2007 - Fellow of the American Association for the Advancement of Science (AAAS)
1992 - Fellow of the American Academy of Arts and Sciences
Ursula Storb spends much of her time researching Molecular biology, Gene, Genetics, Somatic hypermutation and Transgene. Her Molecular biology research incorporates themes from Immunoglobulin gene, Gene rearrangement, Methylation, Gene expression and Recombinase. In her research, Mutation is intimately related to Antibody, which falls under the overarching field of Gene.
The various areas that Ursula Storb examines in her Somatic hypermutation study include Germline mutation, Immunoglobulin class switching, Somatic cell, Germline and DNA mismatch repair. Her research investigates the connection between Germline mutation and topics such as Promoter that intersect with problems in Intron. Her Transcription research focuses on DNA repair and how it relates to Immunoglobulin genes and T-cell receptor.
Her primary scientific interests are in Molecular biology, Gene, Genetics, Somatic hypermutation and Antibody. Her Molecular biology study integrates concerns from other disciplines, such as Gene rearrangement, Genetically modified mouse, Transgene, DNA and Immunoglobulin light chain. Her studies in Transcription, Gene expression, Immunoglobulin gene, Enhancer and Regulation of gene expression are all subfields of Gene research.
Ursula Storb has included themes like Mutation, Germline mutation, Immunoglobulin class switching, Cytidine deaminase and DNA repair in her Somatic hypermutation study. Her Germline mutation research integrates issues from Promoter and Somatic cell. Her Antibody study combines topics from a wide range of disciplines, such as Spleen, Messenger RNA, Immune system and Antigen.
Her main research concerns Somatic hypermutation, Genetics, Molecular biology, Gene and Cytidine deaminase. Her research integrates issues of Mutation, Transcription, DNA and Immunoglobulin class switching in her study of Somatic hypermutation. Her research in the fields of Enhancer, Uracil-DNA glycosylase, Restriction enzyme and Homologous recombination overlaps with other disciplines such as Bacterial artificial chromosome.
The various areas that Ursula Storb examines in her Molecular biology study include Immunoglobulin gene, Antibody, Transgene, DNA mismatch repair and DNA polymerase. Her research brings together the fields of T-cell receptor and Gene. Her biological study spans a wide range of topics, including RNA editing, Cytidine deamination, Activation-induced deaminase, Chromatin and Cell biology.
The scientist’s investigation covers issues in Somatic hypermutation, Genetics, Gene, DNA and Cytidine deaminase. Her work deals with themes such as DNA polymerase, Molecular biology and DNA mismatch repair, which intersect with Somatic hypermutation. In general Genetics, her work in Transcription, Enhancer, Transgene and Immunoglobulin class switching is often linked to Cytosine deaminase linking many areas of study.
Specifically, her work in Gene is concerned with the study of DNA repair. Her study in the field of Immunoglobulin genes is also linked to topics like Deamination. Her study in Antibody is interdisciplinary in nature, drawing from both Mutation, TATA-binding protein and TATA box.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Mutation of BCL-6 gene in normal B cells by the process of somatic hypermutation of Ig genes
Hong Ming Shen;Andrew Peters;Beverly Baron;Xiangdong Zhu.
Somatic Hypermutation of Immunoglobulin Genes Is Linked to Transcription Initiation
Andrew Peters;Ursula Storb.
Pip, a novel IRF family member, is a lymphoid-specific, PU.1-dependent transcriptional activator.
Charles F. Eisenbeis;Harinder Singh;Ursula Storb.
Genes & Development (1995)
Pip, a lymphoid-restricted IRF, contains a regulatory domain that is important for autoinhibition and ternary complex formation with the Ets factor PU.1.
A L Brass;E Kehrli;C F Eisenbeis;U Storb.
Genes & Development (1996)
Allelic exclusion and control of endogenous immunoglobulin gene rearrangement in κ transgenic mice
Kindred A. Ritchie;Ralph L. Brinster;Ursula Storb.
PU.1 is a component of a multiprotein complex which binds an essential site in the murine immunoglobulin lambda 2-4 enhancer.
C F Eisenbeis;H Singh;U Storb.
Molecular and Cellular Biology (1993)
Expression of a microinjected immunoglobulin gene in the spleen of transgenic mice
Ralph L. Brinster;Kindred A. Ritchie;Robert E Hammer;Rebecca L. O'Brien.
A strain-specific modifier on mouse chromosome 4 controls the methylation of independent transgene loci
Peter Engler;Deanna Haasch;Carl A. Pinkert;Lynn Doglio.
AID in somatic hypermutation and class switch recombination
Simonne Longerich;Uttiya Basu;Frederick Alt;Ursula Storb.
Current Opinion in Immunology (2006)
Activation-induced cytidine deaminase (AID) can target both DNA strands when the DNA is supercoiled.
Hong Ming Shen;Ursula Storb.
Proceedings of the National Academy of Sciences of the United States of America (2004)
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