What is he best known for?
The fields of study he is best known for:
Janusz Blasiak mostly deals with DNA damage, DNA repair, Comet assay, Cancer research and Molecular biology.
His DNA damage study combines topics in areas such as Reactive oxygen species and Mutagenesis.
Janusz Blasiak has researched DNA repair in several fields, including Oxidative stress and Cisplatin.
His Comet assay research includes elements of Cancer cell, DNA glycosylase, Melatonin and Genotoxicity.
His Cancer research research integrates issues from Cancer, Breast cancer, ABL, Genotype and breakpoint cluster region.
His biological study spans a wide range of topics, including Imatinib mesylate, Tyrosine kinase, Genome instability, Leukemia and Kinase.
His most cited work include:
- BCR/ABL kinase induces self-mutagenesis via reactive oxygen species to encode imatinib resistance (250 citations)
- BCR/ABL kinase induces self-mutagenesis via reactive oxygen species to encode imatinib resistance (250 citations)
- BCR/ABL kinase induces self-mutagenesis via reactive oxygen species to encode imatinib resistance (250 citations)
What are the main themes of his work throughout his whole career to date?
His scientific interests lie mostly in DNA damage, DNA repair, Cancer research, Molecular biology and Comet assay.
His DNA damage study incorporates themes from Oxidative stress and K562 cells.
Janusz Blasiak interconnects XRCC1, Genome instability and Homologous recombination in the investigation of issues within DNA repair.
His Cancer research study combines topics from a wide range of disciplines, such as Cancer cell, Cancer, breakpoint cluster region and ABL.
The Molecular biology study which covers Genotype that intersects with Endocrinology.
His study looks at the intersection of Comet assay and topics like Antioxidant with Pharmacology.
He most often published in these fields:
- DNA damage (38.01%)
- DNA repair (30.70%)
- Cancer research (30.41%)
What were the highlights of his more recent work (between 2015-2021)?
- Autophagy (6.73%)
- Cell biology (10.53%)
- Cancer research (30.41%)
In recent papers he was focusing on the following fields of study:
His primary areas of investigation include Autophagy, Cell biology, Cancer research, DNA damage and Retinal pigment epithelium.
His work deals with themes such as Oxidative stress and Mitophagy, which intersect with Cell biology.
His study in Cancer research is interdisciplinary in nature, drawing from both Cancer, Breast cancer, microRNA, Cancer epigenetics and Vitamin D and neurology.
His DNA damage research is under the purview of Biochemistry.
His Retinal pigment epithelium research is multidisciplinary, incorporating perspectives in Inflammasome, Senescence and Pathogenesis, Pathology.
His studies in DNA repair integrate themes in fields like Mutagenesis and Homologous recombination.
Between 2015 and 2021, his most popular works were:
- Inflammation and its role in age-related macular degeneration (238 citations)
- Autophagy regulates death of retinal pigment epithelium cells in age-related macular degeneration (80 citations)
- Loss of NRF-2 and PGC-1α genes leads to retinal pigment epithelium damage resembling dry age-related macular degeneration. (48 citations)
In his most recent research, the most cited papers focused on:
His main research concerns Autophagy, Cell biology, Retinal pigment epithelium, Oxidative stress and DNA damage.
His study in Autophagy is interdisciplinary in nature, drawing from both Multicellular organism, Cancer research and Programmed cell death.
His studies in Cell biology integrate themes in fields like Lipofuscin, Cellular detoxification, Sequestosome 1 and Mitophagy.
Janusz Blasiak studied Retinal pigment epithelium and Pathology that intersect with Cell division, Immunology and Chemokine.
The Oxidative stress study combines topics in areas such as STAT3, Signal transduction, Janus kinase 2, Senescence and Mitochondrion.
His study on DNA damage is covered under Biochemistry.
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