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Molecular Biology

D-Index
52
Citations
8430
World Ranking
2449
National Ranking
1207

Overview

John H. Wilson is affiliated with Baylor College of Medicine in the United States, with a research focus spanning medicine, biochemistry, genetics, molecular biology, and neuroscience. Their work includes subfields such as cardiology and cardiovascular medicine, surgery, molecular biology, cellular and molecular neuroscience, and obstetrics and gynecology.

The scientist has contributed to multiple specific topics within these fields, including:

  • Cardiovascular Issues in Pregnancy
  • Retinal Development and Disorders
  • Pregnancy and Preeclampsia Studies
  • Photoreceptor and Optogenetics Research
  • Cellular Transport and Secretion
  • Electrolyte and Hormonal Disorders
  • Cardiac Structural Anomalies and Repair

John H. Wilson has several notable publications in peer-reviewed venues. Selected recent papers include:

  • Soy Protein Nanofiber Scaffolds for Uniform Maturation of Human Induced Pluripotent Stem Cell-Derived Retinal Pigment Epithelium (2020), published in Tissue Engineering Part C Methods
  • Subcellular localization of mutant P23H rhodopsin in an RFP fusion knock-in mouse model of retinitis pigmentosa (2022), published in Disease Models & Mechanisms
  • Histone deacetylase knockouts modify transcription, CAG instability and nuclear pathology in Huntington disease mice (2020), published in eLife
  • Establishing and Sustaining a Culture of Collegial Collaboration (2020), published in PRIMUS
  • Subcellular localization of mutant P23H rhodopsin in an RFP fusion knockin mouse model of retinitis pigmentosa (2021), published in bioRxiv (Cold Spring Harbor Laboratory)

Frequent coauthors who have collaborated on multiple projects with Wilson include Michael A. Robichaux, Vy Nguyen, Fung Yee Chan, Lavanya Kailasam, and Feng He.

The primary publication venues for Wilson's work are:

  • Tissue Engineering Part C Methods
  • Disease Models & Mechanisms
  • eLife
  • PRIMUS
  • bioRxiv (Cold Spring Harbor Laboratory)

Wilson's research intersects clinical and molecular disciplines, notably addressing cardiovascular concerns during pregnancy and retinal disease mechanisms. Their studies contribute to understanding the underlying biological processes in these areas through molecular and cellular approaches.

Best Publications

  • Nonhomologous recombination in mammalian cells: role for short sequence homologies in the joining reaction.

    D B Roth;J H Wilson

  • Manipulating the mammalian genome by homologous recombination

    Karen M. Vasquez;Kathleen Marburger;Zsofia Intody;Zsofia Intody;John H. Wilson

  • Repair of site-specific double-strand breaks in a mammalian chromosome by homologous and illegitimate recombination.

    R G Sargent;M A Brenneman;J H Wilson

  • Mechanisms of nonhomologous recombination in mammalian cells.

    D B Roth;T N Porter;J H Wilson

  • Neural reprogramming in retinal degeneration.

    Robert E. Marc;Bryan W. Jones;James R. Anderson;Krista Kinard

  • DNA end joining becomes less efficient and more error-prone during cellular senescence

    Andrei Seluanov;David Mittelman;Olivia M. Pereira-Smith;John H. Wilson

  • Triplex-directed modification of genes and gene activity

    Karen M. Vasquez;John H. Wilson

  • R loops stimulate genetic instability of CTG·CAG repeats

    Yunfu Lin;Sharon Y. R. Dent;John H. Wilson;Robert D. Wells

  • Relative rates of homologous and nonhomologous recombination in transfected DNA

    David B. Roth;John H. Wilson

  • Transcription promotes contraction of CAG repeat tracts in human cells.

    Yunfu Lin;Vincent Dion;John H Wilson

  • Role of ERCC1 in removal of long non-homologous tails during targeted homologous recombination.

    Gerald M. Adair;Rhonda L. Rolig;Dana Moore-Faver;Marina Zabelshansky

  • How damaged is the biologically active subpopulation of transfected DNA

    C T Wake;T Gudewicz;T Porter;A White

  • Transcription-induced CAG repeat contraction in human cells is mediated in part by transcription-coupled nucleotide excision repair.

    Yunfu Lin;John H. Wilson

  • Role of the nucleotide excision repair gene ERCC1 in formation of recombination-dependent rearrangements in mammalian cells.

    R. Geoffrey Sargent;James L. Meservy;Brian D. Perkins;April E. Kilburn

  • Comparison of filler DNA at immune, nonimmune, and oncogenic rearrangements suggests multiple mechanisms of formation.

    D. B. Roth;Xiu-Bao Chang;J. H. Wilson

  • Gene targeting in normal and amplified cell lines.

    Hui Zheng;John H. Wilson

  • Zinc-finger directed double-strand breaks within CAG repeat tracts promote repeat instability in human cells

    David Mittelman;Christopher Moye;Jason Morton;Kristen Sykoudis

  • Insertion of a telomere repeat sequence into a mammalian gene causes chromosome instability

    April E. Kilburn;Martin J. Shea;R. Geoffrey Sargent;John H. Wilson

  • Targeted homologous recombination at the endogenous adenine phosphoribosyltransferase locus in Chinese hamster cells

    Gerald M. Adair;Rodney S. Nairn;John H. Wilson;Michael M. Seidman

  • Impaired photoreceptor protein transport and synaptic transmission in a mouse model of Bardet-Biedl syndrome.

    Muhammad M. Abd-El-Barr;Kristen Sykoudis;Sara Andrabi;Erica R. Eichers

Frequent Co-Authors

Theodore G. Wensel
Theodore G. Wensel Baylor College of Medicine
Karen M. Vasquez
Karen M. Vasquez The University of Texas at Austin
David Roth
David Roth University of Miami
Andrei Seluanov
Andrei Seluanov University of Rochester
Michael E. Talkowski
Michael E. Talkowski Harvard University
Vera Gorbunova
Vera Gorbunova University of Rochester
William W. Hauswirth
William W. Hauswirth University of Florida
Hui Zheng
Hui Zheng Baylor College of Medicine
Stephen J. Elledge
Stephen J. Elledge Harvard University
James R. Lupski
James R. Lupski Baylor College of Medicine

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