What is he best known for?
The fields of study he is best known for:
His scientific interests lie mostly in Antibody, Molecular biology, Complement system, Biochemistry and Immunology.
His Antibody research includes themes of Plasma protein binding, Cyanogen bromide and Cytolysis.
His Molecular biology research is multidisciplinary, incorporating elements of Virology, Epstein–Barr virus, Pathology, Complement C3d and Heparinase.
The various areas that he examines in his Complement system study include Cytokine and Cell biology.
His Macromolecular Substances, Amino acid and Incubation study in the realm of Biochemistry connects with subjects such as Molecule and Diffusion.
His Immunology research is multidisciplinary, incorporating perspectives in Alzheimer's disease, Amyloid beta and Degenerative disease.
His most cited work include:
- Inflammation and Alzheimer's disease. (3451 citations)
- Complement activation by beta-amyloid in Alzheimer disease (725 citations)
- The classical complement pathway: activation and regulation of the first complement component. (421 citations)
What are the main themes of his work throughout his whole career to date?
His main research concerns Molecular biology, Complement system, Antibody, Biochemistry and Immunology.
His Molecular biology research incorporates elements of Antiserum, Epstein–Barr virus, Complement C3d, Epitope and Monoclonal antibody.
The Antibody study combines topics in areas such as Virology, Immune system, Antigen, Cytolysis and Lysis.
His Biochemistry research incorporates themes from Complement C1s and Calcium.
His Immunology study which covers Amyloid that intersects with Alzheimer's disease.
His work deals with themes such as Neuroscience and Cytokine, which intersect with Inflammation.
He most often published in these fields:
- Molecular biology (30.67%)
- Complement system (28.83%)
- Antibody (27.61%)
What were the highlights of his more recent work (between 1992-2017)?
- Immunology (22.09%)
- Complement system (28.83%)
- Cell biology (14.72%)
In recent papers he was focusing on the following fields of study:
Neil R. Cooper mainly investigates Immunology, Complement system, Cell biology, Inflammation and Molecular biology.
His Microglia, Human b cell, Immune system and Hemolysis study, which is part of a larger body of work in Immunology, is frequently linked to CD23, bridging the gap between disciplines.
His work in the fields of Classical complement pathway and Complement membrane attack complex overlaps with other areas such as In situ hybridization.
His Inflammation research is multidisciplinary, relying on both Cell culture, Cytokine, Disease, Amyloid and Neuroscience.
His studies in Neuroscience integrate themes in fields like Amyloid beta, Acute-phase protein and Pathogenesis.
His research investigates the connection with Molecular biology and areas like Epstein–Barr virus which intersect with concerns in CD19.
Between 1992 and 2017, his most popular works were:
- Inflammation and Alzheimer's disease. (3451 citations)
- Initiation of human cytomegalovirus infection requires initial interaction with cell surface heparan sulfate. (360 citations)
- Molecular and Cellular Characterization of the Membrane Attack Complex, C5b-9, in Alzheimer’s Disease (241 citations)
In his most recent research, the most cited papers focused on:
The scientist’s investigation covers issues in Cell biology, Complement system, Alzheimer's disease, Molecular biology and Neuroscience.
He has included themes like Hippocampal formation, Receptor and Beta-peptide in his Cell biology study.
His study of Classical complement pathway is a part of Complement system.
The study incorporates disciplines such as NF-κB, Signal transduction and Complement receptor in addition to Molecular biology.
His Neuroscience research integrates issues from Inflammation, Amyloid beta and Cytokine.
Neil R. Cooper interconnects Tau protein, Pathogenesis, Microglia, Cell activation and Neuroinflammation in the investigation of issues within Cytokine.
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