What is she best known for?
The fields of study she is best known for:
- Signal transduction
- Phosphorylation
- G protein-coupled receptor
Her primary areas of investigation include Cell biology, Arrestin, Beta-Arrestins, Signal transduction and Receptor.
Her Cell biology research is multidisciplinary, incorporating perspectives in Endocytic cycle and Biochemistry.
Sudha K. Shenoy interconnects G protein-coupled receptor kinase and Protein kinase A in the investigation of issues within Beta-Arrestins.
Her research in Signal transduction is mostly concerned with Cell signaling.
She focuses mostly in the field of Receptor, narrowing it down to matters related to Endocrinology and, in some cases, MAPK/ERK pathway and Pharmacology.
Her G protein-coupled receptor research includes elements of 5-HT5A receptor, Ubiquitin ligase, F-box protein and Mdm2.
Her most cited work include:
- Transduction of receptor signals by beta-arrestins. (1417 citations)
- β-Arrestins and Cell Signaling (1132 citations)
- Regulation of receptor fate by ubiquitination of activated beta 2-adrenergic receptor and beta-arrestin. (731 citations)
What are the main themes of her work throughout her whole career to date?
Her main research concerns Cell biology, Receptor, Arrestin, G protein-coupled receptor and Signal transduction.
Her Cell biology research integrates issues from Endocytic cycle and Ubiquitin ligase.
Her Receptor study incorporates themes from Endocrinology, Kinase and Phosphorylation.
Her study on Arrestin also encompasses disciplines like
- Endosome which intersects with area such as Agonist,
- MAPK/ERK pathway that intertwine with fields like Pharmacology and Carvedilol.
Her G protein-coupled receptor research is multidisciplinary, relying on both F-box protein, G protein and Deubiquitination.
The Signal transduction study combines topics in areas such as Homologous desensitization and Protein kinase A.
She most often published in these fields:
- Cell biology (74.16%)
- Receptor (44.94%)
- Arrestin (46.07%)
What were the highlights of her more recent work (between 2016-2020)?
- Cell biology (74.16%)
- Receptor (44.94%)
- G protein-coupled receptor (42.70%)
In recent papers she was focusing on the following fields of study:
Cell biology, Receptor, G protein-coupled receptor, Arrestin and Signal transducing adaptor protein are her primary areas of study.
Her specific area of interest is Cell biology, where Sudha K. Shenoy studies Signal transduction.
In general Signal transduction study, her work on Beta-Arrestins and G protein often relates to the realm of Small interfering RNA and Transgene, thereby connecting several areas of interest.
Her Receptor research incorporates themes from Kinase, Protein kinase A, Inflammation, Ubiquitin ligase and Contractility.
In general Arrestin, her work in Arrestin beta 2 is often linked to CC chemokine receptors and CCR1 linking many areas of study.
Her Signal transducing adaptor protein study combines topics in areas such as Intracellular, Clathrin and Protein–protein interaction.
Between 2016 and 2020, her most popular works were:
- GPCR desensitization: Acute and prolonged phases (119 citations)
- GPCR desensitization: Acute and prolonged phases (119 citations)
- Manifold roles of β-arrestins in GPCR signaling elucidated with siRNA and CRISPR/Cas9 (96 citations)
In her most recent research, the most cited papers focused on:
- Signal transduction
- Phosphorylation
- G protein-coupled receptor
Her primary areas of study are Signal transduction, G protein-coupled receptor, Cell biology, Beta-Arrestins and Arrestin.
Her work in Cell biology is not limited to one particular discipline; it also encompasses Internalization.
Internalization is a subfield of Receptor that Sudha K. Shenoy tackles.
Her work deals with themes such as Homologous desensitization, Desensitization and Signal transducing adaptor protein, which intersect with G protein-coupled receptor kinase.
Sudha K. Shenoy has included themes like G Protein-Coupled Receptor Signaling and GTPase-activating protein in her Heterotrimeric G protein study.
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