Member of the Association of American Physicians
His primary areas of investigation include Internal medicine, Endocrinology, Osteoblast, Growth factor and Bone cell. Bone resorption, Bone remodeling, Osteoporosis, Parathyroid hormone and Glucocorticoid are the core of his Internal medicine study. The concepts of his Endocrinology study are interwoven with issues in Calvaria, DNA synthesis, Cell biology and Osteocalcin, Alkaline phosphatase.
While the research belongs to areas of Osteoblast, he spends his time largely on the problem of Cellular differentiation, intersecting his research to questions surrounding Notch signaling pathway and Wnt signaling pathway. His work deals with themes such as Transforming growth factor and Cell culture, which intersect with Growth factor. His work investigates the relationship between Bone cell and topics such as Cell growth that intersect with problems in Osteocyte.
Ernesto Canalis mainly focuses on Internal medicine, Endocrinology, Osteoblast, Growth factor and Molecular biology. His Internal medicine research includes themes of Bone morphogenetic protein and DNA synthesis. His Endocrinology study combines topics from a wide range of disciplines, such as Osteocalcin and Cell biology.
His Osteoblast study incorporates themes from Stromal cell, Cycloheximide, Cellular differentiation, Osteoclast and Messenger RNA. His Growth factor research is multidisciplinary, relying on both Cell culture and Northern blot. His studies in Molecular biology integrate themes in fields like Autocrine signalling, RNA, Transcription factor, Collagenase and Binding site.
His primary scientific interests are in Internal medicine, Endocrinology, Bone resorption, Notch signaling pathway and Osteoblast. His study in the field of Bone density is also linked to topics like Endocrine disease. His research integrates issues of Sclerostin and Parathyroid hormone in his study of Endocrinology.
His research in Bone resorption intersects with topics in Osteoprotegerin, Cellular differentiation, Cortical bone, Osteoclast and Osteocalcin. His study in Notch signaling pathway is interdisciplinary in nature, drawing from both Molecular biology, Wnt signaling pathway and Gene expression. His work focuses on many connections between Bone remodeling and other disciplines, such as HEY1, that overlap with his field of interest in Transgene.
Ernesto Canalis mainly investigates Internal medicine, Endocrinology, Bone resorption, Notch signaling pathway and Bone remodeling. His Internal medicine research incorporates themes from Wnt signaling pathway and Cellular differentiation. His Bone resorption research integrates issues from Cortical bone and Sclerostin.
His Cortical bone course of study focuses on Osteoclast and Osteoblast. His work in Notch signaling pathway covers topics such as Cell signaling which are related to areas like NFAT, HEY2, Hairless, Osteocyte and Transcription factor. Ernesto Canalis combines subjects such as Epithelial–mesenchymal transition, Autocrine signalling and Cytokine with his study of Cell biology.
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Glucocorticoid-induced osteoporosis: pathophysiology and therapy.
E. Canalis;G. Mazziotti;A. Giustina;J. P. Bilezikian.
Osteoporosis International (2007)
Bone morphogenetic proteins, their antagonists, and the skeleton.
Ernesto Canalis;Ernesto Canalis;Aris N. Economides;Elisabetta Gazzerro;Elisabetta Gazzerro.
Endocrine Reviews (2003)
Transforming growth factor beta is a bifunctional regulator of replication and collagen synthesis in osteoblast-enriched cell cultures from fetal rat bone.
M Centrella;T L McCarthy;E Canalis.
Journal of Biological Chemistry (1987)
Growth Hormone, Insulin-Like Growth Factors, and the Skeleton
Andrea Giustina;Gherardo Mazziotti;Ernesto Canalis.
Endocrine Reviews (2008)
Insulin-like growth factor I has independent effects on bone matrix formation and cell replication.
Janet M. Hock;Michael Centrella;Ernesto Canalis.
Growth factors and the regulation of bone remodeling.
E Canalis;T McCarthy;M Centrella.
Journal of Clinical Investigation (1988)
Insulin-like growth factor I mediates selective anabolic effects of parathyroid hormone in bone cultures.
E Canalis;M Centrella;W Burch;T L McCarthy.
Journal of Clinical Investigation (1989)
Mechanisms of Anabolic Therapies for Osteoporosis
Ernesto Canalis;Andrea Giustina;John P. Bilezikian.
The New England Journal of Medicine (2007)
The divalent strontium salt S12911 enhances bone cell replication and bone formation in vitro.
E. Canalis;M. Hott;P. Deloffre;Y. Tsouderos.
Clinical review 83: Mechanisms of glucocorticoid action in bone: implications to glucocorticoid-induced osteoporosis.
The Journal of Clinical Endocrinology and Metabolism (1996)
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