The scientist’s investigation covers issues in Cell biology, Internal medicine, Endocrinology, Insulin and Biochemistry. The concepts of his Cell biology study are interwoven with issues in Hormesis, Oxidative stress and Glycolysis. His biological study spans a wide range of topics, including Diabetes mellitus and Type 2 diabetes.
His studies in Insulin integrate themes in fields like Gene targeting and Genetically modified mouse. His work in the fields of Biochemistry, such as Beta sheet and Protein structure, overlaps with other areas such as Frataxin and Side chain. Michael Ristow works mostly in the field of Reactive oxygen species, limiting it down to topics relating to Mitochondrion and, in certain cases, Cell signaling.
Michael Ristow spends much of his time researching Internal medicine, Endocrinology, Cell biology, Biochemistry and Insulin. His Internal medicine research integrates issues from Diabetes mellitus and Type 2 diabetes. His work on Insulin resistance, Carbohydrate metabolism, Adiponectin and Body mass index as part of his general Endocrinology study is frequently connected to Frataxin, thereby bridging the divide between different branches of science.
The study incorporates disciplines such as Hormesis, Oxidative stress and Calorie restriction in addition to Cell biology. His Oxidative stress study incorporates themes from Antioxidant and Vitamin E. Michael Ristow focuses mostly in the field of Insulin, narrowing it down to topics relating to Cre recombinase and, in certain cases, Molecular biology.
Michael Ristow mostly deals with Cell biology, Genetics, Phenotype, Reactive oxygen species and Carbohydrate metabolism. His Cell biology research includes themes of Hormesis, Oxidative stress, Biochemistry, Metabolism and Transcription factor. His work in Phenotype addresses subjects such as Model organism, which are connected to disciplines such as Stress resistance, Endogeny, Metabolite and Compartment.
His research investigates the connection between Reactive oxygen species and topics such as Mitochondrion that intersect with issues in Antioxidant, Metastasis, Adenosine triphosphate and Cell signaling. Michael Ristow's looking at Carbohydrate metabolism as part of his Endocrinology and Internal medicine and Carbohydrate metabolism study. Michael Ristow undertakes multidisciplinary investigations into Internal medicine and Severe acute respiratory syndrome coronavirus 2 in his work.
His scientific interests lie mostly in Cell biology, Hormesis, Metabolic pathway, Caenorhabditis elegans and Skeletal muscle. His Cell biology research is multidisciplinary, relying on both Oxidative stress and Biochemistry. His Hormesis study integrates concerns from other disciplines, such as Reactive nitrogen species, Muscle hypertrophy, Cell and Pharmacology.
He interconnects Dose dependence, Genome, Genome scale and Longevity in the investigation of issues within Metabolic pathway. His Skeletal muscle research incorporates themes from Myocyte, Cellular adaptation, Physical medicine and rehabilitation and Mitochondrial biogenesis. His NRF1 study, which is part of a larger body of work in Mitochondrion, is frequently linked to Cellular homeostasis, bridging the gap between disciplines.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Inflammatory Cytokines and the Risk to Develop Type 2 Diabetes: Results of the Prospective Population-Based European Prospective Investigation into Cancer and Nutrition (EPIC)-Potsdam Study
Joachim Spranger;Anja Kroke;Matthias Möhlig;Kurt Hoffmann.
Antioxidants prevent health-promoting effects of physical exercise in humans.
Michael Ristow;Kim Zarse;Andreas Oberbach;Nora Klöting.
Proceedings of the National Academy of Sciences of the United States of America (2009)
Adiponectin and protection against type 2 diabetes mellitus
Joachim Spranger;Anja Kroke;Matthias Möhlig;Manuela M Bergmann.
The Lancet (2003)
Glucose Restriction Extends Caenorhabditis elegans Life Span by Inducing Mitochondrial Respiration and Increasing Oxidative Stress
Tim J. Schulz;Kim Zarse;Anja Voigt;Nadine Urban.
Cell Metabolism (2007)
Obesity associated with a mutation in a genetic regulator of adipocyte differentiation.
M Ristow;D Müller-Wieland;A Pfeiffer;W Krone.
The New England Journal of Medicine (1998)
How increased oxidative stress promotes longevity and metabolic health: The concept of mitochondrial hormesis (mitohormesis)
Michael Ristow;Kim Zarse.
Experimental Gerontology (2010)
Extending life span by increasing oxidative stress.
Michael Ristow;Sebastian Schmeisser.
Free Radical Biology and Medicine (2011)
Muscle-specific PPARγ-deficient mice develop increased adiposity and insulin resistance but respond to thiazolidinediones
Andrew W. Norris;Lihong Chen;Simon J. Fisher;Ildiko Szanto.
Journal of Clinical Investigation (2003)
Animal models of obesity and diabetes mellitus
Maximilian Kleinert;Christoffer Clemmensen;Susanna M Hofmann;Mary C Moore.
Nature Reviews Endocrinology (2018)
Consuming fructose-sweetened beverages increases body adiposity in mice
Hella Jürgens;Wiltrud Haass;Tamara R. Castañeda;Annette Schürmann.
Obesity Research (2005)
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