Mirjam B. Zeisel spends much of her time researching Hepatitis C virus, Virology, Viral entry, Virus and CD81. The Hepatitis C virus study combines topics in areas such as Liver disease and Tissue tropism. Mirjam B. Zeisel focuses mostly in the field of Liver disease, narrowing it down to topics relating to Hepatitis B virus and, in certain cases, Cancer research.
Her Virology research integrates issues from Antibody, Immunology and Epidermal growth factor receptor. Her Viral entry research includes themes of Tyrosine kinase, EPH receptor A2, Kinase activity and Erythropoietin-producing hepatocellular receptor. As part of the same scientific family, Mirjam B. Zeisel usually focuses on Virus, concentrating on Epitope and intersecting with Viral replication, Hypervariable region, Infectivity and Molecular biology.
Her main research concerns Virology, Hepatitis C virus, Immunology, Virus and Viral entry. Her study in Virology is interdisciplinary in nature, drawing from both Antibody and Monoclonal antibody. Her work deals with themes such as Cell culture, Pathogenesis, Immune system, Liver disease and Hepatocellular carcinoma, which intersect with Hepatitis C virus.
Her Immunology study combines topics in areas such as Chronic liver disease and Liver transplantation. Her work carried out in the field of Virus brings together such families of science as Epitope and Interferon. Her work in Viral entry tackles topics such as Scavenger receptor which are related to areas like Receptor.
Her primary areas of investigation include Liver disease, Hepatocellular carcinoma, Hepatitis C virus, Virology and Virus. Her Liver disease research is multidisciplinary, incorporating elements of Regulation of gene expression, Tight junction and Disease. Mirjam B. Zeisel combines subjects such as Cirrhosis and Epigenetics with her study of Hepatocellular carcinoma.
Her Hepatitis C virus study frequently draws parallels with other fields, such as microRNA. Her Virus research is multidisciplinary, incorporating perspectives in Interferon, Innate immune system and Cell culture. Her Viral entry research includes elements of Chronic infection and Immune system.
The scientist’s investigation covers issues in Liver disease, Hepatocellular carcinoma, Cancer research, Cirrhosis and Virus. Her biological study spans a wide range of topics, including Cancer, Viral entry and Evasion, Immune system. The various areas that she examines in her Hepatocellular carcinoma study include Hepatitis C virus, Downregulation and upregulation, microRNA, Fibrosis and STAT3.
Her work carried out in the field of Hepatitis C virus brings together such families of science as Peroxisome proliferator-activated receptor, Transcriptome, Antibody and Steatohepatitis, Fatty liver. Her biological study spans a wide range of topics, including Pathogenesis, Receptor, Disease, Regulation of gene expression and Tight junction. Her Virus study is concerned with the field of Immunology as a whole.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
EGFR and EphA2 are host factors for hepatitis C virus entry and possible targets for antiviral therapy
Joachim Lupberger;Mirjam B Zeisel;Mirjam B Zeisel;Fei Xiao;Fei Xiao;Christine Thumann;Christine Thumann.
Nature Medicine (2011)
Rapid induction of virus-neutralizing antibodies and viral clearance in a single-source outbreak of hepatitis C
Jan M. Pestka;Mirjam B. Zeisel;Edith Bläser;Peter Schürmann.
Proceedings of the National Academy of Sciences of the United States of America (2007)
miR-122 – A key factor and therapeutic target in liver disease
Simonetta Bandiera;Simonetta Bandiera;Sébastien Pfeffer;Sébastien Pfeffer;Thomas F. Baumert;Thomas F. Baumert;Mirjam B. Zeisel;Mirjam B. Zeisel.
Journal of Hepatology (2015)
Scavenger receptor class B type I is a key host factor for hepatitis C virus infection required for an entry step closely linked to CD81
Mirjam B. Zeisel;Mirjam B. Zeisel;George Koutsoudakis;Eva K. Schnober;Anita Haberstroh;Anita Haberstroh.
Apolipoprotein E interacts with hepatitis C virus nonstructural protein 5A and determines assembly of infectious particles.
Wagane J. A. Benga;Wagane J. A. Benga;Sophie E. Krieger;Sophie E. Krieger;Maria Dimitrova;Maria Dimitrova;Mirjam B. Zeisel;Mirjam B. Zeisel.
Towards an HBV cure: state-of-the-art and unresolved questions—report of the ANRS workshop on HBV cure
Mirjam B. Zeisel;Julie Lucifora;William S. Mason;Camille Sureau.
Hepatitis C Virus Hypervariable Region 1 Modulates Receptor Interactions, Conceals the CD81 Binding Site, and Protects Conserved Neutralizing Epitopes
Dorothea Bankwitz;Eike Steinmann;Julia Bitzegeio;Sandra Ciesek.
Journal of Virology (2010)
Hepatitis C virus entry into hepatocytes: molecular mechanisms and targets for antiviral therapies.
Mirjam B. Zeisel;Mirjam B. Zeisel;Isabel Fofana;Isabel Fofana;Samira Fafi-Kremer;Samira Fafi-Kremer;Thomas F. Baumert;Thomas F. Baumert.
Journal of Hepatology (2011)
Inhibition of hepatitis C virus infection by anti-claudin-1 antibodies is mediated by neutralization of E2-CD81-claudin-1 associations.
Sophie E. Krieger;Mirjam B. Zeisel;Mirjam B. Zeisel;Christopher Davis;Christine Thumann;Christine Thumann.
HRas Signal Transduction Promotes Hepatitis C Virus Cell Entry by Triggering Assembly of the Host Tetraspanin Receptor Complex
Laetitia Zona;Joachim Lupberger;Joachim Lupberger;Nazha Sidahmed-Adrar;Nazha Sidahmed-Adrar;Christine Thumann;Christine Thumann.
Cell Host & Microbe (2013)
If you think any of the details on this page are incorrect, let us know.
We appreciate your kind effort to assist us to improve this page, it would be helpful providing us with as much detail as possible in the text box below: