What is he best known for?
The fields of study he is best known for:
Giovanni Blandino mainly investigates Cancer research, Mutant, Molecular biology, Carcinogenesis and Transcription factor.
His Cancer research study combines topics from a wide range of disciplines, such as Cell culture, Cell growth, Cisplatin, Apoptosis and Immunology.
His Apoptosis research is multidisciplinary, incorporating elements of Endogeny, Transcription and DNA damage.
His Mutant study incorporates themes from In vitro, Mutation, Cancer cell, Tumor suppressor gene and Gene silencing.
His Molecular biology study integrates concerns from other disciplines, such as Downregulation and upregulation, microRNA, Cell biology, C2C12 and Gene isoform.
His studies in Cell biology integrate themes in fields like Cell cycle, Cancer and Biochemistry.
His most cited work include:
- Interaction of c-Abl and p73α and their collaboration to induce apoptosis (514 citations)
- Mutant p53 gain of function: differential effects of different p53 mutants on resistance of cultured cells to chemotherapy (385 citations)
- The prolyl isomerase Pin1 reveals a mechanism to control p53 functions after genotoxic insults (372 citations)
What are the main themes of his work throughout his whole career to date?
His scientific interests lie mostly in Cancer research, microRNA, Cancer, Cell biology and Mutant.
The various areas that he examines in his Cancer research study include Cell culture, Cell growth, Carcinogenesis, Apoptosis and Immunology.
The microRNA study combines topics in areas such as Gene expression, Bioinformatics, Downregulation and upregulation, Lung cancer and Breast cancer.
Giovanni Blandino has researched Cell biology in several fields, including Cellular differentiation and DNA damage.
His Mutant research is multidisciplinary, incorporating perspectives in Mutation, Molecular biology and Transcription factor.
Giovanni Blandino has included themes like Promoter and Transactivation in his Molecular biology study.
He most often published in these fields:
- Cancer research (43.22%)
- microRNA (24.29%)
- Cancer (20.50%)
What were the highlights of his more recent work (between 2018-2021)?
- Cancer research (43.22%)
- microRNA (24.29%)
- Cancer (20.50%)
In recent papers he was focusing on the following fields of study:
His primary areas of investigation include Cancer research, microRNA, Cancer, Internal medicine and Oncology.
His Cancer research research incorporates themes from Cell migration, Carcinogenesis, Hippo signaling pathway, Metastasis and Lung cancer.
His study in Metastasis is interdisciplinary in nature, drawing from both Blockade, Breast cancer and Mutant.
His research in microRNA intersects with topics in Gene expression, Head and neck squamous-cell carcinoma, Downregulation and upregulation, Gene silencing and Computational biology.
He studied Gene expression and Cell biology that intersect with Receptor.
Particularly relevant to Cancer cell is his body of work in Cancer.
Between 2018 and 2021, his most popular works were:
- The circRNA-microRNA code: emerging implications for cancer diagnosis and treatment. (105 citations)
- miR-96-5p targets PTEN expression affecting radio-chemosensitivity of HNSCC cells. (24 citations)
- TRF2 positively regulates SULF2 expression increasing VEGF-A release and activity in tumor microenvironment. (16 citations)
In his most recent research, the most cited papers focused on:
The scientist’s investigation covers issues in microRNA, Cancer, Cancer research, Metastasis and Cancer cell.
His microRNA research integrates issues from Gene expression and Computational biology.
The study incorporates disciplines such as Cell migration, Transcription factor and PTEN in addition to Cancer.
His work carried out in the field of Cancer research brings together such families of science as Carcinogenesis, Head and neck squamous-cell carcinoma, Downregulation and upregulation and Biomarker.
His Carcinogenesis research is multidisciplinary, relying on both DNA damage, Genome instability and DNA repair.
His Metastasis research focuses on Mutant and how it connects with Gene signature and Regulation of gene expression.
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