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Hermann Pavenstädt

Hermann Pavenstädt

D-Index & Metrics

Biology and Biochemistry

D-Index
78
Citations
33117
World Ranking
4451
National Ranking
328

Medicine

D-Index
78
Citations
33908
World Ranking
17821
National Ranking
933

Overview

Hermann Pavenstädt is affiliated with the University of Freiburg in Germany. Their research spans the fields of Medicine as well as Biochemistry, Genetics and Molecular Biology, with a particular focus on Nephrology, Molecular Biology, Epidemiology, Genetics, and Cell Biology.

The primary topics addressed in Pavenstädt's work include:

  • Renal Diseases and Glomerulopathies
  • Chronic Kidney Disease and Diabetes
  • Renal and related cancers
  • Hippo pathway signaling and YAP/TAZ
  • Renal Transplantation Outcomes and Treatments
  • Sepsis Diagnosis and Treatment
  • Trauma, Hemostasis, Coagulopathy, Resuscitation

Pavenstädt has published extensively in various scientific journals, frequently appearing in the following venues:

  • Journal of the American Society of Nephrology
  • Journal of Clinical Medicine
  • International Journal of Molecular Sciences
  • Kidney International
  • The FASEB Journal

Several recent papers showcase the scope of Pavenstädt's research contributions:

  • Identification of novel sublingual parameters to analyze and diagnose microvascular dysfunction in sepsis: the NOSTRADAMUS study, 2021, Critical Care
  • A Deregulated Stress Response Underlies Distinct INF2-Associated Disease Profiles, 2020, Journal of the American Society of Nephrology
  • Crumbs2 Is an Essential Slit Diaphragm Protein of the Renal Filtration Barrier, 2021, Journal of the American Society of Nephrology
  • The Hippo pathway component Wwc2 is a key regulator of embryonic development and angiogenesis in mice, 2021, Cell Death and Disease
  • Identification and validation of objective triggers for initiation of resuscitation management of acutely ill non-trauma patients: the INITIATE IRON MAN study, 2021, Scandinavian Journal of Trauma Resuscitation and Emergency Medicine

Frequent collaborators in Pavenstädt's scientific work include:

  • Thomas Weide
  • Philipp Kümpers
  • Alexandros Rovas
  • Michael P. Krahn
  • Annika Möller-Kerutt

Best Publications

  • Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

    Daniel J. Klionsky;Kotb Abdelmohsen;Akihisa Abe;Joynal Abedin

  • Cell Biology of the Glomerular Podocyte

    Hermann Pavenstädt;Wilhelm Kriz;Matthias Kretzler

  • Effect of Early vs Delayed Initiation of Renal Replacement Therapy on Mortality in Critically Ill Patients With Acute Kidney Injury: The ELAIN Randomized Clinical Trial

    Alexander Zarbock;John A. Kellum;Christoph Schmidt;Hugo Van Aken

  • Rearrangements of the cytoskeleton and cell contacts induce process formation during differentiation of conditionally immortalized mouse podocyte cell lines

    Peter Mundel;Jochen Reiser;Aimée Zúñiga Mejı́a Borja;Hermann Pavenstädt

  • Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

    Daniel J. Klionsky;Kotb Abdelmohsen;Akihisa Abe;Joynal Abedin

  • Autophagy influences glomerular disease susceptibility and maintains podocyte homeostasis in aging mice

    Björn Hartleben;Markus Gödel;Catherine Meyer-Schwesinger;Shuya Liu

  • Everolimus in patients with autosomal dominant polycystic kidney disease.

    Gerd Walz;Klemens Budde;Marwan Mannaa;Jens Nürnberger

  • Role of mTOR in podocyte function and diabetic nephropathy in humans and mice

    Markus Gödel;Björn Hartleben;Nadja Herbach;Shuya Liu

  • Cisplatin Nephrotoxicity Is Critically Mediated via the Human Organic Cation Transporter 2

    Giuliano Ciarimboli;Thomas Ludwig;Detlef Lang;Hermann Pavenstädt

  • Activation of Cardiac Fibroblast Growth Factor Receptor 4 Causes Left Ventricular Hypertrophy

    Alexander Grabner;Ansel P. Amaral;Karla Schramm;Saurav Singh

  • Insulin signaling to the glomerular podocyte is critical for normal kidney function

    Gavin I Welsh;Lorna J Hale;Vera Eremina;Marie Jeansson

  • Organic cation transporter 2 mediates cisplatin-induced oto- and nephrotoxicity and is a target for protective interventions.

    Giuliano Ciarimboli;Dirk Deuster;Arne Knief;Michael Sperling

  • Nephrin and CD2AP associate with phosphoinositide 3-OH kinase and stimulate AKT-dependent signaling.

    Tobias B. Huber;Björn Hartleben;Jeong Kim;Miriam Schmidts

  • Developmental signalling pathways in renal fibrosis: the roles of Notch, Wnt and Hedgehog

    Maria Edeling;Grace Ragi;Shizheng Huang;Hermann Pavenstädt

  • Podocin and MEC-2 bind cholesterol to regulate the activity of associated ion channels.

    Tobias B. Huber;Bernhard Schermer;Roman Ulrich Müller;Martin Höhne

  • Dilemmas in the Management of Atrial Fibrillation in Chronic Kidney Disease

    Holger Reinecke;Eva Brand;Rolf Mesters;Wolf-Rüdiger Schäbitz

  • FGF23 signaling impairs neutrophil recruitment and host defense during CKD

    Jan Rossaint;Jessica Oehmichen;Hugo Van Aken;Stefan Reuter

  • NEPH1 defines a novel family of podocin interacting proteins

    Lorenz Sellin;Tobias B. Huber;Peter Gerke;Ivo Quack

  • Erratum to: Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition) (Autophagy, 12, 1, 1-222, 10.1080/15548627.2015.1100356

    Daniel J. Klionsky;Kotb Abdelmohsen;Akihisa Abe;Joynal Abedin

  • The Soluble VEGF Receptor sFlt1 Contributes to Endothelial Dysfunction in CKD

    Giovana S. Di Marco;Stefan Reuter;Uta Hillebrand;Susanne Amler

Frequent Co-Authors

Eberhard Schlatter
Eberhard Schlatter University of Münster
Tobias B. Huber
Tobias B. Huber Universität Hamburg
Karl Kunzelmann
Karl Kunzelmann University of Regensburg
Christoph Wanner
Christoph Wanner University of Würzburg
Günter Breithardt
Günter Breithardt University of Münster
Hans Oberleithner
Hans Oberleithner University of Münster
Rainer Schreiber
Rainer Schreiber University of Regensburg
Thomas Benzing
Thomas Benzing University of Cologne
Moin A. Saleem
Moin A. Saleem University of Bristol
Florian Lang
Florian Lang University of Tübingen

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