H-Index & Metrics Best Publications

H-Index & Metrics

Discipline name H-index Citations Publications World Ranking National Ranking
Medicine D-index 76 Citations 21,066 178 World Ranking 11368 National Ranking 6048

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Cancer
  • DNA

His primary scientific interests are in Cancer research, Tumor suppressor gene, Pathology, Chromosomal translocation and Cytogenetics. Suresh C. Jhanwar combines subjects such as Cancer, Germline mutation and Gene rearrangement, Gene, Allele with his study of Cancer research. His study in Tumor suppressor gene is interdisciplinary in nature, drawing from both Locus, Neurofibromatosis type 2 and Point mutation.

His work carried out in the field of Pathology brings together such families of science as Fluorescence in situ hybridization and Leukemia. His studies in Chromosomal translocation integrate themes in fields like Molecular biology and Chromosome 17. His Cytogenetics research incorporates themes from Comparative genomic hybridization and Karyotype.

His most cited work include:

  • BRCA1 mutations in primary breast and ovarian carcinomas (1103 citations)
  • Complete remission after treatment of acute promyelocytic leukemia with arsenic trioxide (1093 citations)
  • Primary structure of c-kit: relationship with the CSF-1/PDGF receptor kinase family-oncogenic activation of v-kit involves deletion of extracellular domain and C terminus (657 citations)

What are the main themes of his work throughout his whole career to date?

Suresh C. Jhanwar mostly deals with Pathology, Cancer research, Molecular biology, Genetics and Immunology. The various areas that he examines in his Pathology study include Fluorescence in situ hybridization, Karyotype and Cytogenetics. In Cancer research, Suresh C. Jhanwar works on issues like Retinoblastoma, which are connected to Mdm2.

His biological study spans a wide range of topics, including Cell culture and Southern blot, Chromosome, Gene, Chromosomal translocation. His Immunology study combines topics from a wide range of disciplines, such as Philadelphia chromosome and Oncology. Suresh C. Jhanwar has researched Leukemia in several fields, including Myeloid leukemia and Transplantation.

He most often published in these fields:

  • Pathology (31.78%)
  • Cancer research (29.84%)
  • Molecular biology (23.64%)

What were the highlights of his more recent work (between 2008-2021)?

  • Cancer research (29.84%)
  • Pathology (31.78%)
  • Internal medicine (20.16%)

In recent papers he was focusing on the following fields of study:

Suresh C. Jhanwar focuses on Cancer research, Pathology, Internal medicine, Oncology and Cancer. His Cancer research study integrates concerns from other disciplines, such as Mesothelioma, DNA methylation, Epidermal growth factor receptor, Carcinogenesis and Retinoblastoma. His Pathology study combines topics from a wide range of disciplines, such as Metastasis, Immunophenotyping, Cytogenetics, Fluorescence in situ hybridization and Leukemia.

His Cytogenetics research is multidisciplinary, relying on both Non-Hodgkin's lymphoma, Lymphoma, Disease and Karyotype. His Internal medicine research integrates issues from Gastroenterology and Surgery. The study incorporates disciplines such as Hepatocyte growth factor and Exon in addition to Cancer.

Between 2008 and 2021, his most popular works were:

  • Crizotinib in ALK-rearranged inflammatory myofibroblastic tumor. (555 citations)
  • Intratumoral heterogeneity of receptor tyrosine kinases EGFR and PDGFRA amplification in glioblastoma defines subpopulations with distinct growth factor response (365 citations)
  • Genomic and Biological Characterization of Exon 4 KRAS Mutations in Human Cancer (219 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Cancer
  • DNA

Suresh C. Jhanwar focuses on Cancer research, Fluorescence in situ hybridization, Pathology, Genetics and Melanoma. His Cancer research study integrates concerns from other disciplines, such as Epidermal growth factor receptor, Carcinogenesis, Mdm2, Molecular biology and Cell type. His Molecular biology research focuses on subjects like Regulation of gene expression, which are linked to Gene dosage.

Within one scientific family, he focuses on topics pertaining to Genome under Fluorescence in situ hybridization, and may sometimes address concerns connected to DNA sequencing, Exome sequencing and Double minute. His Pathology research includes elements of Stomach cancer, Real-time polymerase chain reaction, Concordance, Gene duplication and Adenocarcinoma. His research integrates issues of Dermatology, Comparative genomic hybridization and Anatomical pathology in his study of Melanoma.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

BRCA1 mutations in primary breast and ovarian carcinomas

P. Andrew Futreal;Qingyun Liu;Donna Shattuck-Eidens;Charles Cochran.
Science (1994)

1625 Citations

Complete remission after treatment of acute promyelocytic leukemia with arsenic trioxide

Steven L. Soignet;Peter Maslak;Zhu-Gang Wang;Suresh Jhanwar.
The New England Journal of Medicine (1998)

1509 Citations

Primary structure of c-kit: relationship with the CSF-1/PDGF receptor kinase family-oncogenic activation of v-kit involves deletion of extracellular domain and C terminus

Feihua Qiu;Prabir Ray;K. Brown;P. E. Barker.
The EMBO Journal (1988)

821 Citations

Familial colorectal cancer in Ashkenazim due to a hypermutable tract in APC

Steven J. Laken;Gloria M. Petersen;Stephen B. Gruber;Carole Oddoux.
Nature Genetics (1997)

771 Citations

Crizotinib in ALK-rearranged inflammatory myofibroblastic tumor.

James E. Butrynski;David R. D’Adamo;Jason L. Hornick;Paola Dal Cin.
The New England Journal of Medicine (2010)

726 Citations

A Collaborative Survey of 80 Mutations in the BRCA1 Breast and Ovarian Cancer Susceptibility Gene: Implications for Presymptomatic Testing and Screening

Donna Shattuck Eidens;Melody Mcclure;Jacques Simard;Fernand Labrie.
JAMA (1995)

585 Citations

EWS-FLI1 fusion transcript structure is an independent determinant of prognosis in Ewing's sarcoma.

E. De Alava;A. Kawai;J. H. Healey;I. Fligman.
Journal of Clinical Oncology (1998)

545 Citations

Intratumoral heterogeneity of receptor tyrosine kinases EGFR and PDGFRA amplification in glioblastoma defines subpopulations with distinct growth factor response

Nicholas J. Szerlip;Alicia Pedraza;Debyani Chakravarty;Mohammad Azim.
Proceedings of the National Academy of Sciences of the United States of America (2012)

494 Citations

Rearrangement of the bcl-6 gene as a prognostic marker in diffuse large-cell lymphoma

Kenneth Offit;Francesco Lo Coco;Francesco Lo Coco;Diane C. Louie;Nasser Z. Parsa.
The New England Journal of Medicine (1994)

443 Citations

MDM2 Gene Amplification in Metastatic Osteosarcoma

Marc Ladanyi;Charles Cha;Ronald Lewis;Suresh C. Jhanwar.
Cancer Research (1993)

439 Citations

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