D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Molecular Biology D-index 53 Citations 8,418 130 World Ranking 1674 National Ranking 61

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • DNA
  • Enzyme

His primary areas of investigation include DNA repair, Molecular biology, DNA repair protein XRCC4, DNA and DNA damage. Many of his research projects under DNA repair are closely connected to Ku80 with Ku80, tying the diverse disciplines of science together. Bernard Salles performs integrative study on Molecular biology and Philadelphia chromosome in his works.

Bernard Salles regularly links together related areas like DNA ligase in his DNA repair protein XRCC4 studies. His work on Chromatin and G-quadruplex as part of general DNA study is frequently linked to Telomere-binding protein, bridging the gap between disciplines. His work carried out in the field of DNA damage brings together such families of science as DNA supercoil, DNA replication, Cell aging and Telomere.

His most cited work include:

  • Cancer-Associated Adipocytes Exhibit an Activated Phenotype and Contribute to Breast Cancer Invasion (572 citations)
  • Involvement of Poly(ADP-ribose) Polymerase-1 and XRCC1/DNA Ligase III in an Alternative Route for DNA Double-strand Breaks Rejoining (541 citations)
  • Loss of ATM positively regulates the expression of hypoxia inducible factor 1 (HIF-1) through oxidative stress: Role in the physiopathology of the disease. (169 citations)

What are the main themes of his work throughout his whole career to date?

Bernard Salles mainly focuses on Molecular biology, DNA repair, DNA, DNA damage and Nucleotide excision repair. Bernard Salles carries out multidisciplinary research, doing studies in Molecular biology and Ku80. His research in DNA repair intersects with topics in DNA ligase, DNA replication and Cell biology.

Bernard Salles works mostly in the field of DNA, limiting it down to topics relating to Escherichia coli and, in certain cases, Mutant, as a part of the same area of interest. His studies in DNA damage integrate themes in fields like Chromatin, Histone and Mutagenesis. His study on Excinuclease is often connected to Replication protein A as part of broader study in Nucleotide excision repair.

He most often published in these fields:

  • Molecular biology (70.15%)
  • DNA repair (45.52%)
  • DNA (37.31%)

What were the highlights of his more recent work (between 2010-2019)?

  • Cell biology (23.13%)
  • Molecular biology (70.15%)
  • DNA damage (32.09%)

In recent papers he was focusing on the following fields of study:

Bernard Salles mainly investigates Cell biology, Molecular biology, DNA damage, DNA repair and Non-homologous end joining. His work on Kinase activity and Protein kinase A is typically connected to Ataxia Telangiectasia Mutated Proteins as part of general Cell biology study, connecting several disciplines of science. His study deals with a combination of Molecular biology and Ku80.

His DNA damage research is multidisciplinary, incorporating elements of Chromatin and Toxicology. His DNA repair study is concerned with DNA in general. His research integrates issues of DNA ligase and LIG4 in his study of DNA-PKcs.

Between 2010 and 2019, his most popular works were:

  • Cancer-Associated Adipocytes Exhibit an Activated Phenotype and Contribute to Breast Cancer Invasion (572 citations)
  • Ionizing-radiation induced DNA double-strand breaks: a direct and indirect lighting up. (139 citations)
  • DNA damage triggers SAF-A and RNA biogenesis factors exclusion from chromatin coupled to R-loops removal (106 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • DNA
  • Enzyme

His primary scientific interests are in Molecular biology, Chromatin, Autophosphorylation, Ku80 and DNA-PKcs. His Chromatin research integrates issues from DNA damage and DNA End-Joining Repair. His work deals with themes such as Transcription factor and Cell biology, which intersect with DNA damage.

His Autophosphorylation research incorporates elements of Histone, DNA ligase, LIG4 and Acetylation. His DNA-PKcs study deals with the bigger picture of DNA repair. His work on Homologous recombination as part of general DNA study is frequently linked to DNA Single Strand Break, therefore connecting diverse disciplines of science.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Cancer-Associated Adipocytes Exhibit an Activated Phenotype and Contribute to Breast Cancer Invasion

Béatrice Dirat;Ludivine Bochet;Marta Dabek;Danièle Daviaud.
Cancer Research (2011)

877 Citations

Involvement of Poly(ADP-ribose) Polymerase-1 and XRCC1/DNA Ligase III in an Alternative Route for DNA Double-strand Breaks Rejoining

Marc Audebert;Bernard Salles;Patrick Calsou.
Journal of Biological Chemistry (2004)

827 Citations

Loss of ATM positively regulates the expression of hypoxia inducible factor 1 (HIF-1) through oxidative stress: Role in the physiopathology of the disease.

Marielle Ousset;Fanny Bouquet;Frédérique Fallone;Denis Biard.
Cell Cycle (2010)

273 Citations

Ionizing-radiation induced DNA double-strand breaks: a direct and indirect lighting up.

Julien Vignard;Gladys Mirey;Bernard Salles.
Radiotherapy and Oncology (2013)

266 Citations

A G-quadruplex structure within the 5′-UTR of TRF2 mRNA represses translation in human cells

Dennis Gomez;Aurore Guédin;Jean-Louis Mergny;Bernard Salles.
Nucleic Acids Research (2010)

186 Citations

TRF2 and Apollo Cooperate with Topoisomerase 2α to Protect Human Telomeres from Replicative Damage

Jing Ye;Christelle Lenain;Serge Bauwens;Angela Rizzo.
Cell (2010)

184 Citations

DNA-dependent protein kinase and XRCC4-DNA ligase IV mobilization in the cell in response to DNA double strand breaks.

Jérôme Drouet;Christine Delteil;Jacques Lefrançois;Patrick Concannon.
Journal of Biological Chemistry (2005)

179 Citations

Coordinated assembly of Ku and p460 subunits of the DNA-dependent protein kinase on DNA ends is necessary for XRCC4-ligase IV recruitment.

Patrick Calsou;Christine Delteil;Philippe Frit;Jérôme Drouet.
Journal of Molecular Biology (2003)

173 Citations

The Loss of γH2AX Signal is a Marker of DNA Double Strand Breaks Repair Only at Low Levels of DNA Damage

Fanny Bouquet;Catherine Muller;Bernard Salles.
Cell Cycle (2006)

164 Citations

DNA damage triggers SAF-A and RNA biogenesis factors exclusion from chromatin coupled to R-loops removal

Sébastien Britton;Emma Dernoncourt;Christine Delteil;Carine Froment.
Nucleic Acids Research (2014)

154 Citations

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