2012 - Fellow, National Academy of Inventors
John J. Kopchick mainly focuses on Endocrinology, Internal medicine, Growth hormone receptor, Receptor and Adipose tissue. John J. Kopchick has included themes like Genetically modified mouse, Transgene and Growth factor in his Endocrinology study. As part of his studies on Internal medicine, John J. Kopchick often connects relevant areas like Signal transduction.
The study incorporates disciplines such as Diabetes mellitus, Bone growth and Tyrosine phosphorylation in addition to Growth hormone receptor. His Adipose tissue study integrates concerns from other disciplines, such as Lean body mass and Obesity. His Laron syndrome research incorporates elements of Heterozygote advantage and Gene deletion.
Internal medicine, Endocrinology, Growth hormone receptor, Receptor and Growth hormone are his primary areas of study. White adipose tissue, Antagonist, Insulin resistance, Knockout mouse and Hormone are the subjects of his Internal medicine studies. His Antagonist research incorporates themes from Pegvisomant and Acromegaly.
John J. Kopchick combines subjects such as Genetically modified mouse, Transgene and Growth factor with his study of Endocrinology. His work carried out in the field of Genetically modified mouse brings together such families of science as Gene expression and Bovine somatotropin. His study in Growth hormone receptor is interdisciplinary in nature, drawing from both Hypothalamus, Signal transduction and Glucose homeostasis.
His primary areas of study are Internal medicine, Endocrinology, Growth hormone receptor, Adipose tissue and Growth hormone. His Internal medicine research is multidisciplinary, incorporating perspectives in Diabetes mellitus and Transgene. His Endocrinology research focuses on Insulin, White adipose tissue, Lipolysis, Metabolism and Hypothalamus.
His Growth hormone receptor research includes themes of Arcuate nucleus, Cancer research, Knockout mouse, Prolactin and Cell biology. His Adipose tissue study incorporates themes from Transcriptome, Gene expression, Gene and Genetically modified mouse. The various areas that John J. Kopchick examines in his Growth hormone study include Weight loss, Bioinformatics and Longevity.
His scientific interests lie mostly in Internal medicine, Endocrinology, Growth hormone receptor, Adipose tissue and Insulin resistance. His Glucose homeostasis, Growth hormone, Receptor, Hypothalamus and Anterior pituitary investigations are all subjects of Internal medicine research. The concepts of his Growth hormone study are interwoven with issues in Cancer and Metabolism.
His Endocrinology study integrates concerns from other disciplines, such as Histopathology and Longevity. His Growth hormone receptor research is multidisciplinary, relying on both Leptin receptor, White adipose tissue, Knockout mouse, Insulin and Fibrosis. John J. Kopchick combines subjects such as Binding protein, Transcription and Transgene with his study of Adipose tissue.
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A mammalian model for Laron syndrome produced by targeted disruption of the mouse growth hormone receptor/binding protein gene (the Laron mouse)
Yihua Zhou;Bixiong C. Xu;Hiralal G. Maheshwari;Li He.
Proceedings of the National Academy of Sciences of the United States of America (1997)
Assessment of growth parameters and life span of GHR/BP gene-disrupted mice.
Karen T. Coschigano;David Clemmons;Linda L. Bellush;John J. Kopchick;John J. Kopchick.
Endocrinology (2000)
Growth hormone antagonists
John J. Kopchick;Wen Y. Chen.
(1992)
Deletion, but not antagonism, of the mouse growth hormone receptor results in severely decreased body weights, insulin, and insulin-like growth factor I levels and increased life span.
Karen T. Coschigano;Amy N. Holland;Markus E. Riders;Edward O. List.
Endocrinology (2003)
Essential Role of Growth Hormone in Ischemia-Induced Retinal Neovascularization
Lois E. H. Smith;John J. Kopchick;Wen Chen;Joanne Knapp.
Science (1997)
Growth Hormone Receptor Antagonists: Discovery, Development, and Use in Patients with Acromegaly
J J Kopchick;Craig Parkinson;E C Stevens;Peter J Trainer.
Endocrine Reviews (2002)
Interventions to Slow Aging in Humans: Are We Ready?
Valter D. Longo;Adam Antebi;Andrzej Bartke;Nir Barzilai.
Aging Cell (2015)
Prolonged Fasting Reduces IGF-1/PKA to Promote Hematopoietic-Stem-Cell-Based Regeneration and Reverse Immunosuppression
Chia-Wei Cheng;Gregor B. Adams;Laura Perin;Min Wei.
Cell Stem Cell (2014)
The GH/IGF-1 axis in ageing and longevity
Riia K. Junnila;Edward O. List;Darlene E. Berryman;John W. Murrey.
Nature Reviews Endocrinology (2013)
Growth hormone (GH), GH receptor, and signal transduction.
John J. Kopchick;Jerald M. Andry.
Molecular Genetics and Metabolism (2000)
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