D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Medicine D-index 72 Citations 16,979 282 World Ranking 16469 National Ranking 1493

Overview

What is he best known for?

The fields of study Moin A. Saleem is best known for:

  • Gene
  • Enzyme
  • Apoptosis

Moin A. Saleem integrates many fields in his works, including Kidney and Nephrotic syndrome. In his works, he performs multidisciplinary study on Nephrotic syndrome and Kidney. His study brings together the fields of Slit diaphragm and Proteinuria. His Slit diaphragm study often links to related topics such as Endocrinology. Endocrinology is closely attributed to Diabetic nephropathy in his work. He performs multidisciplinary study on Diabetic nephropathy and Proteinuria in his works. Moin A. Saleem merges many fields, such as Podocyte and Glomerulonephritis, in his writings. Moin A. Saleem performs integrative study on Glomerulonephritis and Podocyte. As part of his studies on Internal medicine, he often connects relevant areas like Disease.

His most cited work include:

  • A Conditionally Immortalized Human Podocyte Cell Line Demonstrating Nephrin and Podocin Expression (889 citations)
  • Circulating urokinase receptor as a cause of focal segmental glomerulosclerosis (710 citations)
  • Podocin, a raft-associated component of the glomerular slit diaphragm, interacts with CD2AP and nephrin (442 citations)

What are the main themes of his work throughout his whole career to date

Moin A. Saleem connects Kidney with Focal segmental glomerulosclerosis in his research. His study deals with a combination of Focal segmental glomerulosclerosis and Kidney. He regularly ties together related areas like Slit diaphragm in his Proteinuria studies. Slit diaphragm and Podocyte are commonly linked in his work. His research on Podocyte often connects related areas such as Endocrinology. His Endocrinology study typically links adjacent topics like Glomerulonephritis. Glomerulonephritis and Proteinuria are two areas of study in which Moin A. Saleem engages in interdisciplinary research. Internal medicine is closely attributed to Nephrotic syndrome in his study. His Nephrotic syndrome study frequently links to other fields, such as Internal medicine.

Moin A. Saleem most often published in these fields:

  • Kidney (79.07%)
  • Proteinuria (72.09%)
  • Podocyte (68.84%)

What were the highlights of his more recent work (between 2019-2022)?

  • Internal medicine (70.59%)
  • Kidney (58.82%)
  • Proteinuria (47.06%)

In recent works Moin A. Saleem was focusing on the following fields of study:

His Podocyte study is within the categories of Nephrin and Slit diaphragm. Moin A. Saleem regularly links together related areas like Disease in his Internal medicine studies. His work often combines Kidney and Glomerulonephritis studies. He integrates Glomerulonephritis and Kidney in his research. His work blends Proteinuria and Diabetic nephropathy studies together. His study on Diabetic nephropathy is mostly dedicated to connecting different topics, such as Endocrinology. His research on Endocrinology frequently connects to adjacent areas such as Renal function. Moin A. Saleem integrates Renal function with Podocyte in his study. While working in this field, he studies both Cell biology and Immunology.

Between 2019 and 2022, his most popular works were:

  • IPNA clinical practice recommendations for the diagnosis and management of children with steroid-resistant nephrotic syndrome (128 citations)
  • APOL1 Kidney-Risk Variants Induce Mitochondrial Fission (22 citations)
  • Hyperglycemia alters mitochondrial respiration efficiency and mitophagy in human podocytes (17 citations)

In his most recent research, the most cited works focused on:

  • Gene
  • Mitochondrion
  • Apoptosis

His study of Lipid bilayer brings together topics like Ion channel and Membrane. With his scientific publications, his incorporates both Membrane and Lipid bilayer. Ion channel and Inflammasome are the areas that his Receptor study falls under. His Inflammasome study frequently draws connections to adjacent fields such as Receptor. He undertakes interdisciplinary study in the fields of Cell biology and Biophysics through his research. His multidisciplinary approach integrates Biophysics and Cell biology in his work. Moin A. Saleem undertakes interdisciplinary study in the fields of Biochemistry and Cell through his research. His multidisciplinary approach integrates Cell and Gene in his work. Moin A. Saleem carries out multidisciplinary research, doing studies in Gene and Embryonic stem cell.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

