His scientific interests lie mostly in Immunology, Leishmania major, Microbiology, Leishmania and Interferon gamma. His study ties his expertise on Cytotoxic T cell together with the subject of Immunology. His studies deal with areas such as Cytokine, Intracellular parasite, Virology, Innate immune system and In vivo as well as Leishmania major.
His Microbiology research integrates issues from Apoptosis and Intracellular. The concepts of his Leishmania study are interwoven with issues in Chemotaxis and Granulocyte. His Interferon gamma study integrates concerns from other disciplines, such as Tumor necrosis factor alpha and Molecular biology.
Werner Solbach mostly deals with Immunology, Microbiology, Leishmania major, Immune system and Virology. Immunology and In vivo are commonly linked in his work. His Microbiology study combines topics from a wide range of disciplines, such as Infectivity and Intracellular.
Werner Solbach has included themes like Lymph, Interferon gamma, Intracellular parasite and BALB/c in his Leishmania major study. His Intracellular parasite research is multidisciplinary, incorporating perspectives in Macrophage and Effector. His Leishmania research includes elements of Leishmaniasis and Cutaneous leishmaniasis.
Werner Solbach mainly investigates Immunology, Cell biology, Apoptosis, Microbiology and Neutrophil extracellular traps. The study incorporates disciplines such as Internal medicine and Azithromycin in addition to Immunology. Werner Solbach combines subjects such as T cell, Immune system and Secretion with his study of Cell biology.
His work in Apoptosis tackles topics such as MAPK/ERK pathway which are related to areas like Leishmania major, Leishmania, Intrinsic apoptosis, Caspase 6 and Cytochrome c. His studies in Microbiology integrate themes in fields like Chlamydia trachomatis, Virology and Interferon gamma. The Virology study combines topics in areas such as Chlamydia and Intracellular parasite.
The scientist’s investigation covers issues in Immunology, Cell biology, Neutrophil extracellular traps, Apoptosis and NADPH oxidase. His study in Immunology is interdisciplinary in nature, drawing from both Circadian clock and Shiga toxin. His research in Shiga toxin focuses on subjects like Antibiotics, which are connected to Internal medicine.
His Cell biology research is multidisciplinary, relying on both Cell culture and Chlamydia trachomatis, Chlamydiae. His research in Apoptosis intersects with topics in Reactive oxygen species and Phagocytosis. His research investigates the connection between NADPH oxidase and topics such as Myeloperoxidase that intersect with problems in Degranulation, Proto-oncogene tyrosine-protein kinase Src and Function.
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Cutting Edge: Neutrophil Granulocyte Serves as a Vector for Leishmania Entry into Macrophages
Ger van Zandbergen;Matthias Klinger;Antje Mueller;Sonja Dannenberg.
Journal of Immunology (2004)
Chlamydia pneumoniae Infection in Circulating Human Monocytes Is Refractory to Antibiotic Treatment
Jens Gieffers;Henriette Füllgraf;Jürgen Jahn;Matthias Klinger.
Inhibition of the spontaneous apoptosis of neutrophil granulocytes by the intracellular parasite Leishmania major.
Eresso Aga;Dörthe M. Katschinski;Ger van Zandbergen;Helmut Laufs.
Journal of Immunology (2002)
IL-4-deficient Balb/c mice resist infection with Leishmania major.
M Kopf;F Brombacher;G Köhler;G Kienzle.
Journal of Experimental Medicine (1996)
Neutrophil granulocytes – Trojan horses for Leishmania major and other intracellular microbes?
Tamás Laskay;Ger van Zandbergen;Werner Solbach.
Trends in Microbiology (2003)
T-T cell interactions during cytotoxic T lymphocyte (CTL) responses: T cell derived helper factor (Interleukin 2) as a probe to analyze CTL responsiveness and thymic maturation of CTL progenitors.
Hermann Wagner;Conny Hardt;Klaus Heeg;Klaus Pfizenmaier.
Immunological Reviews (1980)
The host response to Leishmania infection.
Werner Solbach;Tamás Laskay.
Advances in Immunology (1999)
Intracellular survival of Leishmania major in neutrophil granulocytes after uptake in the absence of heat-labile serum factors.
Helmut Laufs;Kerstin Müller;Jens Fleischer;Norbert Reiling.
Infection and Immunity (2002)
Leishmania disease development depends on the presence of apoptotic promastigotes in the virulent inoculum
Ger van Zandbergen;Annalena Bollinger;Alexander Wenzel;Shaden Kamhawi.
Proceedings of the National Academy of Sciences of the United States of America (2006)
Tumor necrosis factor-α in combination with interferon-γ, but not with interleukin 4 activates murine macrophages for elimination of Leishmania major amastigotes.
Christian Bogdan;Heidrun Moll;Werner Solbach;Martin Röllinghoff.
European Journal of Immunology (1990)
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