The scientist’s investigation covers issues in Cell biology, Internal medicine, Endocrinology, Membrane protein and Cilium. His Cell biology research includes themes of Podocin, Slit diaphragm, Podocyte and Genetics. His Internal medicine study combines topics in areas such as Soluble fms-like tyrosine kinase-1 and Preeclampsia.
His research investigates the connection between Endocrinology and topics such as Wnt signaling pathway that intersect with problems in Zebrafish. His Membrane protein research includes elements of Transport protein, Mechanosensation, Ion channel, Signal transducing adaptor protein and Endoplasmic reticulum. His work deals with themes such as Ciliopathies, Microtubule, Nephronophthisis and Polycystic kidney disease, which intersect with Cilium.
Thomas Benzing mostly deals with Cell biology, Internal medicine, Podocyte, Kidney and Endocrinology. His Cell biology research incorporates elements of Slit diaphragm and Membrane protein. His research integrates issues of Nephrin, Biochemistry and Podocin in his study of Slit diaphragm.
His research in Podocyte intersects with topics in Glomerulosclerosis, Proteostasis, Renal glomerulus and Cytoskeleton. Thomas Benzing combines subjects such as Acute kidney injury, Transcriptome, Bioinformatics and Pathology with his study of Kidney. His Cilium study deals with Nephronophthisis intersecting with Cystic kidney.
Cell biology, Internal medicine, Kidney, Disease and Podocyte are his primary areas of study. Thomas Benzing is interested in Cilium, which is a branch of Cell biology. His Internal medicine research is multidisciplinary, relying on both Gastroenterology and Rituximab.
His Kidney research integrates issues from Acute kidney injury, Gene expression and Pathology. His work focuses on many connections between Disease and other disciplines, such as Older patients, that overlap with his field of interest in One year follow up. In his work, Cell type and DNA repair is strongly intertwined with Glomerulosclerosis, which is a subfield of Podocyte.
His primary scientific interests are in Cell biology, Disease, Internal medicine, Podocyte and Proteome. His Cell biology research incorporates themes from RNA, Autophagy and Interactome. His Interquartile range, Clinical endpoint and Retrospective cohort study study in the realm of Internal medicine connects with subjects such as Hazard ratio.
His work carried out in the field of Podocyte brings together such families of science as PI3K/AKT/mTOR pathway, Protein kinase B, Signal transduction, Enzyme replacement therapy and Fabry disease. His work investigates the relationship between Proteome and topics such as Transcriptome that intersect with problems in Slit diaphragm, Proteostasis, Proteomics, Glomerulosclerosis and Filamin. His studies deal with areas such as Translation, Messenger RNA, RNA-binding protein, RNA Stability and DNA repair as well as Cilium.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
A mammalian microRNA expression atlas based on small RNA library sequencing.
Pablo Landgraf;Mirabela Rusu;Robert Sheridan;Alain Sewer;Alain Sewer.
Cell (2007)
When cilia go bad: cilia defects and ciliopathies
Manfred Fliegauf;Thomas Benzing;Heymut Omran.
Nature Reviews Molecular Cell Biology (2007)
Inversin, the gene product mutated in nephronophthisis type II, functions as a molecular switch between Wnt signaling pathways
Matias Simons;Joachim Gloy;Athina Ganner;Axel Bullerkotte.
Nature Genetics (2005)
Mutations in INVS encoding inversin cause nephronophthisis type 2, linking renal cystic disease to the function of primary cilia and left-right axis determination.
Edgar A. Otto;Bernhard Schermer;Tomoko Obara;John F. O'Toole.
Nature Genetics (2003)
Nephrin and CD2AP associate with phosphoinositide 3-OH kinase and stimulate AKT-dependent signaling.
Tobias B. Huber;Björn Hartleben;Jeong Kim;Miriam Schmidts.
Molecular and Cellular Biology (2003)
Interaction with Podocin Facilitates Nephrin Signaling
Tobias B. Huber;Michael Köttgen;Birgit Schilling;Gerd Walz.
Journal of Biological Chemistry (2001)
Mutations in a novel gene, NPHP3, cause adolescent nephronophthisis, tapeto-retinal degeneration and hepatic fibrosis
Heike Olbrich;Manfred Fliegauf;Julia Hoefele;Andreas Kispert.
Nature Genetics (2003)
Exome Capture Reveals ZNF423 and CEP164 Mutations, Linking Renal Ciliopathies to DNA Damage Response Signaling
Moumita Chaki;Rannar Airik;Amiya K. Ghosh;Rachel H. Giles.
Cell (2012)
Pilot Study of Extracorporeal Removal of Soluble Fms-Like Tyrosine Kinase 1 in Preeclampsia
Ravi Thadhani;Tuelay Kisner;Henning Hagmann;Verena Bossung.
Circulation (2011)
Podocyte-Specific Deletion of Dicer Alters Cytoskeletal Dynamics and Causes Glomerular Disease
Scott J. Harvey;George Jarad;Jeanette Cunningham;Seth Goldberg.
Journal of The American Society of Nephrology (2008)
If you think any of the details on this page are incorrect, let us know.
We appreciate your kind effort to assist us to improve this page, it would be helpful providing us with as much detail as possible in the text box below:
University of Cologne
Universität Hamburg
University of Freiburg
Universität Hamburg
Cedars-Sinai Medical Center
Harvard University
University of Houston
University of Münster
German Cancer Research Center
German Sport University Cologne
IBM (United States)
Aalborg University
Google (United States)
Fudan University
Children's Hospital of Philadelphia
North Carolina State University
Nagoya University
Kurume University
University of Bonn
The Nature Conservancy
Leipzig University
University of Maryland, College Park
University of Siena
The University of Texas Southwestern Medical Center
German Cancer Research Center
Stockholm University