D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Biology and Biochemistry D-index 48 Citations 19,211 143 World Ranking 13866 National Ranking 98

Overview

What is she best known for?

The fields of study she is best known for:

  • Enzyme
  • Apoptosis
  • Cancer

Anu Kauppinen focuses on Cell biology, Inflammation, NF-κB, Signal transduction and Oxidative stress. As a member of one scientific family, Anu Kauppinen mostly works in the field of Cell biology, focusing on Autophagy and, on occasion, Mitochondrion and Reactive oxygen species. Her work in Inflammation addresses issues such as Pathology, which are connected to fields such as Protein folding, Homeostasis and NALP3.

In her work, AMP-activated protein kinase is strongly intertwined with Cancer research, which is a subfield of NF-κB. Within one scientific family, Anu Kauppinen focuses on topics pertaining to Innate immune system under Signal transduction, and may sometimes address concerns connected to Immunity and Acquired immune system. Her work is dedicated to discovering how Oxidative stress, Inflammasome are connected with Macular degeneration and other disciplines.

Her most cited work include:

  • Antagonistic crosstalk between NF-κB and SIRT1 in the regulation of inflammation and metabolic disorders. (421 citations)
  • Activation of innate immunity system during aging: NF-kB signaling is the molecular culprit of inflamm-aging. (341 citations)
  • Emerging role of NF-κB signaling in the induction of senescence-associated secretory phenotype (SASP). (295 citations)

What are the main themes of her work throughout her whole career to date?

Her primary areas of study are Cell biology, Inflammation, Autophagy, Inflammasome and Immunology. Her work carried out in the field of Cell biology brings together such families of science as Retinal pigment epithelium and Retinal. Her Inflammation research is multidisciplinary, incorporating perspectives in Oxidative stress, Cancer research, Programmed cell death and Pathogenesis.

The Autophagy study combines topics in areas such as Endoplasmic reticulum, Retina, AMPK and Proteasome. Anu Kauppinen has researched Inflammasome in several fields, including Cytokine and Signal transducing adaptor protein. Her Signal transduction study deals with Innate immune system intersecting with Acquired immune system.

She most often published in these fields:

  • Cell biology (49.29%)
  • Inflammation (42.14%)
  • Autophagy (27.14%)

What were the highlights of her more recent work (between 2017-2021)?

  • Inflammation (42.14%)
  • Cell biology (49.29%)
  • Inflammasome (23.57%)

In recent papers she was focusing on the following fields of study:

Her scientific interests lie mostly in Inflammation, Cell biology, Inflammasome, Retinal pigment epithelium and Autophagy. Her Inflammation research is multidisciplinary, incorporating perspectives in Cancer research, Immune system and Pathogenesis. Her Immune system research is multidisciplinary, incorporating elements of Immunosuppression and Signal transduction.

Her research integrates issues of Retinal degeneration, Oxidative stress and Mitophagy in her study of Cell biology. The concepts of her Inflammasome study are interwoven with issues in Extracellular, Molecular biology, Cell culture and Retinal. As part of one scientific family, Anu Kauppinen deals mainly with the area of Autophagy, narrowing it down to issues related to the Programmed cell death, and often Computational biology, Chaperone-mediated autophagy and Autolysosome.

Between 2017 and 2021, her most popular works were:

  • Loss of NRF-2 and PGC-1α genes leads to retinal pigment epithelium damage resembling dry age-related macular degeneration. (48 citations)
  • Immunosenescence: the potential role of myeloid-derived suppressor cells (MDSC) in age-related immune deficiency. (41 citations)
  • Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition) (38 citations)

In her most recent research, the most cited papers focused on:

  • Enzyme
  • Cancer
  • Apoptosis

The scientist’s investigation covers issues in Autophagy, Inflammation, Cell biology, Myeloid-derived Suppressor Cell and Cancer research. Her biological study spans a wide range of topics, including Reactive oxygen species, MG132, Intracellular and Phosphorylation. Her work carried out in the field of Cell biology brings together such families of science as Retinal pigment epithelium, Oxidative stress and Inflammasome.

Her Oxidative stress research is multidisciplinary, relying on both Lipofuscin, NFE2L2, Proteostasis, Mitochondrion and Mitophagy. Her Cancer research research incorporates elements of Chemokine, Immune system, Immunotherapy, Signal transduction and Anaerobic glycolysis. Her work in the fields of T cell, CD8 and Innate immune system overlaps with other areas such as B cell.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)

Daniel J. Klionsky;Amal Kamal Abdel-Aziz;Sara Abdelfatah;Mahmoud Abdellatif.
Autophagy (2021)

8964 Citations

Antagonistic crosstalk between NF-κB and SIRT1 in the regulation of inflammation and metabolic disorders.

Anu Kauppinen;Tiina Suuronen;Johanna Ojala;Kai Kaarniranta.
Cellular Signalling (2013)

653 Citations

Activation of innate immunity system during aging: NF-kB signaling is the molecular culprit of inflamm-aging.

Antero Salminen;Jari Huuskonen;Johanna Ojala;Anu Kauppinen.
Ageing Research Reviews (2008)

519 Citations

Emerging role of NF-κB signaling in the induction of senescence-associated secretory phenotype (SASP).

Antero Salminen;Anu Kauppinen;Kai Kaarniranta.
Cellular Signalling (2012)

444 Citations

Inflammaging: disturbed interplay between autophagy and inflammasomes

Antero Salminen;Kai Kaarniranta;Anu Kauppinen.
Aging (Albany NY) (2012)

411 Citations

Inflammation in Alzheimer's disease: Amyloid-β oligomers trigger innate immunity defence via pattern recognition receptors

Antero Salminen;Johanna Ojala;Anu Kauppinen;Kai Kaarniranta.
Progress in Neurobiology (2009)

380 Citations

Inflammation and its role in age-related macular degeneration

Anu Kauppinen;Jussi J. Paterno;Janusz Blasiak;Antero Salminen.
Cellular and Molecular Life Sciences (2016)

353 Citations

ER stress in Alzheimer's disease: a novel neuronal trigger for inflammation and Alzheimer's pathology

Antero Salminen;Anu Kauppinen;Tiina Suuronen;Kai Kaarniranta.
Journal of Neuroinflammation (2009)

346 Citations

Clusterin: A forgotten player in Alzheimer's disease

Tapio Nuutinen;Tiina Suuronen;Anu Kauppinen;Antero Salminen.
Brain Research Reviews (2009)

334 Citations

Crosstalk between Oxidative Stress and SIRT1: Impact on the Aging Process.

Antero Salminen;Kai Kaarniranta;Anu Kauppinen.
International Journal of Molecular Sciences (2013)

330 Citations

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