A Conditionally Immortalized Human Podocyte Cell Line Demonstrating Nephrin and Podocin Expression

Moin A Saleem;Michael J O'Hare;Jochen Reiser;Richard J Coward.
Journal of The American Society of Nephrology (2002)

1063 Citations

Circulating urokinase receptor as a cause of focal segmental glomerulosclerosis

Changli Wei;Shafic El Hindi;Jing Li;Alessia Fornoni.
Nature Medicine (2011)

879 Citations

Podocin, a raft-associated component of the glomerular slit diaphragm, interacts with CD2AP and nephrin

Karin Schwarz;Matias Simons;Jochen Reiser;Moin A. Saleem.
Journal of Clinical Investigation (2001)

779 Citations

Insulin signaling to the glomerular podocyte is critical for normal kidney function

Gavin I Welsh;Lorna J Hale;Vera Eremina;Marie Jeansson.
Cell Metabolism (2010)

429 Citations

Carnosine as a protective factor in diabetic nephropathy: association with a leucine repeat of the carnosinase gene CNDP1.

Bart Janssen;Daniela Hohenadel;Paul Brinkkoetter;Verena Peters.
Diabetes (2005)

343 Citations

Molecular basis of the functional podocin–nephrin complex: mutations in the NPHS2 gene disrupt nephrin targeting to lipid raft microdomains

Tobias B. Huber;Matias Simons;Björn Hartleben;Leonie Sernetz.
Human Molecular Genetics (2003)

335 Citations

ADCK4 mutations promote steroid-resistant nephrotic syndrome through CoQ10 biosynthesis disruption

Shazia Ashraf;Heon Yung Gee;Stephanie Woerner;Stephanie Woerner;Letian X. Xie.
Journal of Clinical Investigation (2013)

323 Citations

The human glomerular podocyte is a novel target for insulin action.

Richard J M Coward;Gavin I Welsh;Jing Yang;Candida Tasman.
Diabetes (2005)

306 Citations

WT1 Mutants Reveal SRPK1 to Be a Downstream Angiogenesis Target by Altering VEGF Splicing

Elianna M. Amin;Sebastian Oltean;Jing Hua;Melissa V.R. Gammons.
Cancer Cell (2011)

244 Citations

Transgenic expression of human APOL1 risk variants in podocytes induces kidney disease in mice

Pazit Beckerman;Jing Bi-Karchin;Ae Seo Deok Park;Chengxiang Qiu.
Nature Medicine (2017)

233 Citations

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Best Scientists Citing Moin A. Saleem

Tobias B. Huber

Tobias B. Huber

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Alberto Ortiz

Autonomous University of Madrid

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Jeffrey B. Kopp

Jeffrey B. Kopp

National Institutes of Health

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Corinne Antignac

Corinne Antignac

Université Paris Cité

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Friedhelm Hildebrandt

Friedhelm Hildebrandt

Boston Children's Hospital

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Barry I. Freedman

Barry I. Freedman

Wake Forest University

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Thomas Benzing

Thomas Benzing

University of Cologne

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Lawrence B. Holzman

Lawrence B. Holzman

University of Pennsylvania

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Giuseppe Remuzzi

Giuseppe Remuzzi

Mario Negri Institute for Pharmacological Research

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Matthias Kretzler

Matthias Kretzler

University of Michigan–Ann Arbor

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Stuart J. Shankland

Stuart J. Shankland

University of Washington

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Marta Ruiz-Ortega

Autonomous University of Madrid

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Hermann Pavenstädt

University of Freiburg

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Zhihong Liu

Nanjing University

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Susan E. Quaggin

Susan E. Quaggin

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Franz Schaefer

Franz Schaefer

Heidelberg University

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