2014 - Fellow of the American Association for the Advancement of Science (AAAS)
Karen M. Vasquez focuses on DNA, DNA repair, Gene, Genetics and Nucleotide excision repair. Her DNA repair study incorporates themes from DNA damage and Homologous recombination. Her research in DNA damage intersects with topics in Etoposide, Cisplatin, DNA synthesis and Doxorubicin.
Her study involves Oligonucleotide, Locus, Reporter gene and Genome, a branch of Gene. Her work on Repeated sequence and Human genome as part of general Genome research is frequently linked to Comparative genomics, bridging the gap between disciplines. Her work deals with themes such as Replication protein A, Cell biology, Mutagenesis and DNA mismatch repair, which intersect with Nucleotide excision repair.
Karen M. Vasquez mainly focuses on DNA, Genetics, DNA repair, DNA damage and Gene. Her DNA study integrates concerns from other disciplines, such as Molecular biology, Genome, Computational biology and Mutagenesis. In her study, DNA ligase and Biophysics is inextricably linked to Base pair, which falls within the broad field of Molecular biology.
The study incorporates disciplines such as DNA supercoil, Replication protein A and Cell biology in addition to DNA repair. Her DNA damage research is multidisciplinary, incorporating perspectives in High-mobility group, Cancer research and Cisplatin. The various areas that Karen M. Vasquez examines in her Oligonucleotide study include Plasmid, Triple helix, Gene targeting, Transcription and Binding site.
DNA, DNA repair, Cancer research, Cancer and Computational biology are her primary areas of study. Her work is dedicated to discovering how DNA, Genome are connected with Human cancer, Oligonucleotide and DNA sequencing and other disciplines. Karen M. Vasquez combines subjects such as DNA damage and DNA replication with her study of DNA repair.
Her DNA damage research includes themes of Mutation and Mutagenesis. Her study in Computational biology is interdisciplinary in nature, drawing from both Molecular probe and Triple helix. Gene is the subject of her research, which falls under Genetics.
The scientist’s investigation covers issues in Cancer research, DNA, Biomarker, microRNA and Cancer. Her Angiogenesis study, which is part of a larger body of work in Cancer research, is frequently linked to Enos, bridging the gap between disciplines. Her work in the fields of DNA, such as Nucleotide excision repair and DNA repair, overlaps with other areas such as Stickleback.
Her Nucleotide excision repair study combines topics in areas such as Chromatin, High-mobility group, Cell biology and DNA mismatch repair. Her DNA repair study combines topics from a wide range of disciplines, such as Genome, Chromosomal translocation, Cancer Etiology and Nuclease. Her microRNA research incorporates elements of Druggability, Metastasis and Long non-coding RNA.
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Manipulating the mammalian genome by homologous recombination
Karen M. Vasquez;Kathleen Marburger;Zsofia Intody;Zsofia Intody;John H. Wilson.
Proceedings of the National Academy of Sciences of the United States of America (2001)
Triapine (3-aminopyridine-2-carboxaldehyde- thiosemicarbazone): A potent inhibitor of ribonucleotide reductase activity with broad spectrum antitumor activity
Rick Avery Finch;Mao Chin Liu;Susan P. Grill;William C. Rose.
Biochemical Pharmacology (2000)
Non-B DNA structure-induced genetic instability and evolution
Junhua Zhao;Albino Bacolla;Guliang Wang;Karen M. Vasquez.
Cellular and Molecular Life Sciences (2010)
Specific Mutations Induced by Triplex-Forming Oligonucleotides in Mice
Karen M. Vasquez;Latha Narayanan;Peter M. Glazer.
DNA triple helices: biological consequences and therapeutic potential.
Aklank Jain;Guliang Wang;Karen M. Vasquez.
Triplex-directed modification of genes and gene activity
Karen M. Vasquez;John H. Wilson.
Trends in Biochemical Sciences (1998)
Non-B DNA structure-induced genetic instability.
Guliang Wang;Karen M. Vasquez.
Mutation Research (2006)
High mobility group protein B1 enhances DNA repair and chromatin modification after DNA damage.
Sabine S. Lange;David L. Mitchell;Karen M. Vasquez.
Proceedings of the National Academy of Sciences of the United States of America (2008)
Naturally occurring H-DNA-forming sequences are mutagenic in mammalian cells
Guliang Wang;Karen M. Vasquez.
Proceedings of the National Academy of Sciences of the United States of America (2004)
Z-DNA-forming sequences generate large-scale deletions in mammalian cells
Guliang Wang;Laura A. Christensen;Karen M. Vasquez.
Proceedings of the National Academy of Sciences of the United States of America (2006)
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