Her main research concerns Molecular biology, Mutant, Cell biology, Transfection and Cellular differentiation. Her Molecular biology research integrates issues from Malignant transformation, Spermatocyte, Gene and Spermatogenesis. Her work carried out in the field of Mutant brings together such families of science as Mutation, Tumor suppressor gene and Somatic cell.
She focuses mostly in the field of Tumor suppressor gene, narrowing it down to topics relating to Histone methyltransferase and, in certain cases, Cancer research. Her Cell biology research incorporates themes from Apoptosis, Biochemistry, Transcriptional regulation and Nuclear lamina. She interconnects Cell culture and Cell cycle in the investigation of issues within Cellular differentiation.
Her primary areas of study are Cell biology, Molecular biology, Mutant, Cancer research and Gene. The concepts of her Cell biology study are interwoven with issues in Cell culture, Genetics, Apoptosis, Cell cycle and Programmed cell death. Her Molecular biology research includes themes of Gene expression, Transactivation, Transfection, Complementary DNA and Epitope.
The various areas that she examines in her Mutant study include Mutation, Cancer cell, Protein structure and Immunology. Naomi Goldfinger has included themes like Carcinogenesis, Epithelial–mesenchymal transition, Stem cell and Prostate cancer in her Cancer research study. Her Gene study incorporates themes from Testicle and Pathogenesis.
Naomi Goldfinger mostly deals with Cancer research, Transcription factor, Mutant, Cell biology and Molecular biology. Her biological study spans a wide range of topics, including Epithelial–mesenchymal transition, Carcinogenesis, Tumor microenvironment, Stem cell and Mesenchymal stem cell. Her Transcription factor research is multidisciplinary, relying on both Endocrinology, Microarray analysis techniques, Gene expression, Post-translational regulation and Programmed cell death.
Her studies deal with areas such as Cell culture, Cancer cell, Interferon, Immunology and Mutation as well as Mutant. She connects Cell biology with Pentose phosphate pathway in her study. Her studies in Molecular biology integrate themes in fields like BTG2, Vinculin, Western blot, Gene and Protein structure.
The scientist’s investigation covers issues in Transcription factor, Cancer research, Mutant, Regulation of gene expression and Cell biology. The study incorporates disciplines such as Apoptosis, Programmed cell death, Internal medicine, Energy metabolism and Adipogenesis in addition to Transcription factor. Her research integrates issues of Epithelial–mesenchymal transition, Cancer, Immunology and Telomerase reverse transcriptase in her study of Cancer research.
Her Mutant study combines topics in areas such as Cancer cell and Cell culture. Her Regulation of gene expression study combines topics from a wide range of disciplines, such as BTG2, Enzyme activator, Transfection, Molecular biology and Transcription. Her Cell biology research is multidisciplinary, incorporating elements of G6PC and Bioinformatics.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Nuclear accumulation of p53 protein is mediated by several nuclear localization signals and plays a role in tumorigenesis.
G. Shaulsky;N. Goldfinger;Avri Ben-Ze'ev;V. Rotter.
Molecular and Cellular Biology (1990)
Meth A fibrosarcoma cells express two transforming mutant p53 species.
Eliyahu D;Goldfinger N;Pinhasi-Kimhi O;Shaulsky G.
Epigenetic polymorphism and the stochastic formation of differentially methylated regions in normal and cancerous tissues
Gilad Landan;Netta Mendelson Cohen;Zohar Mukamel;Amir Bar.
Nature Genetics (2012)
Mice with reduced levels of p53 protein exhibit the testicular giant-cell degenerative syndrome.
Varda Rotter;Dov Schwartz;Einat Almon;Naomi Goldfinger.
Proceedings of the National Academy of Sciences of the United States of America (1993)
Nuclear localization is essential for the activity of p53 protein.
G Shaulsky;N Goldfinger;M S Tosky;A J Levine.
Activated p53 suppresses the histone methyltransferase EZH2 gene
Xiaohu Tang;Michael Milyavsky;Igor Shats;Neta Erez.
Involvement of wild-type p53 in pre-B-cell differentiation in vitro.
Gad Shaulsky;Naomi Goldfinger;Alpha Peled;Varda Rotter.
Proceedings of the National Academy of Sciences of the United States of America (1991)
Regulation of AIF expression by p53
P Stambolsky;L Weisz;I Shats;Y Klein.
Cell Death & Differentiation (2006)
Expression of p53 protein in spermatogenesis is confined to the tetraploid pachytene primary spermatocytes
D Schwartz;N Goldfinger;V Rotter.
p53 Plays a Role in Mesenchymal Differentiation Programs, in a Cell Fate Dependent Manner
Alina Molchadsky;Igor Shats;Naomi Goldfinger;Meirav Pevsner-Fischer.
PLOS ONE (2008)
If you think any of the details on this page are incorrect, let us know.
We appreciate your kind effort to assist us to improve this page, it would be helpful providing us with as much detail as possible in the text box below: