World's Best Scientists 2026 revealed!

D-Index & Metrics

Biology and Biochemistry

D-Index
59
Citations
12440
World Ranking
12539
National Ranking
5367

Tomasz Skorski publication distribution in Biology and Biochemistry in 2026

The chart shows the distribution of publications by all Research.com ranked scientists in the field of Biology and Biochemistry in 2026. The highlighted bar marks where Tomasz Skorski sits on this spectrum.

47–56 publications: 8 scientists 57–66 publications: 35 scientists 67–76 publications: 106 scientists 77–86 publications: 231 scientists 87–96 publications: 414 scientists 97–106 publications: 546 scientists 107–116 publications: 704 scientists 117–126 publications: 849 scientists 127–136 publications: 980 scientists 137–146 publications: 942 scientists 147–156 publications: 969 scientists 157–166 publications: 950 scientists 167–176 publications: 951 scientists 177–186 publications: 915 scientists 187–196 publications: 787 scientists 197–206 publications: 841 scientists 207–216 publications: 735 scientists 217–226 publications: 709 scientists 227–236 publications: 651 scientists 237–246 publications: 605 scientists 247–256 publications: 510 scientists 257–266 publications: 524 scientists 267–276 publications: 434 scientists 277–286 publications: 418 scientists 287–296 publications: 350 scientists 297–306 publications: 363 scientists 307–316 publications: 315 scientists 317–326 publications: 296 scientists 327–336 publications: 261 scientists 337–346 publications: 240 scientists 347–356 publications: 219 scientists 357–366 publications: 197 scientists 367–376 publications: 154 scientists 377–386 publications: 161 scientists 387–396 publications: 155 scientists 397–406 publications: 145 scientists 407–416 publications: 124 scientists 417–426 publications: 112 scientists 427–436 publications: 132 scientists 437–446 publications: 116 scientists 447–456 publications: 99 scientists 457–466 publications: 81 scientists 467–476 publications: 91 scientists 477–486 publications: 80 scientists 487–496 publications: 80 scientists 497–506 publications: 60 scientists 507–516 publications: 36 scientists 517–526 publications: 46 scientists 527–536 publications: 54 scientists 537–546 publications: 44 scientists 547–556 publications: 43 scientists 557–566 publications: 43 scientists 567–576 publications: 42 scientists 577–586 publications: 25 scientists 587–596 publications: 34 scientists 597–606 publications: 23 scientists 607–616 publications: 33 scientists 617–626 publications: 31 scientists 627–636 publications: 27 scientists 637–646 publications: 25 scientists 647–656 publications: 28 scientists 657–666 publications: 34 scientists 667–676 publications: 18 scientists 677–686 publications: 16 scientists 687–696 publications: 10 scientists 697–706 publications: 12 scientists 707–716 publications: 21 scientists 717–726 publications: 12 scientists 727–736 publications: 12 scientists 737–746 publications: 10 scientists 747–756 publications: 7 scientists 757–766 publications: 13 scientists 767–776 publications: 15 scientists 777–786 publications: 13 scientists 787–796 publications: 9 scientists 797–806 publications: 9 scientists 807–816 publications: 7 scientists 817–826 publications: 4 scientists 827–836 publications: 9 scientists 837–846 publications: 7 scientists 847–856 publications: 3 scientists 857–866 publications: 5 scientists 867–876 publications: 5 scientists 877–886 publications: 11 scientists 887–896 publications: 3 scientists 897–906 publications: 4 scientists 907–916 publications: 7 scientists 917–926 publications: 5 scientists 927–936 publications: 6 scientists 937–946 publications: 6 scientists 947–956 publications: 3 scientists 957–966 publications: 7 scientists 967–976 publications: 2 scientists 977–986 publications: 2 scientists 987–996 publications: 1 scientists 997–1,006 publications: 5 scientists 1,007–1,016 publications: 2 scientists 1,017–1,026 publications: 2 scientists 1,027 publications: 1 scientists 1,028+ publications: 100 scientists
47 publications 1,028+

This scientist: 218 publications — 57th percentile

57% of scientists in this discipline score the same or lower.

The last bar groups every scientist with 1,028 publications or more.

Tomasz Skorski D-index placement in Biology and Biochemistry in 2026

The chart shows the D-index (discipline H-index) distribution of Biology and Biochemistry scientists ranked by Research.com in 2026. The highlighted bar marks where Tomasz Skorski sits on this spectrum.

40–41 D-Index: 80 scientists 42–43 D-Index: 183 scientists 44–45 D-Index: 317 scientists 46–47 D-Index: 504 scientists 48–49 D-Index: 718 scientists 50–51 D-Index: 900 scientists 52–53 D-Index: 1,026 scientists 54–55 D-Index: 1,150 scientists 56–57 D-Index: 1,236 scientists 58–59 D-Index: 1,253 scientists 60–61 D-Index: 1,163 scientists 62–63 D-Index: 1,131 scientists 64–65 D-Index: 1,032 scientists 66–67 D-Index: 897 scientists 68–69 D-Index: 814 scientists 70–71 D-Index: 715 scientists 72–73 D-Index: 709 scientists 74–75 D-Index: 596 scientists 76–77 D-Index: 512 scientists 78–79 D-Index: 473 scientists 80–81 D-Index: 412 scientists 82–83 D-Index: 373 scientists 84–85 D-Index: 358 scientists 86–87 D-Index: 285 scientists 88–89 D-Index: 273 scientists 90–91 D-Index: 227 scientists 92–93 D-Index: 208 scientists 94–95 D-Index: 193 scientists 96–97 D-Index: 153 scientists 98–99 D-Index: 157 scientists 100–101 D-Index: 148 scientists 102–103 D-Index: 120 scientists 104–105 D-Index: 113 scientists 106–107 D-Index: 100 scientists 108–109 D-Index: 86 scientists 110–111 D-Index: 67 scientists 112–113 D-Index: 72 scientists 114–115 D-Index: 73 scientists 116–117 D-Index: 64 scientists 118–119 D-Index: 53 scientists 120–121 D-Index: 60 scientists 122–123 D-Index: 54 scientists 124–125 D-Index: 43 scientists 126–127 D-Index: 38 scientists 128–129 D-Index: 49 scientists 130–131 D-Index: 26 scientists 132–133 D-Index: 18 scientists 134–135 D-Index: 23 scientists 136–137 D-Index: 32 scientists 138–139 D-Index: 32 scientists 140–141 D-Index: 27 scientists 142–143 D-Index: 19 scientists 144–145 D-Index: 22 scientists 146–147 D-Index: 12 scientists 148–149 D-Index: 16 scientists 150–151 D-Index: 14 scientists 152–153 D-Index: 10 scientists 154–155 D-Index: 13 scientists 156–157 D-Index: 10 scientists 158–159 D-Index: 7 scientists 160–161 D-Index: 9 scientists 162–163 D-Index: 13 scientists 164–165 D-Index: 4 scientists 166 D-Index: 4 scientists 167+ D-Index: 98 scientists
40 D-Index 167+

This scientist: 59 D-Index — 38th percentile

38% of scientists in this discipline score the same or lower.

The last bar groups every scientist with 167 D-Index or more.

Overview

Tomasz Skorski is affiliated with Temple University in the United States and has contributed extensively to the fields of biochemistry, genetics, molecular biology, and medicine. Their research centers on molecular biology and oncology, with significant work in hematology, genetics, and cancer research. The scientist's main research topics include DNA repair mechanisms, PARP inhibition in cancer therapy, acute myeloid leukemia research, CRISPR and genetic engineering, chronic myeloid leukemia treatments, epigenetics and DNA methylation, and cancer therapeutics and mechanisms.

Skorski's notable recent publications include:

  • Discovery of a small-molecule inhibitor that traps Polθ on DNA and synergizes with PARP inhibitors, 2024, Nature Communications
  • 4-1BBL-containing leukemic extracellular vesicles promote immunosuppressive effector regulatory T cells, 2022, Blood Advances
  • ATF3 coordinates serine and nucleotide metabolism to drive cell cycle progression in acute myeloid leukemia, 2021, Molecular Cell
  • TET2 and DNMT3A Mutations Exert Divergent Effects on DNA Repair and Sensitivity of Leukemia Cells to PARP Inhibitors, 2021, Cancer Research
  • TGFβR-SMAD3 Signaling Induces Resistance to PARP Inhibitors in the Bone Marrow Microenvironment, 2020, Cell Reports

The venues where Skorski frequently publishes include:

  • Blood
  • Cancer Research
  • Nature Communications
  • Leukemia
  • bioRxiv (Cold Spring Harbor Laboratory)

Frequent collaborators in Skorski's research are:

  • Monika M. Toma
  • Margaret Nieborowska-Skorska
  • Richard T. Pomerantz
  • Umeshkumar Vekariya
  • Katherine Sullivan-Reed

The scientific contributions of Tomasz Skorski are situated primarily at the interface of molecular mechanisms underlying leukemia and cancer therapeutics, focusing notably on the repair pathways of DNA and the effects of mutations relevant to therapy resistance. Their work covers both fundamental aspects of cell cycle regulation in leukemia cells and applied cancer research investigating the sensitivity and resistance mechanisms to PARP inhibitors.

