Ravi Bhatia mainly focuses on Cancer research, Chronic myelogenous leukemia, Leukemia, Stem cell and Imatinib mesylate. The various areas that Ravi Bhatia examines in his Cancer research study include Haematopoiesis, Progenitor cell, Tyrosine kinase, Kinase activity and Bone marrow. His Chronic myelogenous leukemia research is multidisciplinary, incorporating elements of K562 cells and Philadelphia chromosome.
His research integrates issues of Myeloid, Myeloid leukemia and Transplantation in his study of Leukemia. He does research in Stem cell, focusing on Cancer stem cell specifically. His Imatinib mesylate study integrates concerns from other disciplines, such as breakpoint cluster region and ABL.
His primary areas of study are Cancer research, Stem cell, Progenitor cell, Immunology and Leukemia. The concepts of his Cancer research study are interwoven with issues in Chronic myelogenous leukemia and Tyrosine kinase, ABL. His work investigates the relationship between Chronic myelogenous leukemia and topics such as Integrin that intersect with problems in Cell adhesion and Cell adhesion molecule.
His Stem cell study combines topics in areas such as Myeloid and Bone marrow. His biological study spans a wide range of topics, including Cancer cell, Molecular biology, Apoptosis and CD38. His Leukemia study is related to the wider topic of Internal medicine.
Ravi Bhatia mostly deals with Cancer research, Stem cell, Internal medicine, Leukemia and Myeloid leukemia. His research integrates issues of Cell growth, Progenitor cell, Tyrosine kinase, ABL and Chronic myelogenous leukemia in his study of Cancer research. His research in Stem cell intersects with topics in Immunology and Bone marrow.
His work in Internal medicine tackles topics such as Oncology which are related to areas like Philadelphia chromosome, Discontinuation, Polycythemia vera, Risk stratification and Essential thrombocythemia. His study in the field of Myelogenous also crosses realms of PARP1. His Myeloid leukemia study combines topics from a wide range of disciplines, such as Hematopoietic stem cell transplantation, DNA methylation, CXCR4, CD33 and Drug resistance.
Leukemia, Cancer research, Stem cell, Myeloid leukemia and Internal medicine are his primary areas of study. His study with Leukemia involves better knowledge in Immunology. The Cancer research study combines topics in areas such as Haematopoiesis, Bone marrow, Tyrosine-kinase inhibitor and DNA repair.
His study in Stem cell is interdisciplinary in nature, drawing from both Chronic myelogenous leukemia and Tyrosine kinase, Nilotinib. His Internal medicine research incorporates themes from Philadelphia chromosome and Oncology. Ravi Bhatia studied Hematopoietic stem cell and breakpoint cluster region that intersect with ABL.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Nilotinib (formerly AMN107), a highly selective BCR-ABL tyrosine kinase inhibitor, is effective in patients with Philadelphia chromosome–positive chronic myelogenous leukemia in chronic phase following imatinib resistance and intolerance
Hagop M. Kantarjian;Francis Giles;Norbert Gattermann;Kapil Bhalla.
Blood (2007)
Persistence of malignant hematopoietic progenitors in chronic myelogenous leukemia patients in complete cytogenetic remission following imatinib mesylate treatment.
Ravi Bhatia;Melissa Holtz;Ning Niu;Rachel Gray.
Blood (2003)
miR-328 Functions as an RNA Decoy to Modulate hnRNP E2 Regulation of mRNA Translation in Leukemic Blasts
Anna M. Eiring;Jason G. Harb;Paolo Neviani;Christopher Garton.
Cell (2010)
Activation of p53 by SIRT1 inhibition enhances elimination of CML leukemia stem cells in combination with imatinib
Ling Li;Lisheng Wang;Liang Li;Zhiqiang Wang.
Cancer Cell (2012)
Chronic myeloid leukemia stem cells are not dependent on Bcr-Abl kinase activity for their survival
Ashley Hamilton;G. Vignir Helgason;Mirle Schemionek;Bin Zhang.
Blood (2012)
Imatinib mesylate (STI571) inhibits growth of primitive malignant progenitors in chronic myelogenous leukemia through reversal of abnormally increased proliferation.
Melissa S. Holtz;Marilyn L. Slovak;Feiyu Zhang;Charles L. Sawyers.
Blood (2002)
Altered Microenvironmental Regulation of Leukemic and Normal Stem Cells in Chronic Myelogenous Leukemia
Bin Zhang;Yin Wei Ho;Qin Huang;Takahiro Maeda.
Cancer Cell (2012)
Solid Cancers After Bone Marrow Transplantation
Smita Bhatia;Andrew D. Louie;Ravi Bhatia;Margaret R. O’Donnell.
Journal of Clinical Oncology (2001)
Stem cell quiescence.
Ling Li;Ravi Bhatia.
Clinical Cancer Research (2011)
Predictors of therapy-related leukemia and myelodysplasia following autologous transplantation for lymphoma: an assessment of risk factors.
Amrita Krishnan;Smita Bhatia;Marilyn L. Slovak;Daniel A. Arber.
Blood (2000)
If you think any of the details on this page are incorrect, let us know.
We appreciate your kind effort to assist us to improve this page, it would be helpful providing us with as much detail as possible in the text box below:
City Of Hope National Medical Center
University of Alabama at Birmingham
University of Glasgow
KU Leuven
Fred Hutchinson Cancer Research Center
Augusta University
University of Minnesota
University of Minnesota
Janssen (Belgium)
The University of Texas MD Anderson Cancer Center
Netflix (United States)
Polytechnic University of Milan
Boston Dynamics (United States)
Queensland University of Technology
Philipp University of Marburg
University of Cape Town
University of Bristol
University of New England
University of Helsinki
University of Arizona
Victoria University
Max Planck Society
University of Montana
University of Rome Tor Vergata
University of Münster
James Madison University