His scientific interests lie mostly in TRPV4, Internal medicine, Endocrinology, Transient receptor potential channel and Cell biology. His study in TRPV4 is interdisciplinary in nature, drawing from both Vascular smooth muscle and Calcium signaling. As part of his studies on Endocrinology, Wolfgang Liedtke frequently links adjacent subjects like Inflammation.
The concepts of his Transient receptor potential channel study are interwoven with issues in Ruthenium red, Agonist, Sensory system, Anatomy and Calcitonin gene-related peptide. His Cell biology study integrates concerns from other disciplines, such as Osmotic concentration, Chondrocyte, Tissue engineering and TRPV. His work in Tissue engineering addresses subjects such as Cell, which are connected to disciplines such as Stem cell.
Wolfgang Liedtke focuses on TRPV4, Transient receptor potential channel, Cell biology, Internal medicine and Endocrinology. His TRPV4 study incorporates themes from Mechanotransduction, Neuroscience, Biophysics and Calcium signaling. His work is dedicated to discovering how Transient receptor potential channel, Nociceptor are connected with Capsaicin and other disciplines.
He has researched Cell biology in several fields, including Tissue engineering and Immunology. His work on Neurogenic inflammation, Pulmonary hypertension, Visceral pain and Vasodilation as part of general Internal medicine research is frequently linked to Protease-activated receptor 2, thereby connecting diverse disciplines of science. His Endocrinology research is multidisciplinary, incorporating elements of Agonist, Receptor, Calcitonin gene-related peptide and Hyperalgesia.
His primary areas of investigation include TRPV4, Transient receptor potential channel, Cell biology, Pharmacology and Mechanotransduction. His TRPV4 study is concerned with the field of Ion channel as a whole. The Transient receptor potential channel study combines topics in areas such as Tissue engineering, Nociceptor and Synthetic biology.
His biological study spans a wide range of topics, including TRPV, Pathogenesis, Transcription factor, Inhibitory postsynaptic potential and Degranulation. His Pharmacology study combines topics from a wide range of disciplines, such as Pneumonia, Dermatological disorders, Small molecule, Trigeminal ganglion and Endothelium. His Mechanotransduction research is multidisciplinary, relying on both Chondrocyte, Mechanosensitive channels and Pathology.
The scientist’s investigation covers issues in TRPV4, Transient receptor potential channel, Cell biology, Pharmacology and Neuroscience. His TRPV4 research incorporates themes from Inflammation, Pulmonary edema, Lung and Pathology. Wolfgang Liedtke interconnects Ankyrin and Nociceptor in the investigation of issues within Transient receptor potential channel.
Wolfgang Liedtke has included themes like TRPML, TRPM and TRPC in his Cell biology study. His Pharmacology research also works with subjects such as
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Control of stem cell fate by physical interactions with the extracellular matrix.
Farshid Guilak;Daniel M. Cohen;Bradley T. Estes;Jeffrey M. Gimble.
Cell Stem Cell (2009)
Vanilloid Receptor–Related Osmotically Activated Channel (VR-OAC), a Candidate Vertebrate Osmoreceptor
Wolfgang Liedtke;Yong Choe;Marc A. Martí-Renom;Andrea M. Bell;Andrea M. Bell.
Cell (2000)
Role for Stearoyl-CoA Desaturase-1 in Leptin-Mediated Weight Loss
Paul Cohen;Makoto Miyazaki;Nicholas D. Socci;Aaron Hagge-Greenberg.
Science (2002)
Abnormal osmotic regulation in trpv4-/- mice
Wolfgang Liedtke;Jeffrey M. Friedman.
Proceedings of the National Academy of Sciences of the United States of America (2003)
GFAP Is Necessary for the Integrity of CNS White Matter Architecture and Long-Term Maintenance of Myelination
Wolfgang Liedtke;Winfried Edelmann;Phyllis L. Bieri;Fung Chow Chiu.
Neuron (1996)
Elementary Ca2+ signals through endothelial TRPV4 channels regulate vascular function
Swapnil K. Sonkusare;Adrian D. Bonev;Jonathan Ledoux;Jonathan Ledoux;Wolfgang Liedtke.
Science (2012)
Mutation in the mismatch repair gene Msh6 causes cancer susceptibility
Winfried Edelmann;Kan Yang;Asad Umar;Joerg Heyer.
Cell (1997)
Protease‐activated receptor 2 sensitizes the transient receptor potential vanilloid 4 ion channel to cause mechanical hyperalgesia in mice
Andrew Douglas Grant;Graeme S. Cottrell;Silva Amadesi;Marcello Trevisani.
The Journal of Physiology (2007)
Mammalian TRPV4 (VR-OAC) directs behavioral responses to osmotic and mechanical stimuli in Caenorhabditis elegans.
Wolfgang Liedtke;David M. Tobin;Cornelia I. Bargmann;Jeffrey M. Friedman.
Proceedings of the National Academy of Sciences of the United States of America (2003)
TRPV4-mediated mechanotransduction regulates the metabolic response of chondrocytes to dynamic loading.
Christopher J. O’Conor;Holly A. Leddy;Halei C. Benefield;Wolfgang B. Liedtke.
Proceedings of the National Academy of Sciences of the United States of America (2014)
If you think any of the details on this page are incorrect, let us know.
We appreciate your kind effort to assist us to improve this page, it would be helpful providing us with as much detail as possible in the text box below:
Washington University in St. Louis
Columbia University
Digestive Health Research Institute
Duke University
Rockefeller University
McGill University Health Centre
University of Vermont
Albert Einstein College of Medicine
University College Dublin
Harvard University
University of Cambridge
Université Catholique de Louvain
Carnegie Mellon University
University of Notre Dame
Wuhan University of Technology
Delft University of Technology
University of Perpignan
Osaka University
University of Clermont Auvergne
Cardiff University
Chang Gung University
United States Food and Drug Administration
Australian National University
Clark University
Macquarie University
University of Tübingen