D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Biology and Biochemistry D-index 44 Citations 9,998 123 World Ranking 13471 National Ranking 5734

Overview

What is he best known for?

The fields of study he is best known for:

  • Internal medicine
  • Cancer
  • Cytokine

M. Neale Weitzmann spends much of his time researching Endocrinology, Internal medicine, Bone resorption, Osteoclast and Bone remodeling. M. Neale Weitzmann is involved in the study of Endocrinology that focuses on Osteoporosis in particular. His work carried out in the field of Osteoporosis brings together such families of science as Acquired immune system, Inflammation, Immunology, Immune system and Estrogen.

His biological study spans a wide range of topics, including Osteoprotegerin, Osteoblast and Cell biology. M. Neale Weitzmann combines subjects such as RANKL and RANK Ligand with his study of Osteoclast. His research in Bone remodeling tackles topics such as Bone density which are related to areas like Bone mineral.

His most cited work include:

  • Estrogen deficiency and bone loss: an inflammatory tale (604 citations)
  • IFN-γ stimulates osteoclast formation and bone loss in vivo via antigen-driven T cell activation (326 citations)
  • B cells and T cells are critical for the preservation of bone homeostasis and attainment of peak bone mass in vivo. (323 citations)

What are the main themes of his work throughout his whole career to date?

His primary scientific interests are in Internal medicine, Endocrinology, Bone resorption, Bone remodeling and Osteoporosis. His Internal medicine study combines topics in areas such as Wnt signaling pathway, CD40 and Cell biology. His Endocrinology research includes themes of Cytokine, T cell, Osteoblast, Parathyroid hormone and Bone marrow.

M. Neale Weitzmann combines subjects such as Osteoclast, Peak bone mass, Osteopenia and RANKL with his study of Bone resorption. His research in Bone remodeling intersects with topics in Immune system, Metabolomics, Bone mineral, Bone density and Senile osteoporosis. M. Neale Weitzmann works mostly in the field of Osteoporosis, limiting it down to topics relating to Immunology and, in certain cases, Bone disease and Oncology.

He most often published in these fields:

  • Internal medicine (89.33%)
  • Endocrinology (80.00%)
  • Bone resorption (61.33%)

What were the highlights of his more recent work (between 2016-2021)?

  • Internal medicine (89.33%)
  • Endocrinology (80.00%)
  • Bone resorption (61.33%)

In recent papers he was focusing on the following fields of study:

The scientist’s investigation covers issues in Internal medicine, Endocrinology, Bone resorption, Osteoporosis and Bone remodeling. His study in Internal medicine is interdisciplinary in nature, drawing from both Protein kinase B and Oncology. His Endocrinology research is multidisciplinary, incorporating perspectives in T cell, Osteoclast, CD8, Parathyroid hormone and Bone marrow.

His studies deal with areas such as Tumor necrosis factor alpha, Osteoblast and Cell biology as well as T cell. His biological study spans a wide range of topics, including Cortical bone, Immunology and RANKL. His research integrates issues of Bone mineral and Metabolomics in his study of Bone remodeling.

Between 2016 and 2021, his most popular works were:

  • The Microbial Metabolite Butyrate Stimulates Bone Formation via T Regulatory Cell-Mediated Regulation of WNT10B Expression. (90 citations)
  • The Microbial Metabolite Butyrate Stimulates Bone Formation via T Regulatory Cell-Mediated Regulation of WNT10B Expression. (90 citations)
  • Bone and the Immune System. (53 citations)

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Estrogen deficiency and bone loss: an inflammatory tale

M. Neale Weitzmann;Roberto Pacifici.
Journal of Clinical Investigation (2006)

943 Citations

IFN-γ stimulates osteoclast formation and bone loss in vivo via antigen-driven T cell activation

Yuhao Gao;Francesco Grassi;Michaela Robbie Ryan;Masakazu Terauchi.
Journal of Clinical Investigation (2007)

457 Citations

B cells and T cells are critical for the preservation of bone homeostasis and attainment of peak bone mass in vivo.

Yan Li;Gianluca Toraldo;Aimin Li;Xiaoying Yang.
Blood (2007)

449 Citations

Estrogen Decreases Osteoclast Formation by Down-regulating Receptor Activator of NF-κB Ligand (RANKL)-induced JNK Activation

Sunil Srivastava;Gianluca Toraldo;M. Neale Weitzmann;Simone Cenci.
Journal of Biological Chemistry (2001)

433 Citations

Estrogen deficiency induces bone loss by increasing T cell proliferation and lifespan through IFN-γ-induced class II transactivator

Simone Cenci;Gianluca Toraldo;M. Neale Weitzmann;Cristiana Roggia.
Proceedings of the National Academy of Sciences of the United States of America (2003)

318 Citations

IL-7 induces bone loss in vivo by induction of receptor activator of nuclear factor κB ligand and tumor necrosis factor α from T cells

Gianluca Toraldo;Cristiana Roggia;Wei-Ping Qian;Roberto Pacifici.
Proceedings of the National Academy of Sciences of the United States of America (2003)

294 Citations

Sex steroid deficiency–associated bone loss is microbiota dependent and prevented by probiotics

Jau-Yi Li;Benoit Chassaing;Abdul Malik Tyagi;Chiara Vaccaro.
Journal of Clinical Investigation (2016)

259 Citations

The role of T lymphocytes in bone metabolism.

M. Neale Weitzmann;Roberto Pacifici.
Immunological Reviews (2005)

234 Citations

Endogenous TNFα Lowers Maximum Peak Bone Mass and Inhibits Osteoblastic Smad Activation Through NF‐κB

Yan Li;Aimin Li;Karen Strait;Hongying Zhang.
Journal of Bone and Mineral Research (2007)

225 Citations

T Lymphocytes Amplify the Anabolic Activity of Parathyroid Hormone Through Wnt10b Signaling

Masakazu Terauchi;Jau-Yi Li;Brahmchetna Bedi;Ki-Hyun Baek.
Cell Metabolism (2009)

206 Citations

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