D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Biology and Biochemistry D-index 42 Citations 6,485 86 World Ranking 15429 National Ranking 158

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • DNA
  • Gene expression

His scientific interests lie mostly in Molecular biology, Cell biology, Nonsense-mediated decay, RNA-binding protein and Translation. His work investigates the relationship between Molecular biology and topics such as P-bodies that intersect with problems in Messenger RNP and mRNA surveillance. His work on Stem cell as part of general Cell biology study is frequently linked to Mesenchyme, therefore connecting diverse disciplines of science.

His studies deal with areas such as Translational regulation, Translation initiation complex and Alternative splicing as well as Nonsense-mediated decay. His Translation initiation complex research is multidisciplinary, incorporating elements of Eukaryotic Initiation Factor-4E and Eukaryotic initiation factor. Many of his research projects under Translation are closely connected to Secretory pathway with Secretory pathway, tying the diverse disciplines of science together.

His most cited work include:

  • Mammalian Staufen1 recruits Upf1 to specific mRNA 3'UTRs so as to elicit mRNA decay. (395 citations)
  • Upf1 Phosphorylation Triggers Translational Repression during Nonsense-Mediated mRNA Decay (230 citations)
  • Quantitative microarray profiling provides evidence against widespread coupling of alternative splicing with nonsense-mediated mRNA decay to control gene expression (194 citations)

What are the main themes of his work throughout his whole career to date?

His primary areas of study are Cell biology, Molecular biology, Nonsense-mediated decay, Translation and Messenger RNA. His Cell biology research includes themes of Autophagy, Aggresome, Eukaryotic initiation factor and Transcriptome. Yoon Ki Kim interconnects P-bodies, EIF4E, Regulation of gene expression, Nuclear receptor and RNA-binding protein in the investigation of issues within Molecular biology.

His RNA-binding protein research is multidisciplinary, incorporating perspectives in Amino acid, Peptide sequence, C2C12 and Untranslated region. His Nonsense-mediated decay research is multidisciplinary, incorporating elements of Messenger RNP, Downregulation and upregulation, Mutant and Translation initiation complex. In his work, Nucleic acid sequence is strongly intertwined with RNA, which is a subfield of Messenger RNA.

He most often published in these fields:

  • Cell biology (53.13%)
  • Molecular biology (47.92%)
  • Nonsense-mediated decay (37.50%)

What were the highlights of his more recent work (between 2018-2021)?

  • Cell biology (53.13%)
  • Messenger RNA (23.96%)
  • RNA (20.83%)

In recent papers he was focusing on the following fields of study:

Yoon Ki Kim mainly investigates Cell biology, Messenger RNA, RNA, Transcriptome and Nonsense-mediated decay. His work deals with themes such as Cleavage, Polyadenylation, Frameshift mutation, RNA splicing and Histone, which intersect with Cell biology. His EIF4E study in the realm of Messenger RNA connects with subjects such as Importin.

In his study, which falls under the umbrella issue of RNA, RNA modification, Nucleotide, Nucleic acid sequence and microRNA is strongly linked to Gene expression. His Transcriptome research incorporates elements of Signal transduction, Axon, Regeneration, RNA Helicase A and Regulatory Pathway. The study incorporates disciplines such as Translation, Exon junction complex, mRNA surveillance, Hyperphosphorylation and Apoptosis in addition to Nonsense-mediated decay.

Between 2018 and 2021, his most popular works were:

  • Endoribonucleolytic Cleavage of m6A-Containing RNAs by RNase P/MRP Complex. (116 citations)
  • UPFront and center in RNA decay: UPF1 in nonsense-mediated mRNA decay and beyond (63 citations)
  • Molecular Mechanisms Driving mRNA Degradation by m6A Modification (33 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • DNA
  • Gene expression

The scientist’s investigation covers issues in Cell biology, Gene expression, RNA, Transcriptome and RNase P. Yoon Ki Kim works in the field of Cell biology, namely Retrograde signaling. His RNase P research includes elements of Cleavage and Circular RNA.

His Nucleic acid sequence study combines topics in areas such as RNA modification, Messenger RNA and Nucleotide. His work carried out in the field of Regulatory Pathway brings together such families of science as Nonsense-mediated decay, microRNA, RNA Helicase A and Frameshift mutation. The various areas that Yoon Ki Kim examines in his Molecular mechanism study include Endoribonucleases, Regulation of gene expression, CCR4-NOT complex and Signal transducing adaptor protein.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Mammalian Staufen1 recruits Upf1 to specific mRNA 3'UTRs so as to elicit mRNA decay.

Yoon Ki Kim;Luc Furic;Luc DesGroseillers;Lynne E. Maquat.
Cell (2005)

511 Citations

Upf1 Phosphorylation Triggers Translational Repression during Nonsense-Mediated mRNA Decay

Olaf Isken;Yoon Ki Kim;Nao Hosoda;Greg L. Mayeur.
Cell (2008)

284 Citations

Protein-protein interaction among hnRNPs shuttling between nucleus and cytoplasm

Jong Heon Kim;Bumsuk Hahm;Yoon Ki Kim;Mieyoung Choi.
Journal of Molecular Biology (2000)

259 Citations

Quantitative microarray profiling provides evidence against widespread coupling of alternative splicing with nonsense-mediated mRNA decay to control gene expression

Qun Pan;Arneet L. Saltzman;Yoon Ki Kim;Christine Misquitta.
Genes & Development (2006)

246 Citations

Heterogeneous nuclear ribonucleoprotein L interacts with the 3' border of the internal ribosomal entry site of hepatitis C virus.

Bumsuk Hahm;Yoon Ki Kim;Jong Heon Kim;Tae Yoon Kim.
Journal of Virology (1998)

236 Citations

Staufen1 regulates diverse classes of mammalian transcripts.

Yoon Ki Kim;Luc Furic;Marc Parisien;François Major.
The EMBO Journal (2007)

229 Citations

Regulation of Multiple Core Spliceosomal Proteins by Alternative Splicing-Coupled Nonsense-Mediated mRNA Decay

Arneet L. Saltzman;Yoon Ki Kim;Qun Pan;Matthew M. Fagnani.
Molecular and Cellular Biology (2008)

218 Citations

In vivo regeneration of murine prostate from dissociated cell populations of postnatal epithelia and urogenital sinus mesenchyme.

Li Xin;Hisamitsu Ide;Yoon Kim;Purnima Dubey.
Proceedings of the National Academy of Sciences of the United States of America (2003)

216 Citations

LIN28A Is a Suppressor of ER-Associated Translation in Embryonic Stem Cells

Jun Cho;Hyeshik Chang;S. Chul Kwon;Baekgyu Kim.
Cell (2012)

211 Citations

SMD and NMD are competitive pathways that contribute to myogenesis: effects on PAX3 and myogenin mRNAs

Chenguang Gong;Yoon Ki Kim;Collynn F. Woeller;Yalan Tang.
Genes & Development (2009)

172 Citations

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