2023 - Research.com Biology and Biochemistry in South Korea Leader Award
His primary areas of investigation include Cell biology, Molecular biology, Kinase, Mitogen-activated protein kinase kinase and ASK1. Cell biology connects with themes related to Biochemistry in his study. His work deals with themes such as Apoptosis, Cell culture, Nitric oxide and Transactivation, which intersect with Molecular biology.
In his study, 3T3 cells and Peptide binding is strongly linked to Heat shock protein, which falls under the umbrella field of Mitogen-activated protein kinase kinase. His ASK1 research is multidisciplinary, incorporating elements of Cancer research and MAP kinase kinase kinase. His Programmed cell death study combines topics in areas such as Autophagy, Autophagosome, Enzyme activator and Physiology.
Eui Ju Choi mainly investigates Cell biology, Molecular biology, Kinase, Signal transduction and ASK1. His Cell biology research incorporates themes from Apoptosis, Biochemistry and Programmed cell death. His Molecular biology research focuses on subjects like Mutant, which are linked to Superoxide dismutase.
His study looks at the relationship between Kinase and fields such as Nitric oxide, as well as how they intersect with chemical problems. His Signal transduction research is multidisciplinary, incorporating perspectives in Death-associated protein 6 and Cell fate determination. Eui Ju Choi has researched ASK1 in several fields, including c-Raf, Cancer research and MAP kinase kinase kinase.
The scientist’s investigation covers issues in Cell biology, Signal transduction, Autophagy, Kinase and SOD1. His Cell biology research incorporates elements of Apoptosis, Biochemistry and Programmed cell death. The concepts of his Autophagy study are interwoven with issues in Plasma protein binding, Computational biology and Bioinformatics.
His biological study focuses on ASK1. The Autolysosome research Eui Ju Choi does as part of his general Autophagosome study is frequently linked to other disciplines of science, such as Lysosome, therefore creating a link between diverse domains of science. His Autolysosome research includes themes of BECN1, Chaperone-mediated autophagy, MAP1LC3B, Sequestosome 1 and Physiology.
Eui Ju Choi focuses on Cell biology, Autophagy, Autophagosome, Lysosome and Bioinformatics. Eui Ju Choi works on Cell biology which deals in particular with p38 mitogen-activated protein kinases. While working in this field, Eui Ju Choi studies both Autophagy and Mechanism.
His Autophagosome research includes elements of Sequestosome 1, Plasma protein binding, Physiology and ATG5. The study incorporates disciplines such as Legionella pneumophila, Bacteria, Function and Xenophagy in addition to Bioinformatics. His studies in Autolysosome integrate themes in fields like BECN1, MAP1LC3B, Chaperone-mediated autophagy, Computational biology and Programmed cell death.
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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)
Daniel J. Klionsky;Kotb Abdelmohsen;Akihisa Abe;Joynal Abedin.
Pathological roles of MAPK signaling pathways in human diseases
Eun Kyung Kim;Eui Ju Choi.
Biochimica et Biophysica Acta (2010)
Glutathione S-Transferase Mu Modulates the Stress-activated Signals by Suppressing Apoptosis Signal-regulating Kinase 1
Ssang Goo Cho;Yong Hee Lee;Hee Sae Park;Kanghyun Ryoo.
Journal of Biological Chemistry (2001)
Compromised MAPK signaling in human diseases: an update
Eun Kyung Kim;Eui Ju Choi.
Archives of Toxicology (2015)
Hsp72 functions as a natural inhibitory protein of c-Jun N-terminal kinase
Hee Sae Park;Jae Seon Lee;Sung Ho Huh;Jeong Sun Seo.
The EMBO Journal (2001)
The tumour suppressor RASSF1A regulates mitosis by inhibiting the APC–Cdc20 complex
Min Sup Song;Min Sup Song;Su Jeong Song;Nagi G. Ayad;Jin Sook Chang.
Nature Cell Biology (2004)
Two distinct mechanisms are involved in 6-hydroxydopamine- and MPP+- induced dopaminergic neuronal cell death: Role of caspases, ROS, and JNK
Won Seok Choi;So Young Yoon;Tae H. Oh;Tae H. Oh;Eui Ju Choi.
Journal of Neuroscience Research (1999)
Activation of PI3K/Akt pathway by PTEN reduction and PIK3CA mRNA amplification contributes to cisplatin resistance in an ovarian cancer cell line
Sooyong Lee;Eui Ju Choi;Changbae Jin;Dong Hyun Kim.
Gynecologic Oncology (2005)
A non-enzymatic p21 protein inhibitor of stress-activated protein kinases.
Jaekyung Shim;Heejeong Lee;Jihyun Park;Hanshin Kim.
Stimulation of the type III olfactory adenylyl cyclase by calcium and calmodulin.
Eui Ju Choi;Zhengui Xia;Daniel R. Storm.
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