D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Biology and Biochemistry D-index 44 Citations 8,581 104 World Ranking 13843 National Ranking 628

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Signal transduction
  • G protein-coupled receptor

Signal transduction, Beta-Arrestins, Cell biology, G protein-coupled receptor kinase and Protein kinase A are his primary areas of study. His Signal transduction research integrates issues from Receptor, Internalization and Neuroscience. His work in Beta-Arrestins addresses subjects such as Heterotrimeric G protein, which are connected to disciplines such as RHOA and Gq alpha subunit.

The G protein-coupled receptor kinase study combines topics in areas such as Arrestin, Molecular biology and 5-HT5A receptor. His research investigates the connection between Molecular biology and topics such as Beta adrenergic receptor kinase that intersect with issues in MAPK/ERK pathway. The various areas that Eric Reiter examines in his Protein kinase A study include Angiotensin II and Pertussis toxin.

His most cited work include:

  • β-Arrestin-dependent, G Protein-independent ERK1/2 Activation by the β2 Adrenergic Receptor (609 citations)
  • β-Arrestin-dependent, G Protein-independent ERK1/2 Activation by the β2 Adrenergic Receptor (609 citations)
  • GRKs and β-arrestins: roles in receptor silencing, trafficking and signaling (550 citations)

What are the main themes of his work throughout his whole career to date?

His primary areas of investigation include Cell biology, Receptor, Internal medicine, Endocrinology and G protein-coupled receptor. Signal transduction, Arrestin, G protein and Heterotrimeric G protein are the primary areas of interest in his Cell biology study. Eric Reiter has included themes like Molecular biology and Protein phosphorylation, Protein kinase A in his Signal transduction study.

His studies deal with areas such as Mutant and Northern blot as well as Internal medicine. His research in G protein-coupled receptor is mostly concerned with G protein-coupled receptor kinase. His G protein-coupled receptor kinase study combines topics in areas such as 5-HT5A receptor and Beta adrenergic receptor kinase.

He most often published in these fields:

  • Cell biology (90.11%)
  • Receptor (84.62%)
  • Internal medicine (61.54%)

What were the highlights of his more recent work (between 2017-2021)?

  • Cell biology (90.11%)
  • Receptor (84.62%)
  • Follicle-stimulating hormone receptor (21.98%)

In recent papers he was focusing on the following fields of study:

His main research concerns Cell biology, Receptor, Follicle-stimulating hormone receptor, Internal medicine and Endocrinology. His Signal transduction, G protein and G protein-coupled receptor study are his primary interests in Cell biology. His studies in Signal transduction integrate themes in fields like Autocrine signalling, Paracrine signalling and Protein kinase A.

His study in G protein-coupled receptor is interdisciplinary in nature, drawing from both Biophysics, Regulation of gene expression, Internalization and Gene regulatory network. Eric Reiter combines subjects such as Gene expression and Cellular differentiation with his study of Receptor. His work carried out in the field of Internal medicine brings together such families of science as Mutant and Protein kinase A signaling.

Between 2017 and 2021, his most popular works were:

  • Manifold roles of β-arrestins in GPCR signaling elucidated with siRNA and CRISPR/Cas9 (96 citations)
  • FSH Receptor Signaling: Complexity of Interactions and Signal Diversity. (31 citations)
  • FSH Receptor Signaling: Complexity of Interactions and Signal Diversity. (31 citations)

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

β-Arrestin-dependent, G Protein-independent ERK1/2 Activation by the β2 Adrenergic Receptor

Sudha K. Shenoy;Matthew T. Drake;Christopher D. Nelson;Daniel A. Houtz.
Journal of Biological Chemistry (2006)

802 Citations

GRKs and β-arrestins: roles in receptor silencing, trafficking and signaling

Eric Reiter;Robert J. Lefkowitz;Robert J. Lefkowitz.
Trends in Endocrinology and Metabolism (2006)

772 Citations

Molecular Mechanism of β-Arrestin-Biased Agonism at Seven-Transmembrane Receptors

Eric Reiter;Seungkirl Ahn;Arun K. Shukla;Robert J. Lefkowitz.
Annual Review of Pharmacology and Toxicology (2012)

581 Citations

Distinct β-arrestin- and G protein-dependent pathways for parathyroid hormone receptor-stimulated ERK1/2 activation

Diane Gesty-Palmer;Minyong Chen;Eric Reiter;Seungkirl Ahn.
Journal of Biological Chemistry (2006)

460 Citations

Functional antagonism of different G protein-coupled receptor kinases for β-arrestin-mediated angiotensin II receptor signaling

Jihee Kim;Seungkirl Ahn;Xiu Rong Ren;Erin J. Whalen.
Proceedings of the National Academy of Sciences of the United States of America (2005)

383 Citations

Different G protein-coupled receptor kinases govern G protein and β-arrestin-mediated signaling of V2 vasopressin receptor

Xiu-Rong Ren;Eric Reiter;Seungkirl Ahn;Jihee Kim.
Proceedings of the National Academy of Sciences of the United States of America (2005)

360 Citations

β-Arrestin 1 and Gαq/11 Coordinately Activate RhoA and Stress Fiber Formation following Receptor Stimulation

William G. Barnes;Eric Reiter;Eric Reiter;Jonathan D. Violin;Xiu-Rong Ren.
Journal of Biological Chemistry (2005)

239 Citations

The ERK-dependent signalling is stage-specifically modulated by FSH, during primary Sertoli cell maturation.

Pascale Crépieux;Sébastien Marion;Nadine Martinat;Véronique Fafeur.
Oncogene (2001)

214 Citations

A Phosphorylation Cluster of Five Serine and Threonine Residues in the C-Terminus of the Follicle-Stimulating Hormone Receptor Is Important for Desensitization But Not for β-Arrestin-Mediated ERK Activation

Elodie Kara;Pascale Crépieux;Christophe Gauthier;Nadine Martinat.
Molecular Endocrinology (2006)

187 Citations

β-Arrestin 2-dependent angiotensin II type 1A receptor-mediated pathway of chemotaxis

Dacia L. Hunton;William G. Barnes;Jihee Kim;Xiu-Rong Ren.
Molecular Pharmacology (2005)

142 Citations

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