D-Index & Metrics Best Publications
Microbiology
Japan
2022

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Microbiology D-index 89 Citations 53,519 208 World Ranking 419 National Ranking 14
Medicine D-index 89 Citations 54,018 246 World Ranking 7820 National Ranking 213

Research.com Recognitions

Awards & Achievements

2022 - Research.com Microbiology in Japan Leader Award

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Immune system
  • Cytokine

His primary areas of investigation include Cell biology, Immunology, Toll-like receptor, Signal transduction and Receptor. The various areas that he examines in his Cell biology study include CD40, T cell, Interleukin 21, Myeloid Differentiation Factor 88 and Proinflammatory cytokine. His Immunology research focuses on Innate immune system, Immune system, Inflammation and Interferon.

His Innate immune system research is mostly focused on the topic Pattern recognition receptor. His work in the fields of TLR7, Chemokine and Innate lymphoid cell overlaps with other areas such as Cyclic GMP-AMP synthase. His Pathogen-associated molecular pattern study integrates concerns from other disciplines, such as Acquired immune system and NLRC5.

His most cited work include:

  • Pathogen Recognition and Innate Immunity (8453 citations)
  • Differential roles of MDA5 and RIG-I helicases in the recognition of RNA viruses (2867 citations)
  • Loss of the autophagy protein Atg16L1 enhances endotoxin-induced IL-1beta production. (1511 citations)

What are the main themes of his work throughout his whole career to date?

Immunology, Immune system, Cell biology, Innate immune system and Toll-like receptor are his primary areas of study. His Immunology study incorporates themes from Receptor and Flagellin. His work investigates the relationship between Immune system and topics such as Cytotoxic T cell that intersect with problems in CD8.

The concepts of his Cell biology study are interwoven with issues in Biochemistry, Cytokine and Innate lymphoid cell. As a part of the same scientific family, he mostly works in the field of Innate immune system, focusing on Microbiology and, on occasion, TLR2. Satoshi Uematsu has included themes like TRIF and Myeloid Differentiation Factor 88 in his Signal transduction study.

He most often published in these fields:

  • Immunology (51.97%)
  • Immune system (34.77%)
  • Cell biology (28.67%)

What were the highlights of his more recent work (between 2017-2021)?

  • Immunology (51.97%)
  • Internal medicine (17.92%)
  • Endocrinology (17.56%)

In recent papers he was focusing on the following fields of study:

Satoshi Uematsu spends much of his time researching Immunology, Internal medicine, Endocrinology, Immune system and Dysbiosis. While working in this field, he studies both Immunology and CpG Oligodeoxynucleotide. His Internal medicine study deals with Microsomal Prostaglandin E Synthase-1 intersecting with Acute stress, Neuroinflammation, Microglia and Inflammation.

His work in Endocrinology addresses issues such as Interleukin, which are connected to fields such as Excretion and Schistosoma mansoni worm. His Immune system study combines topics from a wide range of disciplines, such as Spleen and Antigen. His research investigates the connection between Microbiology and topics such as Interleukin 33 that intersect with problems in Cell biology.

Between 2017 and 2021, his most popular works were:

  • A Refined Culture System for Human Induced Pluripotent Stem Cell-Derived Intestinal Epithelial Organoids (36 citations)
  • Eosinophil depletion suppresses radiation-induced small intestinal fibrosis (22 citations)
  • Eosinophil depletion suppresses radiation-induced small intestinal fibrosis (22 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Immune system
  • Cytokine

Satoshi Uematsu mainly focuses on Cancer research, Chemokine, Receptor, Internal medicine and Endocrinology. Satoshi Uematsu combines subjects such as CCR3, Innate immune system, Small intestine and Eosinophil with his study of Cancer research. His Chemokine research integrates issues from Proinflammatory cytokine, Liver injury and Prostaglandin E2.

His studies in Receptor integrate themes in fields like Secretion, Gut flora and Carbohydrate. His work on Integrin alpha M, Prostaglandin E and Reperfusion injury as part of his general Internal medicine study is frequently connected to Miglitol, thereby bridging the divide between different branches of science. Specifically, his work in Endocrinology is concerned with the study of Oral administration.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Pathogen Recognition and Innate Immunity

Shizuo Akira;Satoshi Uematsu;Osamu Takeuchi.
Cell (2006)

13388 Citations

Differential roles of MDA5 and RIG-I helicases in the recognition of RNA viruses

Hiroki Kato;Osamu Takeuchi;Shintaro Sato;Mitsutoshi Yoneyama.
Nature (2006)

4027 Citations

Loss of the autophagy protein Atg16L1 enhances endotoxin-induced IL-1beta production.

Tatsuya Saitoh;Naonobu Fujita;Myoung Ho Jang;Satoshi Uematsu.
Nature (2008)

2055 Citations

Cell type-specific involvement of RIG-I in antiviral response

Hiroki Kato;Shintaro Sato;Mitsutoshi Yoneyama;Masahiro Yamamoto.
Immunity (2005)

1908 Citations

Sequence-specific potent induction of IFN-alpha by short interfering RNA in plasmacytoid dendritic cells through TLR7.

Veit Hornung;Margit Guenthner-Biller;Carole Bourquin;Andrea Ablasser.
Nature Medicine (2005)

1621 Citations

TRAM is specifically involved in the Toll-like receptor 4-mediated MyD88-independent signaling pathway

Masahiro Yamamoto;Shintaro Sato;Hiroaki Hemmi;Satoshi Uematsu.
Nature Immunology (2003)

1370 Citations

Essential role for TIRAP in activation of the signalling cascade shared by TLR2 and TLR4

Masahiro Yamamoto;Shintaro Sato;Hiroaki Hemmi;Hideki Sanjo.
Nature (2002)

1339 Citations

Nucleic acids of mammalian origin can act as endogenous ligands for Toll-like receptors and may promote systemic lupus erythematosus

Franck J. Barrat;Thea Meeker;Josh Gregorio;Jean H. Chan.
Journal of Experimental Medicine (2005)

1286 Citations

Interferon-|[alpha]| induction through Toll-like receptors involves a direct interaction of IRF7 with MyD88 and TRAF6

Taro Kawai;Shintaro Sato;Ken J. Ishii;Cevayir Coban.
Nature Immunology (2004)

1219 Citations

A Toll-like receptor–independent antiviral response induced by double-stranded B-form DNA

Ken J Ishii;Cevayir Coban;Hiroki Kato;Ken Takahashi.
Nature Immunology (2006)

921 Citations

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