Giovanni Blandino mainly investigates Cancer research, Mutant, Molecular biology, Carcinogenesis and Transcription factor. His Cancer research study combines topics from a wide range of disciplines, such as Cell culture, Cell growth, Cisplatin, Apoptosis and Immunology. His Apoptosis research is multidisciplinary, incorporating elements of Endogeny, Transcription and DNA damage.
His Mutant study incorporates themes from In vitro, Mutation, Cancer cell, Tumor suppressor gene and Gene silencing. His Molecular biology study integrates concerns from other disciplines, such as Downregulation and upregulation, microRNA, Cell biology, C2C12 and Gene isoform. His studies in Cell biology integrate themes in fields like Cell cycle, Cancer and Biochemistry.
His scientific interests lie mostly in Cancer research, microRNA, Cancer, Cell biology and Mutant. The various areas that he examines in his Cancer research study include Cell culture, Cell growth, Carcinogenesis, Apoptosis and Immunology. The microRNA study combines topics in areas such as Gene expression, Bioinformatics, Downregulation and upregulation, Lung cancer and Breast cancer.
Giovanni Blandino has researched Cell biology in several fields, including Cellular differentiation and DNA damage. His Mutant research is multidisciplinary, incorporating perspectives in Mutation, Molecular biology and Transcription factor. Giovanni Blandino has included themes like Promoter and Transactivation in his Molecular biology study.
His primary areas of investigation include Cancer research, microRNA, Cancer, Internal medicine and Oncology. His Cancer research research incorporates themes from Cell migration, Carcinogenesis, Hippo signaling pathway, Metastasis and Lung cancer. His study in Metastasis is interdisciplinary in nature, drawing from both Blockade, Breast cancer and Mutant.
His research in microRNA intersects with topics in Gene expression, Head and neck squamous-cell carcinoma, Downregulation and upregulation, Gene silencing and Computational biology. He studied Gene expression and Cell biology that intersect with Receptor. Particularly relevant to Cancer cell is his body of work in Cancer.
The scientist’s investigation covers issues in microRNA, Cancer, Cancer research, Metastasis and Cancer cell. His microRNA research integrates issues from Gene expression and Computational biology. The study incorporates disciplines such as Cell migration, Transcription factor and PTEN in addition to Cancer.
His work carried out in the field of Cancer research brings together such families of science as Carcinogenesis, Head and neck squamous-cell carcinoma, Downregulation and upregulation and Biomarker. His Carcinogenesis research is multidisciplinary, relying on both DNA damage, Genome instability and DNA repair. His Metastasis research focuses on Mutant and how it connects with Gene signature and Regulation of gene expression.
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Interaction of c-Abl and p73α and their collaboration to induce apoptosis
Reuven Agami;Giovanni Blandino;Moshe Oren;Yosef Shaul.
Mutant p53 gain of function: differential effects of different p53 mutants on resistance of cultured cells to chemotherapy
Giovanni Blandino;Arnold J Levine;Moshe Oren.
The prolyl isomerase Pin1 reveals a mechanism to control p53 functions after genotoxic insults
Paola Zacchi;Paola Zacchi;Monica Gostissa;Takafumi Uchida;Clio Salvagno;Clio Salvagno.
Physical interaction with Yes-associated protein enhances p73 transcriptional activity.
Sabrina Strano;Eliana Munarriz;Mario Rossi;Luisa Castagnoli.
Journal of Biological Chemistry (2001)
Gain of function of mutant p53: The mutant p53/NF-Y protein complex reveals an aberrant transcriptional mechanism of cell cycle regulation
Silvia Di Agostino;Sabrina Strano;Velia Emiliozzi;Valentina Zerbini.
Cancer Cell (2006)
DNA Damage-Dependent Acetylation of p73 Dictates the Selective Activation of Apoptotic Target Genes
Antonio Costanzo;Antonio Costanzo;Paola Merlo;Natalia Pediconi;Marcella Fulco.
Molecular Cell (2002)
The transcriptional coactivator Yes-associated protein drives p73 gene-target specificity in response to DNA Damage.
Sabrina Strano;Olimpia Monti;Natalia Pediconi;Alessia Baccarini.
Molecular Cell (2005)
Mutant p53: an oncogenic transcription factor
S Strano;S Dell'Orso;S Di Agostino;G Fontemaggi.
Physical interaction with human tumor-derived p53 mutants inhibits p63 activities
Sabrina Strano;Giulia Fontemaggi;Antonio Costanzo;Maria Giulia Rizzo.
Journal of Biological Chemistry (2002)
Physical and Functional Interaction between p53 Mutants and Different Isoforms of p73
Sabrina Strano;Eliana Munarriz;Mario Rossi;Barbara Cristofanelli.
Journal of Biological Chemistry (2000)
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