D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Biology and Biochemistry D-index 59 Citations 17,076 144 World Ranking 8209 National Ranking 144

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Enzyme
  • Internal medicine

The scientist’s investigation covers issues in Cell biology, Internal medicine, Endocrinology, Insulin and Biochemistry. The concepts of his Cell biology study are interwoven with issues in Hormesis, Oxidative stress and Glycolysis. His biological study spans a wide range of topics, including Diabetes mellitus and Type 2 diabetes.

His studies in Insulin integrate themes in fields like Gene targeting and Genetically modified mouse. His work in the fields of Biochemistry, such as Beta sheet and Protein structure, overlaps with other areas such as Frataxin and Side chain. Michael Ristow works mostly in the field of Reactive oxygen species, limiting it down to topics relating to Mitochondrion and, in certain cases, Cell signaling.

His most cited work include:

  • Inflammatory Cytokines and the Risk to Develop Type 2 Diabetes: Results of the Prospective Population-Based European Prospective Investigation into Cancer and Nutrition (EPIC)-Potsdam Study (1139 citations)
  • Antioxidants prevent health-promoting effects of physical exercise in humans. (1099 citations)
  • Adiponectin and protection against type 2 diabetes mellitus (981 citations)

What are the main themes of his work throughout his whole career to date?

Michael Ristow spends much of his time researching Internal medicine, Endocrinology, Cell biology, Biochemistry and Insulin. His Internal medicine research integrates issues from Diabetes mellitus and Type 2 diabetes. His work on Insulin resistance, Carbohydrate metabolism, Adiponectin and Body mass index as part of his general Endocrinology study is frequently connected to Frataxin, thereby bridging the divide between different branches of science.

The study incorporates disciplines such as Hormesis, Oxidative stress and Calorie restriction in addition to Cell biology. His Oxidative stress study incorporates themes from Antioxidant and Vitamin E. Michael Ristow focuses mostly in the field of Insulin, narrowing it down to topics relating to Cre recombinase and, in certain cases, Molecular biology.

He most often published in these fields:

  • Internal medicine (44.07%)
  • Endocrinology (41.81%)
  • Cell biology (29.38%)

What were the highlights of his more recent work (between 2015-2021)?

  • Cell biology (29.38%)
  • Genetics (11.86%)
  • Phenotype (5.08%)

In recent papers he was focusing on the following fields of study:

Michael Ristow mostly deals with Cell biology, Genetics, Phenotype, Reactive oxygen species and Carbohydrate metabolism. His Cell biology research includes themes of Hormesis, Oxidative stress, Biochemistry, Metabolism and Transcription factor. His work in Phenotype addresses subjects such as Model organism, which are connected to disciplines such as Stress resistance, Endogeny, Metabolite and Compartment.

His research investigates the connection between Reactive oxygen species and topics such as Mitochondrion that intersect with issues in Antioxidant, Metastasis, Adenosine triphosphate and Cell signaling. Michael Ristow's looking at Carbohydrate metabolism as part of his Endocrinology and Internal medicine and Carbohydrate metabolism study. Michael Ristow undertakes multidisciplinary investigations into Internal medicine and Severe acute respiratory syndrome coronavirus 2 in his work.

Between 2015 and 2021, his most popular works were:

  • Animal models of obesity and diabetes mellitus (215 citations)
  • Do antioxidant supplements interfere with skeletal muscle adaptation to exercise training (122 citations)
  • Nuclear factor erythroid-derived 2-like 2 (NFE2L2, Nrf2) mediates exercise-induced mitochondrial biogenesis and the anti-oxidant response in mice. (102 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Enzyme
  • Internal medicine

His scientific interests lie mostly in Cell biology, Hormesis, Metabolic pathway, Caenorhabditis elegans and Skeletal muscle. His Cell biology research is multidisciplinary, relying on both Oxidative stress and Biochemistry. His Hormesis study integrates concerns from other disciplines, such as Reactive nitrogen species, Muscle hypertrophy, Cell and Pharmacology.

He interconnects Dose dependence, Genome, Genome scale and Longevity in the investigation of issues within Metabolic pathway. His Skeletal muscle research incorporates themes from Myocyte, Cellular adaptation, Physical medicine and rehabilitation and Mitochondrial biogenesis. His NRF1 study, which is part of a larger body of work in Mitochondrion, is frequently linked to Cellular homeostasis, bridging the gap between disciplines.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Inflammatory Cytokines and the Risk to Develop Type 2 Diabetes: Results of the Prospective Population-Based European Prospective Investigation into Cancer and Nutrition (EPIC)-Potsdam Study

Joachim Spranger;Anja Kroke;Matthias Möhlig;Kurt Hoffmann.
Diabetes (2003)

2018 Citations

Antioxidants prevent health-promoting effects of physical exercise in humans.

Michael Ristow;Kim Zarse;Andreas Oberbach;Nora Klöting.
Proceedings of the National Academy of Sciences of the United States of America (2009)

1722 Citations

Adiponectin and protection against type 2 diabetes mellitus

Joachim Spranger;Anja Kroke;Matthias Möhlig;Manuela M Bergmann.
The Lancet (2003)

1406 Citations

Glucose Restriction Extends Caenorhabditis elegans Life Span by Inducing Mitochondrial Respiration and Increasing Oxidative Stress

Tim J. Schulz;Kim Zarse;Anja Voigt;Nadine Urban.
Cell Metabolism (2007)

1131 Citations

Obesity associated with a mutation in a genetic regulator of adipocyte differentiation.

M Ristow;D Müller-Wieland;A Pfeiffer;W Krone.
The New England Journal of Medicine (1998)

719 Citations

How increased oxidative stress promotes longevity and metabolic health: The concept of mitochondrial hormesis (mitohormesis)

Michael Ristow;Kim Zarse.
Experimental Gerontology (2010)

706 Citations

Extending life span by increasing oxidative stress.

Michael Ristow;Sebastian Schmeisser.
Free Radical Biology and Medicine (2011)

691 Citations

Muscle-specific PPARγ-deficient mice develop increased adiposity and insulin resistance but respond to thiazolidinediones

Andrew W. Norris;Lihong Chen;Simon J. Fisher;Ildiko Szanto.
Journal of Clinical Investigation (2003)

507 Citations

Animal models of obesity and diabetes mellitus

Maximilian Kleinert;Christoffer Clemmensen;Susanna M Hofmann;Mary C Moore.
Nature Reviews Endocrinology (2018)

463 Citations

Consuming fructose-sweetened beverages increases body adiposity in mice

Hella Jürgens;Wiltrud Haass;Tamara R. Castañeda;Annette Schürmann.
Obesity Research (2005)

409 Citations

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