Rong Wang focuses on Endocrinology, Internal medicine, Biochemistry, Amyloid and Cell biology. The various areas that he examines in his Endocrinology study include Alzheimer's disease, Amyloid precursor protein secretase, Amyloid precursor protein and Genetically modified mouse. Rong Wang has researched Genetically modified mouse in several fields, including Pathogenesis and Pathology.
His work on Presenilin and Semagacestat as part of general Internal medicine study is frequently connected to Indometacin, therefore bridging the gap between diverse disciplines of science and establishing a new relationship between them. His study in Amyloid is interdisciplinary in nature, drawing from both Caloric theory, β amyloid and Beta. Rong Wang has included themes like Guanosine, Cell culture, Transmembrane domain and Cytosol in his Cell biology study.
His primary scientific interests are in Biochemistry, Cell biology, Amyloid precursor protein, Presenilin and Peptide. His work in Biochemistry addresses subjects such as Amyloid precursor protein secretase, which are connected to disciplines such as Gamma secretase. He interconnects Secretion, Receptor and Neuroscience in the investigation of issues within Cell biology.
His Amyloid precursor protein research is multidisciplinary, incorporating perspectives in Molecular biology and Amyloid. His studies deal with areas such as Endocrinology, Genetically modified mouse and Beta as well as Amyloid. His Presenilin research incorporates themes from Genetics and Mutant.
Rong Wang spends much of his time researching Biochemistry, Amyloid precursor protein, Cell biology, Regulation of gene expression and Chromatography. The study incorporates disciplines such as P3 peptide and Amyloid in addition to Biochemistry. The concepts of his Amyloid precursor protein study are interwoven with issues in Neurogenesis and Neuroscience.
His Cell biology study incorporates themes from Secretion, Postsynaptic potential and Immunology. His Regulation of gene expression research incorporates elements of Endocrinology, Gene expression and Gene knockdown. His Amyloid precursor protein secretase research is multidisciplinary, incorporating elements of Black cohosh, Toxicity, Gamma secretase and Presenilin.
Rong Wang mostly deals with Biochemistry, Amyloid precursor protein, Amyloid precursor protein secretase, Cell biology and Neuroscience. His research integrates issues of P3 peptide, Presenilin and Senile plaques in his study of Biochemistry. His Amyloid precursor protein research is multidisciplinary, relying on both Gamma secretase and Aminothiazole.
His work deals with themes such as Black cohosh, Natural product, Toxicity and Peptide, which intersect with Amyloid precursor protein secretase. In his study, Amyloid and PI3K/AKT/mTOR pathway is strongly linked to Neurotoxicity, which falls under the umbrella field of Cell biology. His Neuroscience research integrates issues from Huntingtin and Genetically modified mouse.
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Familial Alzheimer's Disease–Linked Presenilin 1 Variants Elevate Aβ1–42/1–40 Ratio In Vitro and In Vivo
David R. Borchelt;Gopal Thinakaran;Christopher B. Eckman;Christopher B. Eckman;Michael K. Lee.
A subset of NSAIDs lower amyloidogenic Aβ42 independently of cyclooxygenase activity
Sascha Weggen;Jason L. Eriksen;Pritam Das;Sarah A. Sagi.
Hypercholesterolemia accelerates the Alzheimer's amyloid pathology in a transgenic mouse model.
Lorenzo M. Refolo;Miguel A. Pappolla;Brian Malester;John LaFrancois.
Neurobiology of Disease (2000)
Structural alterations in the peptide backbone of beta-amyloid core protein may account for its deposition and stability in Alzheimer's disease.
A E Roher;J D Lowenson;S Clarke;C Wolkow.
Journal of Biological Chemistry (1993)
Proteomic Analysis of Proteins Associated with Lipid Droplets of Basal and Lipolytically Stimulated 3T3-L1 Adipocytes *
Dawn L. Brasaemle;Georgia Dolios;Lawrence Shapiro;Rong Wang.
Journal of Biological Chemistry (2004)
A Cholesterol-Lowering Drug Reduces β-Amyloid Pathology in a Transgenic Mouse Model of Alzheimer's Disease
L. M. Refolo;Miguel Pappolla;J. LaFrancois;B. Malester.
Neurobiology of Disease (2001)
Estrogen reduces neuronal generation of Alzheimer beta-amyloid peptides.
Huaxi Xu;Gunnar K. Gouras;Jeffrey P. Greenfield;Bruno Vincent.
Nature Medicine (1998)
Aβ42 Is Essential for Parenchymal and Vascular Amyloid Deposition in Mice
Eileen McGowan;Fiona Pickford;Jungsu Kim;Luisa Onstead.
Stimulation of β-Amyloid Precursor Protein Trafficking by Insulin Reduces Intraneuronal β-Amyloid and Requires Mitogen-Activated Protein Kinase Signaling
Laura Gasparini;Gunnar K. Gouras;Rong Wang;Rachel S. Gross.
The Journal of Neuroscience (2001)
Longer forms of amyloid beta protein: implications for the mechanism of intramembrane cleavage by gamma-secretase.
Yue Qi-Takahara;Maho Morishima-Kawashima;Yu Tanimura;Georgia Dolios.
The Journal of Neuroscience (2005)
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