D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Medicine D-index 74 Citations 14,568 227 World Ranking 15427 National Ranking 7889

Overview

What is he best known for?

The fields of study he is best known for:

  • Internal medicine
  • Enzyme
  • Surgery

Michael L. Steer mainly focuses on Pancreatitis, Internal medicine, Endocrinology, Acute pancreatitis and Pancreatic disease. Michael L. Steer interconnects Lung injury, Ceruletide and Pancreas in the investigation of issues within Pancreatitis. Michael L. Steer has researched Internal medicine in several fields, including Gastroenterology and Lysosome.

His research investigates the connection between Endocrinology and topics such as Cathepsin B that intersect with problems in Trypsin. His work carried out in the field of Pancreatic disease brings together such families of science as Surgery, Radiology and Pathology. The various areas that Michael L. Steer examines in his Acinar cell study include Pancreatic injury and Programmed cell death.

His most cited work include:

  • Relationship between severity, necrosis, and apoptosis in five models of experimental acute pancreatitis. (262 citations)
  • Role of substance P and the neurokinin 1 receptor in acute pancreatitis and pancreatitis-associated lung injury (261 citations)
  • The role of intercellular adhesion molecule 1 and neutrophils in acute pancreatitis and pancreatitis-associated lung injury. (235 citations)

What are the main themes of his work throughout his whole career to date?

His primary areas of study are Internal medicine, Pancreatitis, Endocrinology, Acute pancreatitis and Acinar cell. His studies in Internal medicine integrate themes in fields like Gastroenterology, Trypsinogen and Cathepsin B. His work deals with themes such as Apoptosis, Immunology, Necrosis, Ceruletide and Pancreatic disease, which intersect with Pancreatitis.

The study incorporates disciplines such as Cholecystokinin and Amylase in addition to Endocrinology. Michael L. Steer combines subjects such as Edema, Pathogenesis, Pathology, Lung injury and Bile reflux with his study of Acute pancreatitis. The concepts of his Acinar cell study are interwoven with issues in Pancreatic injury, Cathepsin, Programmed cell death, Pharmacology and Hyperamylasemia.

He most often published in these fields:

  • Internal medicine (59.82%)
  • Pancreatitis (43.75%)
  • Endocrinology (42.41%)

What were the highlights of his more recent work (between 1998-2019)?

  • Pancreatitis (43.75%)
  • Internal medicine (59.82%)
  • Acinar cell (23.66%)

In recent papers he was focusing on the following fields of study:

Michael L. Steer spends much of his time researching Pancreatitis, Internal medicine, Acinar cell, Acute pancreatitis and Endocrinology. His research integrates issues of Cancer research, Pharmacology, Proinflammatory cytokine, Ceruletide and Pancreatic disease in his study of Pancreatitis. His Internal medicine research includes elements of Gastroenterology and Trypsinogen.

His Acinar cell research is multidisciplinary, incorporating perspectives in MDia1, Cytoskeleton, Arp2/3 complex and Cell biology. His Acute pancreatitis research includes themes of Lung injury and Immunology, Pathogenesis, Pathology. His Endocrinology study integrates concerns from other disciplines, such as Cholecystokinin, Zymogen activation and Intracellular.

Between 1998 and 2019, his most popular works were:

  • The role of intercellular adhesion molecule 1 and neutrophils in acute pancreatitis and pancreatitis-associated lung injury. (235 citations)
  • Secretagogue-induced digestive enzyme activation and cell injury in rat pancreatic acini. (163 citations)
  • Inhibition of ubiquitin-proteasome pathway–mediated IκBα degradation by a naturally occurring antibacterial peptide (147 citations)

In his most recent research, the most cited papers focused on:

  • Internal medicine
  • Enzyme
  • Surgery

His primary scientific interests are in Pancreatitis, Pancreatic disease, Acute pancreatitis, Internal medicine and Acinar cell. His Pancreatitis study combines topics from a wide range of disciplines, such as Proteases, Receptor, Ceruletide and Trypsin. His Pancreatic disease study combines topics in areas such as Serine, Ligand, Immunology, Necrosis and Lung injury.

His Internal medicine study frequently links to related topics such as Endocrinology. Michael L. Steer has included themes like Trypsinogen and Trypsinogen activation in his Endocrinology study. In his study, Cathepsin B is inextricably linked to Cell activation, which falls within the broad field of Trypsinogen.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Relationship between severity, necrosis, and apoptosis in five models of experimental acute pancreatitis.

A. M. Kaiser;A. K. Saluja;Ashok Sengupta;Manju Saluja.
American Journal of Physiology-cell Physiology (1995)

476 Citations

The role of intercellular adhesion molecule 1 and neutrophils in acute pancreatitis and pancreatitis-associated lung injury.

Jean-Louis Frossard;Ashok Saluja;Lakshmi Bhagat;Hong Sik Lee.
Gastroenterology (1999)

385 Citations

Role of substance P and the neurokinin 1 receptor in acute pancreatitis and pancreatitis-associated lung injury

Madhav Bhatia;Ashok K. Saluja;Bernd Hofbauer;Jean Louis Frossard.
Proceedings of the National Academy of Sciences of the United States of America (1998)

355 Citations

Intra-acinar cell activation of trypsinogen during caerulein-induced pancreatitis in rats

B. Hofbauer;A. K. Saluja;M. M. Lerch;L. Bhagat.
American Journal of Physiology-gastrointestinal and Liver Physiology (1998)

349 Citations

Supramaximal caerulein stimulation and ultrastructure of rat pancreatic acinar cell: early morphological changes during development of experimental pancreatitis

O. Watanabe;F. M. Baccino;M. L. Steer;J. Meldolesi.
American Journal of Physiology-gastrointestinal and Liver Physiology (1984)

341 Citations

Pancreatic duct obstruction triggers acute necrotizing pancreatitis in the opossum

Markus M. Lerch;Ashok K. Saluja;Ashok K. Saluja;Michael Rünzi;Michael Rünzi;Rajinder Dawra;Rajinder Dawra.
Gastroenterology (1993)

317 Citations

The Cell Biology of Experimental Pancreatitis

Michael L. Steer;Jacopo Meldolesi.
The New England Journal of Medicine (1987)

311 Citations

The use of helical CT and CT angiography to predict vascular involvement from pancreatic cancer: correlation with findings at surgery.

V Raptopoulos;M L Steer;R G Sheiman;T G Vrachliotis.
American Journal of Roentgenology (1997)

289 Citations

Cerulein-induced in vitro activation of trypsinogen in rat pancreatic acini is mediated by cathepsin B

Ashok K. Saluja;Erin A. Donovan;Kenji Yamanaka;Yoshikazu Yamaguchi.
Gastroenterology (1997)

276 Citations

Targeted disruption of the beta-chemokine receptor CCR1 protects against pancreatitis-associated lung injury.

Craig Gerard;Jean Louis Frossard;Madhav Bhatia;Ashok Saluja.
Journal of Clinical Investigation (1997)

263 Citations

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