Immunology, Cell biology, Molecular biology, Transcription factor and Interleukin 21 are his primary areas of study. Michael J. Grusby works mostly in the field of Immunology, limiting it down to topics relating to Signal transduction and, in certain cases, STAT6 and Receptor. As a member of one scientific family, Michael J. Grusby mostly works in the field of Cell biology, focusing on CD40 and, on occasion, Antibody.
His study on Molecular biology also encompasses disciplines like
Michael J. Grusby mainly investigates Immunology, Molecular biology, Cell biology, T cell and Cytotoxic T cell. His Immunology study frequently draws connections to adjacent fields such as STAT4. As a part of the same scientific family, Michael J. Grusby mostly works in the field of Molecular biology, focusing on Regulation of gene expression and, on occasion, T helper cell.
His Cell biology study combines topics from a wide range of disciplines, such as Transcription factor, Cellular differentiation, Cell growth and CD40. Michael J. Grusby interconnects Lipase and Gene in the investigation of issues within Cytotoxic T cell. His Major histocompatibility complex study incorporates themes from Autoimmunity and CD8.
His primary scientific interests are in Immunology, Molecular biology, Cell biology, Transcription factor and Interleukin 21. His Immunology research incorporates elements of Fibrosis and Type 1 diabetes. His work deals with themes such as Protein tyrosine phosphatase, SOCS3, Protein inhibitor of activated STAT, Zinc finger and Transcription, which intersect with Molecular biology.
His study in Cell biology is interdisciplinary in nature, drawing from both Cellular differentiation, Cell division and Cell growth. His Transcription factor research incorporates themes from Psychological repression, Regulation of gene expression and Methylation. His studies examine the connections between Cytokine and genetics, as well as such issues in Receptor, with regards to Cell fate determination.
His primary areas of study are Immunology, Molecular biology, Interleukin 21, Cytokine and Cytotoxic T cell. His work focuses on many connections between Molecular biology and other disciplines, such as Transcription factor, that overlap with his field of interest in Protein subunit, Derepression, Gene expression and Psychological repression. His studies deal with areas such as Interleukin-21 receptor and Nod, Insulitis as well as Interleukin 21.
His Cytokine study integrates concerns from other disciplines, such as Receptor, Type 1 diabetes, NOD mice and Insulin. Cytotoxic T cell is closely attributed to Cell biology in his study. His work carried out in the field of Immune system brings together such families of science as STAT4 and STAT1.
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Stat6 Is Required for Mediating Responses to IL-4 and for the Development of Th2 Cells
Mark H Kaplan;Ulrike Schindler;Stephen T Smiley;Michael J Grusby.
Impaired IL-12 responses and enhanced development of Th2 cells in Stat4-deficient mice
Mark H. Kaplan;Ya-Lin Sun;Timothy Hoey;Michael J. Grusby.
Plasma cell differentiation requires the transcription factor XBP-1
Andreas M. Reimold;Neal N. Iwakoshi;John Manis;Prashanth Vallabhajosyula.
Spontaneous development of inflammatory bowel disease in T cell receptor mutant mice
Peter Mombaerts;Emiko Mizoguchi;Michael J. Grusby;Laurie H. Glimcher.
Essential role of NKT cells producing IL-4 and IL-13 in the development of allergen-induced airway hyperreactivity
Omid Akbari;Philippe Stock;Everett Meyer;Mitchell Kronenberg.
Nature Medicine (2003)
Depletion of CD4+ T cells in major histocompatibility complex class II-deficient mice.
Michael J. Grusby;Randall S. Johnson;Virginia E. Papaioannou;Laurie H. Glimcher.
The transcription factor NF-ATc is essential for cardiac valve formation
Ann M. Ranger;Michael J. Grusby;Martin R. Hodge;Martin R. Hodge;Ellen M. Gravallese.
Hyperproliferation and Dysregulation ofIL-4 Expression in NF-ATp-Deficient Mice
Martin R Hodge;Ann M Ranger;Fabienne Charles de la Brousse;Timothy Hoey.
An essential role in liver development for transcription factor XBP-1
Andreas M. Reimold;Amit Etkin;Isabelle Clauss;Andrew Perkins.
Genes & Development (2000)
IL-21 limits NK cell responses and promotes antigen-specific T cell activation: a mediator of the transition from innate to adaptive immunity.
Marion T Kasaian;Matthew J Whitters;Laura L Carter;Leslie D Lowe.
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