Best Publications

  • Transformation of hematopoietic cells by BCR/ABL requires activation of a PI-3k/Akt-dependent pathway

    Tomasz Skorski;Alfonso Bellacosa;Margaret Nieborowska‐Skorska;Miroslaw Majewski

  • Selective inhibition of leukemia cell proliferation by BCR-ABL antisense oligodeoxynucleotides

    Cezary Szczylik;Tomasz Skorski;Nicholas C. Nicolaides;Livia Manzella

  • Chronic myeloid leukemia: mechanisms of blastic transformation

    Danilo Perrotti;Catriona Jamieson;John Goldman;Tomasz Skorski

  • Signal Transducer and Activator of Transcription (STAT)5 Activation by BCR/ABL Is Dependent on Intact Src Homology (SH)3 and SH2 Domains of BCR/ABL and Is Required for Leukemogenesis

    Malgorzata Nieborowska-Skorska;Mariusz A. Wasik;Artur Slupianek;Paolo Salomoni

  • Multilevel Dysregulation of STAT3 Activation in Anaplastic Lymphoma Kinase-Positive T/Null-Cell Lymphoma

    Qian Zhang;Puthryaveett N. Raghunath;Liquan Xue;Miroslaw Majewski

  • BCR/ABL regulates mammalian RecA homologs, resulting in drug resistance.

    Artur Slupianek;Christoph Schmutte;Gregory Tombline;Malgorzata Nieborowska-Skorska

  • BCR/ABL kinase induces self-mutagenesis via reactive oxygen species to encode imatinib resistance

    Mateusz Koptyra;Rafal Falinski;Rafal Falinski;Rafal Falinski;Michal O. Nowicki;Michal O. Nowicki;Michal O. Nowicki;Tomasz Stoklosa;Tomasz Stoklosa;Tomasz Stoklosa

  • Role of Phosphatidylinositol 3-Kinase-Akt Pathway in Nucleophosmin/Anaplastic Lymphoma Kinase-mediated Lymphomagenesis

    Artur Slupianek;Margaret Nieborowska-Skorska;Grazyna Hoser;Andrea Morrione

  • BCR/ABL oncogenic kinase promotes unfaithful repair of the reactive oxygen species-dependent DNA double-strand breaks

    Michal O. Nowicki;Michal O. Nowicki;Rafal Falinski;Rafal Falinski;Mateusz Koptyra;Mateusz Koptyra;Artur Slupianek;Artur Slupianek

  • De novo decorin gene expression suppresses the malignant phenotype in human colon cancer cells

    Manoranjan Santra;Tomasz Skorski;Bruno Calabretta;Edmund C. Lattime

  • The Src family kinase Hck couples BCR/ABL to STAT5 activation in myeloid leukemia cells

    Agata Klejman;Steven J. Schreiner;Malgorzata Nieborowska‐Skorska;Artur Slupianek

  • Fusion tyrosine kinases induce drug resistance by stimulation of homology-dependent recombination repair, prolongation of G(2)/M phase, and protection from apoptosis.

    Artur Slupianek;Grazyna Hoser;Ireneusz Majsterek;Agnieszka Bronisz

  • Suppression of Philadelphia1 leukemia cell growth in mice by BCR-ABL antisense oligodeoxynucleotide

    Tomasz Skorski;Malgorzata Nieborowska-Skorska;Nicholas C. Nicolaides;Cezary Szczylik

  • Rac2-MRC-cIII–generated ROS cause genomic instability in chronic myeloid leukemia stem cells and primitive progenitors

    Margaret Nieborowska-Skorska;Piotr K. Kopinski;Regina Ray;Grazyna Hoser

  • Phosphatidylinositol-3 kinase inhibitors enhance the anti-leukemia effect of STI571.

    Agata Klejman;Lori Rushen;Andrea Morrione;Artur Slupianek

  • Oncogenic tyrosine kinases and the dna-damage response

    Tomasz Skorski

  • BCR/ABL regulates response to DNA damage: the role in resistance to genotoxic treatment and in genomic instability.

    Tomasz Skorski

  • Activation of mitochondrial Raf-1 is involved in the antiapoptotic effects of Akt.

    Miroslaw Majewski;Malgorzata Nieborowska-Skorska;Paolo Salomoni;Artur Slupianek

  • TLS/FUS, a pro‐oncogene involved in multiple chromosomal translocations, is a novel regulator of BCR/ABL‐mediated leukemogenesis

    Danilo Perrotti;Silvia Bonatti;Rossana Trotta;Robert Martinez

  • BCR/ABL kinase induces self-mutagenesis via reactive oxygen species to encode imatinib resistance. Commentary

    Ravi Bhatia;Mateusz Koptyra;Rafal Falinski;Michal O. Nowicki

Frequent Co-Authors

Janusz Blasiak
Janusz Blasiak University of Łódź
Mariusz A. Wasik
Mariusz A. Wasik University of Pennsylvania
Bruno Calabretta
Bruno Calabretta Thomas Jefferson University
Peter Valent
Peter Valent Medical University of Vienna
Krzysztof Reiss
Krzysztof Reiss Louisiana State University Health Sciences Center New Orleans
Ravi Bhatia
Ravi Bhatia University of Alabama at Birmingham
Paolo Salomoni
Paolo Salomoni German Center for Neurodegenerative Diseases
Markus Müschen
Markus Müschen Yale University
Renato V. Iozzo
Renato V. Iozzo Thomas Jefferson University
Richard Fishel
Richard Fishel The Ohio State University

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