What is he best known for?
The fields of study he is best known for:
- Gene
- Internal medicine
- Genetics
Tobias B. Huber mainly investigates Cell biology, Podocyte, Internal medicine, Endocrinology and Slit diaphragm.
His Cell biology study integrates concerns from other disciplines, such as Autophagy and Cellular differentiation.
His study in the fields of Synaptopodin under the domain of Podocyte overlaps with other disciplines such as Fusion protein.
He combines subjects such as Diabetes mellitus and Kinase with his study of Internal medicine.
His research integrates issues of mTORC1 and PI3K/AKT/mTOR pathway in his study of Endocrinology.
His study in Slit diaphragm is interdisciplinary in nature, drawing from both Podocyte foot, Podocin, Nephrin, Immunoglobulin domain and Molecular biology.
His most cited work include:
- Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition) (4170 citations)
- Guidelines for the use and interpretation of assays for monitoring autophagy (3242 citations)
- Multiorgan and Renal Tropism of SARS-CoV-2. (582 citations)
What are the main themes of his work throughout his whole career to date?
The scientist’s investigation covers issues in Cell biology, Podocyte, Internal medicine, Kidney and Endocrinology.
The various areas that Tobias B. Huber examines in his Cell biology study include Autophagy, Nephrin and Cytoskeleton.
Tobias B. Huber focuses mostly in the field of Podocyte, narrowing it down to topics relating to Glomerulosclerosis and, in certain cases, Focal segmental glomerulosclerosis.
His Kidney research includes themes of Cancer research, Immunology, Kidney disease and Pathology.
Endocrinology is closely attributed to mTORC1 in his study.
The concepts of his Slit diaphragm study are interwoven with issues in Podocyte foot and Podocin.
He most often published in these fields:
- Cell biology (50.15%)
- Podocyte (41.54%)
- Internal medicine (25.23%)
What were the highlights of his more recent work (between 2019-2021)?
- Cell biology (50.15%)
- Podocyte (41.54%)
- Kidney (24.00%)
In recent papers he was focusing on the following fields of study:
His primary areas of investigation include Cell biology, Podocyte, Kidney, Internal medicine and Severe acute respiratory syndrome coronavirus 2.
His Cell biology research integrates issues from Autophagy, Transgene, Gene knockdown, Receptor and Cell type.
When carried out as part of a general Autophagy research project, his work on Chaperone-mediated autophagy and Autolysosome is frequently linked to work in Set and Knowledge base, therefore connecting diverse disciplines of study.
His studies deal with areas such as Glomerulosclerosis, Immune system, Focal segmental glomerulosclerosis and Membranous nephropathy as well as Podocyte.
His Kidney research is multidisciplinary, relying on both Biomarker discovery and Pathology.
The Internal medicine study combines topics in areas such as Endocrinology and Epithelial sodium channel.
Between 2019 and 2021, his most popular works were:
- Multiorgan and Renal Tropism of SARS-CoV-2. (582 citations)
- SARS-CoV-2 renal tropism associates with acute kidney injury. (67 citations)
- Cellular and Molecular Probing of Intact Human Organs (49 citations)
In his most recent research, the most cited papers focused on:
- Gene
- Internal medicine
- Genetics
Tobias B. Huber mostly deals with Cell biology, Severe acute respiratory syndrome coronavirus 2, Kidney disease, Autophagy and Pandemic.
His Cell biology study combines topics from a wide range of disciplines, such as Human kidney, Transgene and Human brain.
Tobias B. Huber has researched Autophagy in several fields, including Tyrosine, Myeloid leukemia, Mutant and Phosphorylation.
His Internal medicine research is multidisciplinary, incorporating perspectives in Epithelial sodium channel and Sodium.
In his work, Podocyte is strongly intertwined with Genetically modified mouse, which is a subfield of Pathology.
His Podocyte study frequently intersects with other fields, such as PI3K/AKT/mTOR pathway.
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