1986 - Fellow of Alfred P. Sloan Foundation
Member of the Association of American Physicians
His scientific interests lie mostly in Internal medicine, Endocrinology, Podocyte, Pathology and Kidney disease. As a part of the same scientific study, he usually deals with the Internal medicine, concentrating on Cell cycle and frequently concerns with Protein kinase A and Mesangial cell. His Endocrinology study combines topics from a wide range of disciplines, such as Cyclin-dependent kinase and Cancer research.
His studies deal with areas such as Glomerulonephritis, Complement membrane attack complex and Pathogenesis as well as Podocyte. His work in Glomerulonephritis covers topics such as Immunology which are related to areas like Cell biology and Glomerular basement membrane. The various areas that Stuart J. Shankland examines in his Pathology study include Rapidly progressive glomerulonephritis and Osteopontin.
The scientist’s investigation covers issues in Podocyte, Internal medicine, Endocrinology, Cell biology and Pathology. His work carried out in the field of Podocyte brings together such families of science as Focal segmental glomerulosclerosis, Glomerulonephritis, Glomerulosclerosis, Immunology and Molecular biology. Stuart J. Shankland combines subjects such as Cell cycle and Cancer research with his study of Endocrinology.
Stuart J. Shankland has included themes like Cell type and Cell growth in his Cell cycle study. His research investigates the connection with Cell biology and areas like Cyclin A which intersect with concerns in Cyclin A2. His Pathology research incorporates elements of Extracellular matrix, Immune system and Regeneration.
Stuart J. Shankland spends much of his time researching Podocyte, Cell biology, Kidney, Internal medicine and Pathology. His research integrates issues of CD44, Progenitor cell, Focal segmental glomerulosclerosis, Molecular biology and Extracellular matrix in his study of Podocyte. His Cell biology study combines topics in areas such as Nephrin, Downregulation and upregulation and Podocin.
The study of Internal medicine is intertwined with the study of Endocrinology in a number of ways. His research in Endocrinology intersects with topics in Senescence and Cancer research. His study in Pathology is interdisciplinary in nature, drawing from both Glomerulosclerosis, Pericyte and Bowman Capsule.
Stuart J. Shankland mainly investigates Podocyte, Cell biology, Kidney, Nephrin and Podocin. Stuart J. Shankland interconnects Progenitor, Glomerulosclerosis, Immunology, Molecular biology and Cellular compartment in the investigation of issues within Podocyte. The Cell biology study combines topics in areas such as Tissue engineering, Cell, Membrane and Reabsorption.
He is investigating Kidney as part of his Internal medicine and Endocrinology and Kidney study. His Nephrin research includes elements of Glomerulus, Transdifferentiation, Cell Transdifferentiation, Transgene and Fate mapping. His Podocin research is multidisciplinary, relying on both Cell type, Mesangium and Adult stem cell.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
The podocyte's response to injury: Role in proteinuria and glomerulosclerosis
Kidney International (2006)
Hematopoietic Stem Cells Contribute to the Regeneration of Renal Tubules after Renal Ischemia-Reperfusion Injury in Mice
Fangming Lin;Kimberly Cordes;Linheng Li;Leroy Hood.
Journal of The American Society of Nephrology (2003)
Proteinuria in diabetic kidney disease : A mechanistic viewpoint
J.A. Jefferson;S.J. Shankland;R.H. Pichler.
Kidney International (2008)
Activation of a local tissue angiotensin system in podocytes by mechanical strain
Raghu V. Durvasula;Arndt T. Petermann;Arndt T. Petermann;Keiju Hiromura;Keiju Hiromura;Mary Blonski;Mary Blonski.
Kidney International (2004)
Podocytes in culture: past, present, and future.
S.J. Shankland;J.W. Pippin;J. Reiser;P. Mundel.
Kidney International (2007)
Expression of transforming growth factor-β1 during diabetic renal hypertrophy
Stuart J. Shankland;James W. Scholey;Hao Ly;Kerri Thai.
Kidney International (1994)
Obstructive uropathy in the mouse: role of osteopontin in interstitial fibrosis and apoptosis.
Vuddhidej Ophascharoensuk;Cecilia M. Giachelli;Cecilia M. Giachelli;Katherine Gordon;Katherine Gordon;Jeremy Hughes;Jeremy Hughes.
Kidney International (1999)
Induction of TRPC6 Channel in Acquired Forms of Proteinuric Kidney Disease
Clemens C. Möller;Changli Wei;Mehmet M. Altintas;Jing Li.
Journal of The American Society of Nephrology (2007)
Anti–microRNA-21 oligonucleotides prevent Alport nephropathy progression by stimulating metabolic pathways
Ivan G. Gomez;Deidre A. MacKenna;Bryce G. Johnson;Vivek Kaimal.
Journal of Clinical Investigation (2015)
Tubulointerstitial disease in aging: evidence for underlying peritubular capillary damage, a potential role for renal ischemia.
Susan E. Thomas;Sharon Anderson;Katherine L. Gordon;Terry T. Oyama.
Journal of The American Society of Nephrology (1998)
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