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Medicine

D-Index
116
Citations
60894
World Ranking
4299
National Ranking
2349

Research.com Recognitions

  • Member of the Association of American Physicians
  • Member of the Association of American Physicians

Overview

Stavros C. Manolagas is a researcher affiliated with the University of Arkansas for Medical Sciences in the United States. Their work primarily spans the fields of Immunology and Microbiology, Medicine, and Biochemistry, Genetics, and Molecular Biology. Within these broader areas, their research focuses on specialized subfields such as Immunology, Molecular Biology, Radiology, Nuclear Medicine and Imaging, and Oncology.

Manolagas has contributed to research on various scientific topics including:

  • Atherosclerosis and Cardiovascular Diseases
  • Monoclonal and Polyclonal Antibodies Research
  • T-cell and B-cell Immunology
  • Bone Metabolism and Diseases
  • Cytokine Signaling Pathways and Interactions
  • S100 Proteins and Annexins

Their recent scientific papers reflect their research interests in immunology and bone metabolism. Some of the notable publications include:

  • "Plasma levels of anti phosphocholine IgM antibodies are negatively correlated with bone mineral density in humans," 2025, published in Scientific Reports
  • "Deletion of the scavenger receptor Scarb1 in osteoblast progenitors and myeloid cells does not affect bone mass," 2025, published in bioRxiv (Cold Spring Harbor Laboratory)
  • "Deletion of the scavenger receptor Scarb1 in osteoblast progenitors and myeloid cells does not affect bone mass," 2025, published in PLoS ONE

Frequent collaborators in Manolagas's research include Michela Palmieri, Elena Ambrogini, Teenamol E. Joseph, Horacio Gomez-Acevedo, and Ha-Neui Kim. Collaboration with these co-authors has resulted in multiple publications, particularly in the areas of bone metabolism and immunology.

Their work has been published in journals and venues such as Scientific Reports, bioRxiv (Cold Spring Harbor Laboratory), and PLoS ONE.

Among professional recognitions, Manolagas is noted as a member of the Association of American Physicians, reflecting engagement with the medical research community.

Best Publications

  • Birth and death of bone cells: basic regulatory mechanisms and implications for the pathogenesis and treatment of osteoporosis.

    Stavros C. Manolagas

  • Bone Marrow, Cytokines, and Bone Remodeling — Emerging Insights into the Pathophysiology of Osteoporosis

    Stavros C. Manolagas;Robert L. Jilka

  • Inhibition of osteoblastogenesis and promotion of apoptosis of osteoblasts and osteocytes by glucocorticoids. Potential mechanisms of their deleterious effects on bone.

    R S Weinstein;R L Jilka;A M Parfitt;S C Manolagas

  • Increased osteoclast development after estrogen loss: mediation by interleukin-6

    Robert L. Jilka;Giao Hangoc;Giuseppe Girasole;Giovanni Passeri

  • The Pathophysiologic Roles of Interleukin-6 in Human Disease

    Dimitris A. Papanicolaou;Ronald L. Wilder;Stavros C. Manolagas;George P. Chrousos

  • Nongenotropic, sex-nonspecific signaling through the estrogen or androgen receptors: dissociation from transcriptional activity.

    S. Kousteni;T. Bellido;L. I. Plotkin;C. A. O'Brien

  • 1,25-dihydroxyvitamin D3 receptors in human leukocytes

    DM Provvedini;CD Tsoukas;LJ Deftos;SC Manolagas

  • Matrix-embedded cells control osteoclast formation

    Jinhu Xiong;Melda Onal;Robert L Jilka;Robert S Weinstein

  • Increased bone formation by prevention of osteoblast apoptosis with parathyroid hormone

    Robert L. Jilka;Robert S. Weinstein;Teresita Bellido;Paula Roberson

  • From Estrogen-Centric to Aging and Oxidative Stress: A Revised Perspective of the Pathogenesis of Osteoporosis

    Stavros C. Manolagas

  • Prevention of osteocyte and osteoblast apoptosis by bisphosphonates and calcitonin

    Lilian I. Plotkin;Robert S. Weinstein;Robert S. Weinstein;A. Michael Parfitt;Paula K. Roberson

  • 17 beta-estradiol inhibits interleukin-6 production by bone marrow-derived stromal cells and osteoblasts in vitro: a potential mechanism for the antiosteoporotic effect of estrogens.

    G Girasole;R L Jilka;G Passeri;S Boswell

  • Glucocorticoids act directly on osteoblasts and osteocytes to induce their apoptosis and reduce bone formation and strength.

    Charles A. O’Brien;Dan Jia;Lilian I. Plotkin;Teresita Bellido

  • Osteoblast Programmed Cell Death (Apoptosis): Modulation by Growth Factors and Cytokines

    Robert L. Jilka;Robert S. Weinstein;Teresita Bellido;A. Michael Parfitt

  • Skeletal Involution by Age-associated Oxidative Stress and Its Acceleration by Loss of Sex Steroids

    Maria Almeida;Li Han;Marta Martin-Millan;Lilian I. Plotkin

  • 1,25-dihydroxyvitamin D3: a novel immunoregulatory hormone.

    Constantine D. Tsoukas;Diego M. Provvedini;Stavros C. Manolagas

  • Sex steroids and bone.

    Manolagas Sc;Kousteni S;Jilka Rl

  • Estrogens and Androgens in Skeletal Physiology and Pathophysiology.

    Maria Almeida;Michaël R. Laurent;Vanessa Dubois;Frank Claessens

  • Chronic elevation of parathyroid hormone in mice reduces expression of sclerostin by osteocytes: a novel mechanism for hormonal control of osteoblastogenesis.

    T. Bellido;A. A. Ali;I. Gubrij;L. I. Plotkin

  • 92238400 Increased osteoclast development after estrogen loss: Mediation by interleukin-6

    R.L. Jilka;G. Hangoc;G. Girasole;G. Passeri

Frequent Co-Authors

Robert L. Jilka
Robert L. Jilka University of Arkansas for Medical Sciences
Robert S. Weinstein
Robert S. Weinstein University of Arkansas for Medical Sciences
Teresita Bellido
Teresita Bellido University of Arkansas for Medical Sciences
Haibo Zhao
Haibo Zhao Huazhong University of Science and Technology
Leonard J. Deftos
Leonard J. Deftos University of California, San Diego
Rafael de Cabo
Rafael de Cabo National Institutes of Health
Daohong Zhou
Daohong Zhou University of Florida
John A. Katzenellenbogen
John A. Katzenellenbogen University of Illinois at Urbana-Champaign
Brendan F. Boyce
Brendan F. Boyce University of Rochester Medical Center
Evan T. Keller
Evan T. Keller University of Michigan–Ann Arbor

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