2022 - Research.com Microbiology in Japan Leader Award
His primary areas of investigation include Cell biology, Immunology, Toll-like receptor, Signal transduction and Receptor. The various areas that he examines in his Cell biology study include CD40, T cell, Interleukin 21, Myeloid Differentiation Factor 88 and Proinflammatory cytokine. His Immunology research focuses on Innate immune system, Immune system, Inflammation and Interferon.
His Innate immune system research is mostly focused on the topic Pattern recognition receptor. His work in the fields of TLR7, Chemokine and Innate lymphoid cell overlaps with other areas such as Cyclic GMP-AMP synthase. His Pathogen-associated molecular pattern study integrates concerns from other disciplines, such as Acquired immune system and NLRC5.
Immunology, Immune system, Cell biology, Innate immune system and Toll-like receptor are his primary areas of study. His Immunology study incorporates themes from Receptor and Flagellin. His work investigates the relationship between Immune system and topics such as Cytotoxic T cell that intersect with problems in CD8.
The concepts of his Cell biology study are interwoven with issues in Biochemistry, Cytokine and Innate lymphoid cell. As a part of the same scientific family, he mostly works in the field of Innate immune system, focusing on Microbiology and, on occasion, TLR2. Satoshi Uematsu has included themes like TRIF and Myeloid Differentiation Factor 88 in his Signal transduction study.
Satoshi Uematsu spends much of his time researching Immunology, Internal medicine, Endocrinology, Immune system and Dysbiosis. While working in this field, he studies both Immunology and CpG Oligodeoxynucleotide. His Internal medicine study deals with Microsomal Prostaglandin E Synthase-1 intersecting with Acute stress, Neuroinflammation, Microglia and Inflammation.
His work in Endocrinology addresses issues such as Interleukin, which are connected to fields such as Excretion and Schistosoma mansoni worm. His Immune system study combines topics from a wide range of disciplines, such as Spleen and Antigen. His research investigates the connection between Microbiology and topics such as Interleukin 33 that intersect with problems in Cell biology.
Satoshi Uematsu mainly focuses on Cancer research, Chemokine, Receptor, Internal medicine and Endocrinology. Satoshi Uematsu combines subjects such as CCR3, Innate immune system, Small intestine and Eosinophil with his study of Cancer research. His Chemokine research integrates issues from Proinflammatory cytokine, Liver injury and Prostaglandin E2.
His studies in Receptor integrate themes in fields like Secretion, Gut flora and Carbohydrate. His work on Integrin alpha M, Prostaglandin E and Reperfusion injury as part of his general Internal medicine study is frequently connected to Miglitol, thereby bridging the divide between different branches of science. Specifically, his work in Endocrinology is concerned with the study of Oral administration.
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Pathogen Recognition and Innate Immunity
Shizuo Akira;Satoshi Uematsu;Osamu Takeuchi.
Differential roles of MDA5 and RIG-I helicases in the recognition of RNA viruses
Hiroki Kato;Osamu Takeuchi;Shintaro Sato;Mitsutoshi Yoneyama.
Loss of the autophagy protein Atg16L1 enhances endotoxin-induced IL-1beta production.
Tatsuya Saitoh;Naonobu Fujita;Myoung Ho Jang;Satoshi Uematsu.
Cell type-specific involvement of RIG-I in antiviral response
Hiroki Kato;Shintaro Sato;Mitsutoshi Yoneyama;Masahiro Yamamoto.
Sequence-specific potent induction of IFN-alpha by short interfering RNA in plasmacytoid dendritic cells through TLR7.
Veit Hornung;Margit Guenthner-Biller;Carole Bourquin;Andrea Ablasser.
Nature Medicine (2005)
TRAM is specifically involved in the Toll-like receptor 4-mediated MyD88-independent signaling pathway
Masahiro Yamamoto;Shintaro Sato;Hiroaki Hemmi;Satoshi Uematsu.
Nature Immunology (2003)
Essential role for TIRAP in activation of the signalling cascade shared by TLR2 and TLR4
Masahiro Yamamoto;Shintaro Sato;Hiroaki Hemmi;Hideki Sanjo.
Nucleic acids of mammalian origin can act as endogenous ligands for Toll-like receptors and may promote systemic lupus erythematosus
Franck J. Barrat;Thea Meeker;Josh Gregorio;Jean H. Chan.
Journal of Experimental Medicine (2005)
Interferon-|[alpha]| induction through Toll-like receptors involves a direct interaction of IRF7 with MyD88 and TRAF6
Taro Kawai;Shintaro Sato;Ken J. Ishii;Cevayir Coban.
Nature Immunology (2004)
A Toll-like receptor–independent antiviral response induced by double-stranded B-form DNA
Ken J Ishii;Cevayir Coban;Hiroki Kato;Ken Takahashi.
Nature Immunology (2006)
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