His scientific interests lie mostly in Virology, Hepatitis C virus, Virus, Viral entry and Immunology. His Virology research is multidisciplinary, relying on both Epitope, Antibody and Immune system. His work deals with themes such as Virus-like particle and Antigen, which intersect with Antibody.
His Hepatitis C virus research integrates issues from Liver disease, Hepatitis C and Cell culture. The concepts of his Liver disease study are interwoven with issues in Hepatocellular carcinoma, Cirrhosis, Liver transplantation and Hepatitis B virus. His research in Viral entry intersects with topics in Epidermal growth factor receptor, Tyrosine kinase, Kinase activity, Internalization and Host factor.
Thomas F. Baumert mainly focuses on Virology, Hepatitis C virus, Immunology, Virus and Liver disease. The Virology study combines topics in areas such as Immune system and Antibody, Monoclonal antibody. His research integrates issues of Epitope, Hepatocellular carcinoma, Hepatitis C and Viral replication in his study of Hepatitis C virus.
His study in Immunology is interdisciplinary in nature, drawing from both Chronic liver disease and Liver transplantation, Transplantation. His study ties his expertise on Cell culture together with the subject of Virus. His Liver disease research includes themes of Cancer, Pathogenesis, Cell entry, Viral hepatitis and Disease.
His primary scientific interests are in Hepatocellular carcinoma, Hepatitis B virus, Virus, Liver disease and Cirrhosis. His biological study spans a wide range of topics, including Aspirin, Hepatitis C and Hepatitis C virus. His Hepatitis C virus study frequently draws connections to other fields, such as Liver cancer.
Virology and Immunology are inherently bound to his Hepatitis B virus studies. His study connects RNA interference and Virology. He has researched Virus in several fields, including Gene silencing, Cell biology and Innate immune system.
His primary areas of investigation include Hepatocellular carcinoma, Liver disease, Cancer research, Hepatitis C virus and Cell biology. His studies in Hepatocellular carcinoma integrate themes in fields like Cirrhosis, Radiology, Computed tomography, Fibrosis and Radiogenomics. He interconnects Cancer, Pathogenesis, Bioinformatics and Steatohepatitis, Fatty liver in the investigation of issues within Liver disease.
His Cancer research study combines topics from a wide range of disciplines, such as Cell, Cell type and Cholangiocyte. His Hepatitis C virus research includes elements of Lipid signaling, Flavivirus and Gene, DNA replication. His Hepatitis B virus research is within the category of Immunology.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
EGFR and EphA2 are host factors for hepatitis C virus entry and possible targets for antiviral therapy
Joachim Lupberger;Mirjam B Zeisel;Mirjam B Zeisel;Fei Xiao;Fei Xiao;Christine Thumann;Christine Thumann.
Nature Medicine (2011)
Cellular Binding of Hepatitis C Virus Envelope Glycoprotein E2 Requires Cell Surface Heparan Sulfate
Heidi Barth;Christiane Schäfer;Mohammed I. Adah;Fuming Zhang.
Journal of Biological Chemistry (2003)
Rapid induction of virus-neutralizing antibodies and viral clearance in a single-source outbreak of hepatitis C
Jan M. Pestka;Mirjam B. Zeisel;Edith Bläser;Peter Schürmann.
Proceedings of the National Academy of Sciences of the United States of America (2007)
Hepatitis C Virus Structural Proteins Assemble into Viruslike Particles in Insect Cells
Thomas F. Baumert;Susumu Ito;David T. Wong;T. Jake Liang.
Journal of Virology (1998)
miR-122 – A key factor and therapeutic target in liver disease
Simonetta Bandiera;Simonetta Bandiera;Sébastien Pfeffer;Sébastien Pfeffer;Thomas F. Baumert;Thomas F. Baumert;Mirjam B. Zeisel;Mirjam B. Zeisel.
Journal of Hepatology (2015)
Exosome-mediated transmission of hepatitis C virus between human hepatoma Huh7.5 cells
Vedashree Ramakrishnaiah;Christine Thumann;Isabel Fofana;Francois Habersetzer.
Proceedings of the National Academy of Sciences of the United States of America (2013)
Scavenger receptor class B type I is a key host factor for hepatitis C virus infection required for an entry step closely linked to CD81
Mirjam B. Zeisel;Mirjam B. Zeisel;George Koutsoudakis;Eva K. Schnober;Anita Haberstroh;Anita Haberstroh.
Initiation of Hepatitis C Virus Infection Is Dependent on Cholesterol and Cooperativity between CD81 and Scavenger Receptor B Type I
Sharookh B. Kapadia;Heidi Barth;Thomas Baumert;Jane A. McKeating.
Journal of Virology (2007)
Neutralizing Antibody-Resistant Hepatitis C Virus Cell-to-Cell Transmission
Claire L. Brimacombe;Joe Grove;Luke W. Meredith;Ke Hu.
Journal of Virology (2011)
Pathogenesis of hepatitis B virus infection.
Thomas F Baumert;Robert Thimme;Fritz von Weizsäcker.
World Journal of Gastroenterology (2007)
If you think any of the details on this page are incorrect, let us know.
We appreciate your kind effort to assist us to improve this page, it would be helpful providing us with as much detail as possible in the text box below: