His main research concerns Immunology, Eosinophil, Molecular biology, Internal medicine and T cell. Cytokine, Allergic inflammation, Inflammation, CD8 and Interferon gamma are subfields of Immunology in which his conducts study. Hiroshi Nakajima has included themes like Infiltration, Interleukin 5, Eosinophilia, Antigen and Adoptive cell transfer in his Eosinophil study.
His Molecular biology research includes themes of IL-2 receptor, Janus kinase 3, Cellular differentiation and Signal transduction, Cytokine receptor. His research investigates the connection between Internal medicine and topics such as Endocrinology that intersect with problems in Interleukin 6 and Gout. In his research on the topic of T cell, Lymphocyte function-associated antigen 1, Eosinophil cationic protein, Cell adhesion molecule and Intercellular Adhesion Molecule-1 is strongly related with T lymphocyte.
The scientist’s investigation covers issues in Immunology, Internal medicine, Astrophysics, Molecular biology and Eosinophil. His study in Antigen, Cytokine, Allergic inflammation, Inflammation and Pathogenesis falls within the category of Immunology. His work in the fields of Internal medicine, such as Rheumatoid arthritis, intersects with other areas such as In patient.
His Astrophysics research is multidisciplinary, relying on both Electron, Astronomy and Microwave. His research on Molecular biology frequently connects to adjacent areas such as IL-2 receptor. Hiroshi Nakajima combines subjects such as T cell and Interleukin 5 with his study of Eosinophil.
Hiroshi Nakajima mainly focuses on Immunology, Internal medicine, Optics, Optoelectronics and Rheumatoid arthritis. His study in Pathogenesis, Asthma, Inflammation, Innate lymphoid cell and Allergic inflammation is carried out as part of his Immunology studies. His Innate lymphoid cell study deals with Lung intersecting with Airway and Cytokine.
His work carried out in the field of Internal medicine brings together such families of science as Gastroenterology and Cardiology. Hiroshi Nakajima interconnects Layer and Laser in the investigation of issues within Optoelectronics. His study explores the link between Rheumatoid arthritis and topics such as Physical therapy that cross with problems in Rheumatoid factor.
Hiroshi Nakajima mainly investigates Immunology, Cytokine, Pathogenesis, Asthma and Immune system. His Immunology study frequently draws connections between related disciplines such as Lung. Hiroshi Nakajima has researched Cytokine in several fields, including Proinflammatory cytokine, Molecular biology, Transcription factor and Gene knockdown.
His studies deal with areas such as STAT3 Transcription Factor, Downregulation and upregulation, IL-2 receptor and Transcriptome as well as Molecular biology. The study incorporates disciplines such as Mast cell, Mucus, Interleukin 21, CXCR5 and Gene silencing in addition to Pathogenesis. His research in House dust mite intersects with topics in Chemokine, Receptor and Eosinophil.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Mutation of Jak3 in a Patient with SCID: Essential Role of Jak3 in Lymphoid Development
Sarah M. Russell;Nahid Tayebi;Hiroshi Nakajima;Mary C. Riedy.
IL-23 and Th17 Cells Enhance Th2-Cell–mediated Eosinophilic Airway Inflammation in Mice
Hidefumi Wakashin;Koichi Hirose;Yuko Maezawa;Shin-ichiro Kagami.
American Journal of Respiratory and Critical Care Medicine (2008)
The Hard X-ray Telescope (HXT) for the SOLAR-A Mission
T. Kosugi;K. Makishima;T. Murakami;T. Sakao.
Solar Physics (1991)
CD4+ T-lymphocytes and interleukin-5 mediate antigen-induced eosinophil infiltration into the mouse trachea.
Hiroshi Nakajima;Itsuo Iwamoto;Sanae Tomoe;Ryutaro Matsumura.
The American review of respiratory disease (1992)
Interferon gamma regulates antigen-induced eosinophil recruitment into the mouse airways by inhibiting the infiltration of CD4+ T cells.
I Iwamoto;H Nakajima;H Endo;S Yoshida.
Journal of Experimental Medicine (1993)
Stat5b Is Essential for Natural Killer Cell–mediated Proliferation and Cytolytic Activity
Kazunori Imada;Eda T. Bloom;Hiroshi Nakajima;Judith A. Horvath-Arcidiacono.
Journal of Experimental Medicine (1998)
Role of vascular cell adhesion molecule 1/very late activation antigen 4 and intercellular adhesion molecule 1/lymphocyte function-associated antigen 1 interactions in antigen-induced eosinophil and T cell recruitment into the tissue.
H Nakajima;H Sano;T Nishimura;S Yoshida.
Journal of Experimental Medicine (1994)
Fermi observations of GRB 090510 : A short-hard gamma-ray burst with an additional, hard power-law component from 10 keV to GeV energies
Markus Ackermann;K. Asano;W. B. Atwood;Magnus Axelsson;Magnus Axelsson.
The Astrophysical Journal (2010)
An Indirect Effect of Stat5a in IL-2–Induced Proliferation: A Critical Role for Stat5a in IL-2–Mediated IL-2 Receptor α Chain Induction
Hiroshi Nakajima;Xiu Wen Liu;Anthony Wynshaw-Boris;Louis A. Rosenthal.
Polymorphisms in the methylenetetrahydrofolate reductase gene were associated with both the efficacy and the toxicity of methotrexate used for the treatment of rheumatoid arthritis, as evidenced by single locus and haplotype analyses.
Wako Urano;Atsuo Taniguchi;Hisashi Yamanaka;Eiichi Tanaka.
If you think any of the details on this page are incorrect, let us know.
We appreciate your kind effort to assist us to improve this page, it would be helpful providing us with as much detail as possible in the text box below: