Matthias Kretzler focuses on Internal medicine, Endocrinology, Podocyte, Pathology and Kidney. His Internal medicine research includes elements of Gastroenterology and Gene expression. When carried out as part of a general Endocrinology research project, his work on Glomerulosclerosis, Diabetic nephropathy and Kidney disease is frequently linked to work in Thrombin, therefore connecting diverse disciplines of study.
His work deals with themes such as CXCR4, Glomerulonephritis and Laser capture microdissection, which intersect with Pathology. His Kidney research is multidisciplinary, relying on both Epithelium, In situ hybridization, Actin cytoskeleton, Immunology and Molecular biology. His Slit diaphragm study incorporates themes from Podocyte foot, Glomerular basement membrane and Cell biology.
His primary areas of investigation include Internal medicine, Kidney, Pathology, Endocrinology and Kidney disease. His study connects Diabetes mellitus and Internal medicine. His Kidney research is multidisciplinary, incorporating elements of Cancer research, Gene expression, Nephropathy, Immunology and Lupus nephritis.
His research integrates issues of Focal segmental glomerulosclerosis, Glomerulonephritis and Gene expression profiling in his study of Pathology. Matthias Kretzler focuses mostly in the field of Endocrinology, narrowing it down to matters related to Cell biology and, in some cases, CCL21. His Kidney disease study combines topics in areas such as Acute kidney injury, Urinary system and Disease.
Matthias Kretzler mainly focuses on Kidney, Internal medicine, Kidney disease, Diabetes mellitus and Renal function. His research in Kidney is mostly focused on Diabetic nephropathy. His Internal medicine study integrates concerns from other disciplines, such as Endocrinology and Oncology.
His Diabetes mellitus research focuses on Inflammation and how it connects with Fibrosis, Kidney metabolism and Knockout mouse. His Nephrotic syndrome study combines topics from a wide range of disciplines, such as Focal segmental glomerulosclerosis, Minimal change disease and Pathology. His Immunology research incorporates themes from Lupus nephritis and Cell.
Matthias Kretzler mainly investigates Kidney disease, Internal medicine, Diabetes mellitus, Kidney and Renal function. Matthias Kretzler has researched Kidney disease in several fields, including Precision medicine, MEDLINE, Disease, Acute kidney injury and Molecular classification. In his study, which falls under the umbrella issue of Acute kidney injury, Pathology is strongly linked to In situ hybridization.
The Internal medicine study combines topics in areas such as Human leukocyte antigen, Allele and Locus. Matthias Kretzler interconnects Inflammation and Podocyte in the investigation of issues within Diabetes mellitus. Matthias Kretzler studies Kidney, focusing on Diabetic nephropathy in particular.
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A reference panel of 64,976 haplotypes for genotype imputation
Shane McCarthy;Sayantan Das;Warren Kretzschmar;Olivier Delaneau.
Nature Genetics (2016)
Cell Biology of the Glomerular Podocyte
Hermann Pavenstädt;Wilhelm Kriz;Matthias Kretzler.
Physiological Reviews (2003)
Netting Neutrophils Induce Endothelial Damage, Infiltrate Tissues, and Expose Immunostimulatory Molecules in Systemic Lupus Erythematosus
Eneida Villanueva;Srilakshmi Yalavarthi;Celine C. Berthier;Jeffrey B. Hodgin.
Journal of Immunology (2011)
Hypoxia promotes fibrogenesis in vivo via HIF-1 stimulation of epithelial-to-mesenchymal transition
Debra F. Higgins;Kuniko Kimura;Wanja M. Bernhardt;Nikita Shrimanker.
Journal of Clinical Investigation (2007)
Mouse Models of Diabetic Nephropathy
Frank C. Brosius;Charles E. Alpers;Erwin P. Bottinger;Matthew D. Breyer.
Journal of The American Society of Nephrology (2009)
Global kidney health 2017 and beyond: a roadmap for closing gaps in care, research, and policy
Adeera Levin;Marcello Tonelli;Joseph Bonventre;Josef Coresh.
The Lancet (2017)
Induction of B7-1 in podocytes is associated with nephrotic syndrome
Jochen Reiser;Jochen Reiser;Gero Von Gersdorff;Martin Loos;Jun Oh.
Journal of Clinical Investigation (2004)
Modification of kidney barrier function by the urokinase receptor
Changli Wei;Clemens C. Möller;Mehmet M. Altintas;Jing Li.
Nature Medicine (2008)
The Glomerular Slit Diaphragm Is a Modified Adherens Junction
Jochen Reiser;Wilhelm Kriz;Matthias Kretzler;Peter Mundel.
Journal of The American Society of Nephrology (2000)
mTORC1 activation in podocytes is a critical step in the development of diabetic nephropathy in mice
Ken Inoki;Hiroyuki Mori;Junying Wang;Tsukasa Suzuki.
Journal of Clinical Investigation (2011)
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