2008 - Fellow of the American Association for the Advancement of Science (AAAS)
Gary L. Wenk focuses on Neuroscience, Endocrinology, Internal medicine, Basal forebrain and Cholinergic. Gary L. Wenk has researched Neuroscience in several fields, including Alzheimer's disease and Memantine. His Endocrinology study combines topics from a wide range of disciplines, such as NMDA receptor, Neuroinflammation and Microglia.
His Neuroinflammation study is concerned with the larger field of Inflammation. His Basal forebrain study combines topics in areas such as Ibotenic acid, Radial arm maze and Cholinergic neuron. His work on Choline acetyltransferase as part of general Cholinergic study is frequently connected to Eyeblink conditioning, therefore bridging the gap between diverse disciplines of science and establishing a new relationship between them.
Gary L. Wenk spends much of his time researching Neuroscience, Internal medicine, Endocrinology, Neuroinflammation and Cholinergic. His research investigates the link between Neuroscience and topics such as NMDA receptor that cross with problems in Glutamate receptor and Pharmacology. His work in the fields of Choline acetyltransferase, Acetylcholine and Cerebral cortex overlaps with other areas such as Rett syndrome.
His work deals with themes such as Alzheimer's disease, Neurogenesis, Microglia and Endocannabinoid system, which intersect with Neuroinflammation. His study in Cholinergic is interdisciplinary in nature, drawing from both Cholinesterase, Serotonergic, Neurotransmission and Neurotransmitter. His study on Basal forebrain also encompasses disciplines like
His main research concerns Neuroinflammation, Neuroscience, Hippocampus, Internal medicine and Endocrinology. His Neuroinflammation study integrates concerns from other disciplines, such as Ryanodine receptor, Microglia, Endocannabinoid system, Pharmacology and Morris water navigation task. Within the field of Inflammation and Immunology Gary L. Wenk studies Microglia.
Gary L. Wenk has included themes like Agonist, Cannabinoid and Dantrolene in his Neuroscience study. His Hippocampus research is multidisciplinary, incorporating elements of Glutamatergic, Central nervous system, Neuron, Long-term potentiation and PI3K/AKT/mTOR pathway. Gary L. Wenk combines subjects such as Immune system, Signal transduction, Cytokine and Pathogenesis with his study of Endocrinology.
His main research concerns Neuroinflammation, Hippocampus, Neuroscience, Internal medicine and Microglia. His Neuroinflammation research is within the category of Immunology. Gary L. Wenk works mostly in the field of Neuroscience, limiting it down to topics relating to Agonist and, in certain cases, Memory impairment and Stimulation, as a part of the same area of interest.
His Internal medicine study frequently draws parallels with other fields, such as Endocrinology. Stratum lucidum, Long-term potentiation and Ageing is closely connected to Pathology in his research, which is encompassed under the umbrella topic of Microglia. His Pharmacology research includes elements of Nicotinic agonist and Cholinergic.
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Inflammation and Alzheimer's disease.
H Akiyama;S Barger;S Barnum;B Bradt.
Neurobiology of Aging (2000)
Neuropathologic changes in Alzheimer's disease.
Gary L Wenk.
The Journal of Clinical Psychiatry (2003)
Lesions in nucleus basalis magnocellularis and medial septal area of rats produce qualitatively similar memory impairments
DJ Hepler;DS Olton;GL Wenk;JT Coyle.
The Journal of Neuroscience (1985)
The Neuropharmacological Basis for the Use of Memantine in the Treatment of Alzheimer's Disease
Michael A. Rogawski;Gary L. Wenk.
Cns Drug Reviews (2006)
The Nucleus Basalis Magnocellularis Cholinergic System: One Hundred Years of Progress
Gary L. Wenk.
Neurobiology of Learning and Memory (1997)
Chronic neuroinflammation in rats reproduces components of the neurobiology of Alzheimer's disease
Beatrice Hauss-Wegrzyniak;Pawel Dobrzanski;James D Stoehr;Gary L Wenk.
Brain Research (1998)
Microglial Activation and β-Amyloid Deposit Reduction Caused by a Nitric Oxide-Releasing Nonsteroidal Anti-Inflammatory Drug in Amyloid Precursor Protein Plus Presenilin-1 Transgenic Mice
Paul T. Jantzen;Karen E. Connor;Giovanni DiCarlo;Gary L. Wenk.
The Journal of Neuroscience (2002)
Memory impairments following basal forebrain lesions.
David J. Hepler;Gary L. Wenk;Brenda L. Cribbs;David S. Olton.
Brain Research (1985)
Behavioral, biochemical, histological, and electrophysiological effects of 192 IgG-saporin injections into the basal forebrain of rats
Gary L. Wenk;James D. Stoehr;George Quintana;Sherri Mobley.
The Journal of Neuroscience (1994)
Age-related changes in multiple neurotransmitter systems in the monkey brain.
Gary L. Wenk;Donna J. Pierce;Robert G. Struble;Donald L. Price.
Neurobiology of Aging (1989)
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