His main research concerns Biochemistry, Molecular biology, Biosynthesis, Endoplasmic reticulum and Gene. His Biochemistry study frequently draws connections to adjacent fields such as Cell biology. His work deals with themes such as Decay-accelerating factor, Antibody, Complement system, Paroxysmal nocturnal hemoglobinuria and Mutant, which intersect with Molecular biology.
Taroh Kinoshita has included themes like Hematopoietic stem cell, Haematopoiesis and CD59 in his Paroxysmal nocturnal hemoglobinuria study. His Biosynthesis research integrates issues from Glycosylphosphatidylinositol, Protozoa and Saccharomyces cerevisiae. Taroh Kinoshita usually deals with Endoplasmic reticulum and limits it to topics linked to Signal peptide and CD59 antigen.
Taroh Kinoshita focuses on Biochemistry, Cell biology, Paroxysmal nocturnal hemoglobinuria, Immunology and Molecular biology. His work in Endoplasmic reticulum, Biosynthesis, Phosphatidylinositol, Mannose and Mutant are all subfields of Biochemistry research. His Cell biology study integrates concerns from other disciplines, such as Cell, Receptor, Membrane protein and Glycan.
As part of the same scientific family, Taroh Kinoshita usually focuses on Paroxysmal nocturnal hemoglobinuria, concentrating on Germline mutation and intersecting with Somatic cell. His research on Immunology often connects related areas such as Clone. His research integrates issues of Decay-accelerating factor, Antibody and Gene in his study of Molecular biology.
His primary areas of study are Cell biology, Genetics, Gene, Immunology and Endoplasmic reticulum. His study in Cell biology is interdisciplinary in nature, drawing from both Lactosylceramide, Biosynthesis, Mutant, HEK 293 cells and Membrane protein. His Gene research is multidisciplinary, relying on both Immunoglobulin D, Glycosylation, Intellectual disability and Epilepsy.
As a part of the same scientific family, Taroh Kinoshita mostly works in the field of Immunology, focusing on Bone marrow failure and, on occasion, Hematopoietic stem cell. The concepts of his Endoplasmic reticulum study are interwoven with issues in Protein folding, Genetic screen and Glycan. His Golgi apparatus study contributes to a more complete understanding of Biochemistry.
His scientific interests lie mostly in Genetics, Gene, Mutation, Compound heterozygosity and Cell biology. His study in the fields of Phenotype, Exome sequencing, Exome and Missense mutation under the domain of Genetics overlaps with other disciplines such as Phosphorus metabolism disorder. His Gene study combines topics in areas such as Endoplasmic reticulum, Internal medicine, Glycosylation and Endocrinology.
The study incorporates disciplines such as Molecular biology, Untranslated region and Consanguinity in addition to Mutation. His Cell biology research incorporates themes from Glycosylphosphatidylinositol, Biochemistry and Membrane protein. Taroh Kinoshita regularly ties together related areas like Flow cytometry in his Biochemistry studies.
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Essentials of Glycobiology [Internet]
Ajit Varki;Richard D Cummings;Jeffrey D Esko;Pamela Stanley.
Deficiency of the GPI anchor caused by a somatic mutation of the PIG-A gene in paroxysmal nocturnal hemoglobinuria.
Junji Takeda;Toshio Miyata;Kazuyoshi Kawagoe;Yoshiyasu Iida;Yoshiyasu Iida.
Diagnosis and management of paroxysmal nocturnal hemoglobinuria.
Charles Parker;Mitsuhiro Omine;Stephen Richards;Jun-Ichi Nishimura.
Direct recognition of the mycobacterial glycolipid, trehalose dimycolate, by C-type lectin Mincle
Eri Taniguchi Ishikawa;Tetsuaki Ishikawa;Tetsuaki Ishikawa;Yasu S Morita;Kenji Toyonaga.
Journal of Experimental Medicine (2009)
Inhibition of complement activation on the surface of cells after incorporation of decay-accelerating factor (DAF) into their membranes.
M E Medof;T Kinoshita;Nussenzweig.
Journal of Experimental Medicine (1984)
Symbol Nomenclature for Graphical Representations of Glycans.
Ajit Varki;Richard D. Cummings;Markus Aebi;Nicole H. Packer.
The Cloning of PIG-A, a Component in the Early Step of GPI-Anchor Biosynthesis
Toshio Miyata;Junji Takeda;Yoshiyasu Iida;Norio Yamada.
β2-Microglobulin modified with advanced glycation end products is a major component of hemodialysis-associated amyloidosis
Toshio Miyata;Osamu Oda;Reiko Inagi;Yoshiyasu Iida.
Journal of Clinical Investigation (1993)
Distribution of decay-accelerating factor in the peripheral blood of normal individuals and patients with paroxysmal nocturnal hemoglobinuria.
T Kinoshita;M E Medof;R Silber;V Nussenzweig.
Journal of Experimental Medicine (1985)
Paroxysmal nocturnal haemoglobinuria (PNH) is caused by somatic mutations in the PIG-A gene.
M Bessler;P J Mason;P Hillmen;T Miyata.
The EMBO Journal (1994)
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