His primary areas of investigation include Cancer research, Myeloid leukemia, Leukemia, Stem cell and Progenitor cell. His research in the fields of Imatinib mesylate overlaps with other disciplines such as Oligodendrocyte Transcription Factor 2. Xiaoyan Jiang performs multidisciplinary studies into Myeloid leukemia and Interleukin 3 in his work.
The Leukemia study combines topics in areas such as CD34 and Autocrine signalling. His Stem cell research integrates issues from Chronic myelogenous leukemia and Philadelphia chromosome. As a part of the same scientific family, Xiaoyan Jiang mostly works in the field of Haematopoiesis, focusing on Autophagy and, on occasion, Physiology.
His primary scientific interests are in Cancer research, Stem cell, Progenitor cell, Myeloid leukemia and CD34. The study incorporates disciplines such as Tyrosine kinase, Leukemia, K562 cells and Bone marrow in addition to Cancer research. His studies in Leukemia integrate themes in fields like Cancer stem cell, Autocrine signalling and Drug resistance.
The various areas that Xiaoyan Jiang examines in his Stem cell study include Molecular biology, Immunology and Imatinib mesylate. His Progenitor cell study incorporates themes from Autophagy, microRNA and Tyrosine-kinase inhibitor. His Myeloid leukemia research is multidisciplinary, incorporating elements of Gene silencing, Philadelphia chromosome and Small interfering RNA.
Xiaoyan Jiang focuses on Cancer research, Myeloid leukemia, Progenitor cell, Internal medicine and Ageing. His Cancer research research is multidisciplinary, relying on both Apoptosis, Cancer stem cell, Stem cell, Leukemia and Drug resistance. In Stem cell, Xiaoyan Jiang works on issues like microRNA, which are connected to Cell culture and Growth factor.
Xiaoyan Jiang interconnects Tyrosine kinase, K562 cells and Kinase in the investigation of issues within Myeloid leukemia. His research in Progenitor cell intersects with topics in Therapy resistant, Cell cycle and Xenotransplantation. Xiaoyan Jiang works mostly in the field of SiRNA binding, limiting it down to topics relating to Cell biology and, in certain cases, Autophagy, as a part of the same area of interest.
Xiaoyan Jiang mainly focuses on Cancer research, Myeloid leukemia, Tyrosine kinase, Imatinib and K562 cells. His work on Leukemia expands to the thematically related Cancer research. In his research, Autophagy, Drug, Disease and Cytotoxic T cell is intimately related to Drug resistance, which falls under the overarching field of Leukemia.
His Imatinib research is multidisciplinary, incorporating perspectives in Gene silencing and Protein kinase B. Xiaoyan Jiang has researched Gene silencing in several fields, including Jurkat cells, Small interfering RNA, Oncogene, Genetic enhancement and Tyrosine-kinase inhibitor. Xiaoyan Jiang has included themes like breakpoint cluster region, Stem cell, Kinase and Focal adhesion in his ABL study.
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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)
Daniel J. Klionsky;Kotb Abdelmohsen;Akihisa Abe;Joynal Abedin.
Autophagy (2016)
Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)
Daniel J. Klionsky;Kotb Abdelmohsen;Akihisa Abe;Joynal Abedin.
Parasites & Vectors (2016)
Isolation of a highly quiescent subpopulation of primitive leukemic cells in chronic myeloid leukemia.
Tessa Holyoake;Xiaoyan Jiang;Connie Eaves;Allen Eaves.
Blood (1999)
Chronic myeloid leukemia stem cells possess multiple unique features of resistance to BCR-ABL targeted therapies.
X Jiang;X Jiang;Y Zhao;C Smith;C Smith;M Gasparetto.
Leukemia (2007)
Autocrine production and action of IL-3 and granulocyte colony-stimulating factor in chronic myeloid leukemia
Xiaoyan Jiang;Angel Lopez;Tessa Holyoake;Allen Eaves.
Proceedings of the National Academy of Sciences of the United States of America (1999)
Stable Suppression of a Novel Oncogene, AHI-1, in Human Cutaneous T-Cell Leukemia Cells Normalizes Its Transforming Activity In Vitro and In Vivo and Aberrant Expression of AHI-1 Is Also Present in Leukemic Sezary Cells from Patients with Sezary Syndrome.
Ashley Ringrose;Youwen Zhou;Emily Pang;Ann E.-J. Lin.
Blood (2005)
Erratum to: Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition) (Autophagy, 12, 1, 1-222, 10.1080/15548627.2015.1100356
Daniel J. Klionsky;Kotb Abdelmohsen;Akihisa Abe;Joynal Abedin.
Autophagy (2016)
Primitive quiescent leukemic cells from patients with chronic myeloid leukemia spontaneously initiate factor-independent growth in vitro in association with up-regulation of expression of interleukin-3
Tessa L. Holyoake;Xiaoyan Jiang;Xiaoyan Jiang;Heather G. Jorgensen;Heather G. Jorgensen;Susan Graham;Susan Graham.
Blood (2001)
A novel role of phospholipase A2 in mediating spinal cord secondary injury.
Nai Kui Liu;Yi Ping Zhang;William Lee Titsworth;Xiaoyan Jiang.
Annals of Neurology (2006)
The core autophagy protein ATG4B is a potential biomarker and therapeutic target in CML stem/progenitor cells.
Katharina Rothe;Hanyang Lin;Hanyang Lin;Kevin B. L. Lin;Amy Leung.
Blood (2014)
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