His main research concerns Immunology, Molecular biology, Eosinophil, T cell and Cytokine. His is involved in several facets of Immunology study, as is seen by his studies on Interleukin 5, Interleukin and Immunoglobulin E. His Molecular biology study incorporates themes from Interleukin 3, IL-2 receptor, Endocrinology, Cytokine receptor and Internal medicine.
His work deals with themes such as Eosinophilia, Antigen and Allergic inflammation, which intersect with Eosinophil. His T cell research is multidisciplinary, incorporating elements of Receptor and T lymphocyte. The Cytokine study combines topics in areas such as Immune system and Cell biology.
His primary scientific interests are in Immunology, Eosinophil, Molecular biology, Antigen and T cell. His Eosinophil research is multidisciplinary, relying on both Infiltration, Eosinophilia, Inflammation, Granulocyte and In vivo. His Molecular biology research incorporates elements of Endocrinology, Cytotoxic T cell, Interleukin 21, IL-2 receptor and Internal medicine.
His work is dedicated to discovering how IL-2 receptor, Cellular differentiation are connected with Interleukin 4 and other disciplines. Itsuo Iwamoto combines subjects such as Antibody and Monoclonal antibody with his study of Antigen. His primary area of study in T cell is in the field of T-cell receptor.
Itsuo Iwamoto mainly focuses on Immunology, Eosinophil, Allergic inflammation, Molecular biology and Asthma. His research in the fields of Interleukin 5, Interleukin 4 and Inflammation overlaps with other disciplines such as Index. As a part of the same scientific study, Itsuo Iwamoto usually deals with the Inflammation, concentrating on STAT6 and frequently concerns with T cell, Interleukin 17 and Adoptive cell transfer.
His studies deal with areas such as Antigen, Pathology, Substance P, Granulocyte and Allergy as well as Eosinophil. His research in Allergic inflammation focuses on subjects like Cytokine, which are connected to Proinflammatory cytokine. In his research on the topic of Molecular biology, IL-2 receptor and CD40 is strongly related with Interleukin 21.
Itsuo Iwamoto spends much of his time researching Immunology, Allergic inflammation, Eosinophil, Molecular biology and Cytokine. His research links Case-control study with Immunology. The Allergic inflammation study which covers Interleukin 13 that intersects with Interleukin 31, Interleukin 5, Interleukin 33 and Interleukin 25.
His research in Molecular biology tackles topics such as Interleukin 21 which are related to areas like CD28, CD40 and IL-2 receptor. His Cytokine study combines topics from a wide range of disciplines, such as Inflammation, Allergy and Antigen. His Antigen research is multidisciplinary, incorporating perspectives in T cell, Interleukin 22, Proinflammatory cytokine, Thymic stromal lymphopoietin and Antibody.
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Increased interleukin‐17 production in patients with systemic sclerosis
Kazuhiro Kurasawa;Koichi Hirose;Hideki Sano;Hideharu Endo.
Arthritis & Rheumatism (2000)
Transient gene transfer and expression of Smad7 prevents bleomycin-induced lung fibrosis in mice
Atsuhito Nakao;Makiko Fujii;Ryutaro Matsumura;Kotaro Kumano.
Journal of Clinical Investigation (1999)
CD4+ T-lymphocytes and interleukin-5 mediate antigen-induced eosinophil infiltration into the mouse trachea.
Hiroshi Nakajima;Itsuo Iwamoto;Sanae Tomoe;Ryutaro Matsumura.
The American review of respiratory disease (1992)
Selective reduction of T cells bearing invariant V alpha 24J alpha Q antigen receptor in patients with systemic sclerosis.
T Sumida;A Sakamoto;H Murata;Y Makino.
Journal of Experimental Medicine (1995)
Interferon gamma regulates antigen-induced eosinophil recruitment into the mouse airways by inhibiting the infiltration of CD4+ T cells.
I Iwamoto;H Nakajima;H Endo;S Yoshida.
Journal of Experimental Medicine (1993)
Role of vascular cell adhesion molecule 1/very late activation antigen 4 and intercellular adhesion molecule 1/lymphocyte function-associated antigen 1 interactions in antigen-induced eosinophil and T cell recruitment into the tissue.
H Nakajima;H Sano;T Nishimura;S Yoshida.
Journal of Experimental Medicine (1994)
Mast cells produce interleukin-25 upon FcεRI-mediated activation
Kei Ikeda;Hiroshi Nakajima;Kotaro Suzuki;Shin-ichiro Kagami.
IL-25 enhances allergic airway inflammation by amplifying a TH2 cell–dependent pathway in mice
Tomohiro Tamachi;Yuko Maezawa;Kei Ikeda;Shin-ichiro Kagami.
The Journal of Allergy and Clinical Immunology (2006)
MgcRacGAP Is Involved in Cytokinesis through Associating with Mitotic Spindle and Midbody
Koichi Hirose;Toshiyuki Kawashima;Itsuo Iwamoto;Tetsuya Nosaka.
Journal of Biological Chemistry (2001)
Blockade of transforming growth factor beta/Smad signaling in T cells by overexpression of Smad7 enhances antigen-induced airway inflammation and airway reactivity
Atsuhito Nakao;Satoshi Miike;Masahiko Hatano;Ko Okumura.
Journal of Experimental Medicine (2000)
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