What is he best known for?
The fields of study he is best known for:
- Gene
- Enzyme
- Signal transduction
His primary areas of study are Cell biology, Receptor, Biochemistry, Arrestin and G protein-coupled receptor.
Jeffrey L. Benovic interconnects Peptide sequence, Ubiquitin and Endocytosis in the investigation of issues within Cell biology.
He focuses mostly in the field of Receptor, narrowing it down to topics relating to Phosphorylation and, in certain cases, Wild type.
His Arrestin study combines topics from a wide range of disciplines, such as Biophysics, Chemokine receptor and Clathrin.
Within one scientific family, Jeffrey L. Benovic focuses on topics pertaining to Arrestin beta 2 under Arrestin beta 1, and may sometimes address concerns connected to Binding selectivity and Binding site.
The G protein-coupled receptor kinase study combines topics in areas such as Enzyme-linked receptor and Kinase.
His most cited work include:
- Beta-arrestin acts as a clathrin adaptor in endocytosis of the beta2-adrenergic receptor. (1183 citations)
- Cloning of the gene and cDNA for mammalian β -adrenergic receptor and homology with rhodopsin (1093 citations)
- Beta-arrestin: a protein that regulates beta-adrenergic receptor function (1009 citations)
What are the main themes of his work throughout his whole career to date?
His primary areas of investigation include Cell biology, Receptor, G protein-coupled receptor kinase, Biochemistry and Arrestin.
His Cell biology study frequently links to related topics such as Internalization.
His Receptor research integrates issues from Endocrinology and Intracellular.
His studies deal with areas such as 5-HT5A receptor, Molecular biology and Kinase, Protein kinase A as well as G protein-coupled receptor kinase.
His research in the fields of Beta adrenergic receptor kinase, Protein kinase C and MAP2K7 overlaps with other disciplines such as Tropomyosin receptor kinase C.
His research in Arrestin intersects with topics in Endocytosis, Clathrin and Signal transducing adaptor protein.
He most often published in these fields:
- Cell biology (53.16%)
- Receptor (44.61%)
- G protein-coupled receptor kinase (37.55%)
What were the highlights of his more recent work (between 2013-2021)?
- Cell biology (53.16%)
- G protein-coupled receptor (30.86%)
- Arrestin (31.97%)
In recent papers he was focusing on the following fields of study:
His scientific interests lie mostly in Cell biology, G protein-coupled receptor, Arrestin, Receptor and Signal transduction.
His Cell biology research includes themes of Molecular biology, β2 adrenergic receptor and Cell regulation.
He studies G protein-coupled receptor, focusing on G protein-coupled receptor kinase in particular.
His G protein-coupled receptor kinase study is concerned with the field of Biochemistry as a whole.
His Arrestin study incorporates themes from Endocytic cycle, Contractility and Signal transducing adaptor protein, Phosphorylation.
His study focuses on the intersection of Receptor and fields such as Pharmacology with connections in the field of Mediator.
Between 2013 and 2021, his most popular works were:
- Role of β-arrestins and arrestin domain-containing proteins in G protein-coupled receptor trafficking (129 citations)
- Structural and Functional Analysis of a β2-Adrenergic Receptor Complex with GRK5. (84 citations)
- β-Arrestins and G protein-coupled receptor trafficking. (76 citations)
In his most recent research, the most cited papers focused on:
- Gene
- Enzyme
- Signal transduction
His main research concerns Arrestin, G protein-coupled receptor, Cell biology, G protein-coupled receptor kinase and Signal transduction.
His Arrestin research is multidisciplinary, relying on both Caspase 3, Apoptosome and Phosphorylation.
As a part of the same scientific family, Jeffrey L. Benovic mostly works in the field of G protein-coupled receptor, focusing on GTPase-activating protein and, on occasion, Arrestin beta 2, Heterotrimeric G protein and Arrestin beta 1.
Jeffrey L. Benovic has included themes like Cytochrome c, Caspase, Programmed cell death, Intrinsic apoptosis and Molecular biology in his Cell biology study.
His G protein-coupled receptor kinase research entails a greater understanding of Biochemistry.
Jeffrey L. Benovic interconnects Agonist, Receptor and Pharmacology in the investigation of issues within Signal transduction.
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