D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Immunology D-index 44 Citations 6,819 105 World Ranking 3219 National Ranking 116

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Immune system
  • Cytokine

His main research concerns Immunology, Molecular biology, Eosinophil, T cell and Cytokine. His is involved in several facets of Immunology study, as is seen by his studies on Interleukin 5, Interleukin and Immunoglobulin E. His Molecular biology study incorporates themes from Interleukin 3, IL-2 receptor, Endocrinology, Cytokine receptor and Internal medicine.

His work deals with themes such as Eosinophilia, Antigen and Allergic inflammation, which intersect with Eosinophil. His T cell research is multidisciplinary, incorporating elements of Receptor and T lymphocyte. The Cytokine study combines topics in areas such as Immune system and Cell biology.

His most cited work include:

  • Increased interleukin‐17 production in patients with systemic sclerosis (361 citations)
  • Transient gene transfer and expression of Smad7 prevents bleomycin-induced lung fibrosis in mice (358 citations)
  • CD4+ T-lymphocytes and interleukin-5 mediate antigen-induced eosinophil infiltration into the mouse trachea. (327 citations)

What are the main themes of his work throughout his whole career to date?

His primary scientific interests are in Immunology, Eosinophil, Molecular biology, Antigen and T cell. His Eosinophil research is multidisciplinary, relying on both Infiltration, Eosinophilia, Inflammation, Granulocyte and In vivo. His Molecular biology research incorporates elements of Endocrinology, Cytotoxic T cell, Interleukin 21, IL-2 receptor and Internal medicine.

His work is dedicated to discovering how IL-2 receptor, Cellular differentiation are connected with Interleukin 4 and other disciplines. Itsuo Iwamoto combines subjects such as Antibody and Monoclonal antibody with his study of Antigen. His primary area of study in T cell is in the field of T-cell receptor.

He most often published in these fields:

  • Immunology (61.22%)
  • Eosinophil (23.98%)
  • Molecular biology (21.94%)

What were the highlights of his more recent work (between 2005-2019)?

  • Immunology (61.22%)
  • Eosinophil (23.98%)
  • Allergic inflammation (7.65%)

In recent papers he was focusing on the following fields of study:

Itsuo Iwamoto mainly focuses on Immunology, Eosinophil, Allergic inflammation, Molecular biology and Asthma. His research in the fields of Interleukin 5, Interleukin 4 and Inflammation overlaps with other disciplines such as Index. As a part of the same scientific study, Itsuo Iwamoto usually deals with the Inflammation, concentrating on STAT6 and frequently concerns with T cell, Interleukin 17 and Adoptive cell transfer.

His studies deal with areas such as Antigen, Pathology, Substance P, Granulocyte and Allergy as well as Eosinophil. His research in Allergic inflammation focuses on subjects like Cytokine, which are connected to Proinflammatory cytokine. In his research on the topic of Molecular biology, IL-2 receptor and CD40 is strongly related with Interleukin 21.

Between 2005 and 2019, his most popular works were:

  • IL-25 enhances allergic airway inflammation by amplifying a TH2 cell–dependent pathway in mice (185 citations)
  • c‐Maf activates the promoter and enhancer of the IL‐21 gene, and TGF‐β inhibits c‐Maf‐induced IL‐21 production in CD4+ T cells (99 citations)
  • T-bet inhibits both TH2 cell-mediated eosinophil recruitment and TH17 cell-mediated neutrophil recruitment into the airways. (95 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Immune system
  • Internal medicine

Itsuo Iwamoto spends much of his time researching Immunology, Allergic inflammation, Eosinophil, Molecular biology and Cytokine. His research links Case-control study with Immunology. The Allergic inflammation study which covers Interleukin 13 that intersects with Interleukin 31, Interleukin 5, Interleukin 33 and Interleukin 25.

His research in Molecular biology tackles topics such as Interleukin 21 which are related to areas like CD28, CD40 and IL-2 receptor. His Cytokine study combines topics from a wide range of disciplines, such as Inflammation, Allergy and Antigen. His Antigen research is multidisciplinary, incorporating perspectives in T cell, Interleukin 22, Proinflammatory cytokine, Thymic stromal lymphopoietin and Antibody.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Increased interleukin‐17 production in patients with systemic sclerosis

Kazuhiro Kurasawa;Koichi Hirose;Hideki Sano;Hideharu Endo.
Arthritis & Rheumatism (2000)

563 Citations

Transient gene transfer and expression of Smad7 prevents bleomycin-induced lung fibrosis in mice

Atsuhito Nakao;Makiko Fujii;Ryutaro Matsumura;Kotaro Kumano.
Journal of Clinical Investigation (1999)

520 Citations

CD4+ T-lymphocytes and interleukin-5 mediate antigen-induced eosinophil infiltration into the mouse trachea.

Hiroshi Nakajima;Itsuo Iwamoto;Sanae Tomoe;Ryutaro Matsumura.
The American review of respiratory disease (1992)

463 Citations

Selective reduction of T cells bearing invariant V alpha 24J alpha Q antigen receptor in patients with systemic sclerosis.

T Sumida;A Sakamoto;H Murata;Y Makino.
Journal of Experimental Medicine (1995)

454 Citations

Interferon gamma regulates antigen-induced eosinophil recruitment into the mouse airways by inhibiting the infiltration of CD4+ T cells.

I Iwamoto;H Nakajima;H Endo;S Yoshida.
Journal of Experimental Medicine (1993)

433 Citations

Role of vascular cell adhesion molecule 1/very late activation antigen 4 and intercellular adhesion molecule 1/lymphocyte function-associated antigen 1 interactions in antigen-induced eosinophil and T cell recruitment into the tissue.

H Nakajima;H Sano;T Nishimura;S Yoshida.
Journal of Experimental Medicine (1994)

430 Citations

Mast cells produce interleukin-25 upon FcεRI-mediated activation

Kei Ikeda;Hiroshi Nakajima;Kotaro Suzuki;Shin-ichiro Kagami.
Blood (2003)

306 Citations

IL-25 enhances allergic airway inflammation by amplifying a TH2 cell–dependent pathway in mice

Tomohiro Tamachi;Yuko Maezawa;Kei Ikeda;Shin-ichiro Kagami.
The Journal of Allergy and Clinical Immunology (2006)

276 Citations

MgcRacGAP Is Involved in Cytokinesis through Associating with Mitotic Spindle and Midbody

Koichi Hirose;Toshiyuki Kawashima;Itsuo Iwamoto;Tetsuya Nosaka.
Journal of Biological Chemistry (2001)

245 Citations

Blockade of transforming growth factor beta/Smad signaling in T cells by overexpression of Smad7 enhances antigen-induced airway inflammation and airway reactivity

Atsuhito Nakao;Satoshi Miike;Masahiko Hatano;Ko Okumura.
Journal of Experimental Medicine (2000)

206 Citations

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