Timothy J. Williams mostly deals with Immunology, Internal medicine, Eosinophil, Endocrinology and Eotaxin. His studies deal with areas such as Zymosan, Chemotaxis, Inflammatory bowel disease and Pathology as well as Immunology. In the subject of general Internal medicine, his work in Inflammation, Nitric oxide, Guinea pig and Kidney disease is often linked to Medical examiner, thereby combining diverse domains of study.
His Eosinophil research is multidisciplinary, incorporating elements of Atopic dermatitis, In vivo and Interleukin 5. His Endocrinology study incorporates themes from Substance P and Calcitonin gene-related peptide. His Eotaxin research is under the purview of Chemokine.
His primary areas of study are Immunology, Inflammation, Eosinophil, Internal medicine and Pharmacology. His Immunology research integrates issues from Zymosan, Chemotaxis and In vivo. His Inflammation course of study focuses on Vascular permeability and Extravasation.
The Eosinophil study combines topics in areas such as Interleukin 5, Eosinophilia, Guinea pig, Bronchoalveolar lavage and Allergic inflammation. As part of his studies on Internal medicine, Timothy J. Williams often connects relevant areas like Endocrinology. Timothy J. Williams has researched Endocrinology in several fields, including Calcitonin gene-related peptide, Bradykinin and Substance P.
Timothy J. Williams spends much of his time researching Immunology, Chemokine, Cell biology, Chemokine receptor and Inflammation. His Immunology study focuses mostly on Allergy, Eosinophil, Eotaxin, Interleukin 5 and Allergic inflammation. His Eosinophil study combines topics in areas such as Eosinophilia and Nasal Lavage.
He interconnects Stem cell factor and Chemotaxis in the investigation of issues within Chemokine. His Cell biology research incorporates elements of Bradykinin, Intravital microscopy, Bone marrow and Chemokine Receptor Antagonist. Timothy J. Williams combines subjects such as Tumor necrosis factor alpha, Vascular permeability, Extravasation and Cremaster muscle with his study of Inflammation.
His main research concerns Immunology, Cell biology, Chemokine, Chemokine receptor and XCL2. Many of his research projects under Immunology are closely connected to Effector functions and Extramural with Effector functions and Extramural, tying the diverse disciplines of science together. Timothy J. Williams usually deals with Cell biology and limits it to topics linked to Bone marrow and Stem cell, Haematopoiesis, Mast cell, Myelokathexis and CXC chemokine receptors.
His studies examine the connections between Stem cell and genetics, as well as such issues in Prostaglandin E2 receptor, with regards to Endocrinology. The CXCR4 research Timothy J. Williams does as part of his general Chemokine study is frequently linked to other disciplines of science, such as Neutrophil homeostasis, therefore creating a link between diverse domains of science. His Chemokine receptor study integrates concerns from other disciplines, such as Molecular biology, Chemotaxis and G protein-coupled receptor.
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Calcitonin gene-related peptide is a potent vasodilator
S. D. Brain;T. J. Williams;J. R. Tippins;H. R. Morris.
Nature (1985)
Control of vascular permeability by polymorphonuclear leukocytes in inflammation.
Caroline V. Wedmore;T. J. Williams.
Nature (1981)
Eotaxin: a potent eosinophil chemoattractant cytokine detected in a guinea pig model of allergic airways inflammation.
P J Jose;D A Griffiths-Johnson;P D Collins;D T Walsh.
Journal of Experimental Medicine (1994)
Cooperation between interleukin-5 and the chemokine eotaxin to induce eosinophil accumulation in vivo.
P D Collins;S Marleau;D A Griffiths-Johnson;P J Jose.
Journal of Experimental Medicine (1995)
Chemokines Acting via CXCR2 and CXCR4 Control the Release of Neutrophils from the Bone Marrow and Their Return following Senescence
Coralie Martin;Peter C E Burdon;Gary Bridger;Jose Carlos Gutierrez-Ramos.
Immunity (2003)
Role of prostaglandin-mediated vasodilatation in inflammation
T J Williams;M J Peck.
Nature (1977)
Enhanced expression of eotaxin and CCR3 mRNA and protein in atopic asthma. Association with airway hyperresponsiveness and predominant co-localization of eotaxin mRNA to bronchial epithelial and endothelial cells.
Sun Ying;Douglas S. Robinson;Qiu Meng;James Rottman.
European Journal of Immunology (1997)
Nitric oxide attenuates cardiac myocyte contraction
A. J. B. Brady;J. B. Warren;P. A. Poole-Wilson;T. J. Williams.
American Journal of Physiology-heart and Circulatory Physiology (1993)
Angiogenic and HIV-Inhibitory Functions of KSHV-Encoded Chemokines
Chris Boshoff;Yoshio Endo;Paul D. Collins;Yasuhiro Takeuchi.
Science (1997)
Inflammatory oedema induced by synergism between calcitonin gene-related peptide (CGRP) and mediators of increased vascular permeability.
S.D. Brain;T.J. Williams.
British Journal of Pharmacology (1985)
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