D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Immunology D-index 71 Citations 17,014 176 World Ranking 1023 National Ranking 546
Medicine D-index 76 Citations 19,528 202 World Ranking 11573 National Ranking 6135

Research.com Recognitions

Awards & Achievements

Member of the Association of American Physicians

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Immune system
  • Cytokine

David H. Broide mostly deals with Immunology, Eosinophil, Inflammation, Cytokine and Immune system. His research combines Transforming growth factor beta and Immunology. His Eosinophil research incorporates themes from Allergic inflammation, Interleukin 5, Integrin, Immunoglobulin E and In vivo.

His Interleukin 5 study incorporates themes from Lung and Interferon gamma. David H. Broide combines subjects such as STAT6, Lung cancer, Signal transduction and Cell growth with his study of Inflammation. His Cytokine research focuses on Bronchoalveolar lavage and how it connects with Interleukin 6 and Respiratory disease.

His most cited work include:

  • Mutation of a new gene encoding a putative pyrin-like protein causes familial cold autoinflammatory syndrome and Muckle-Wells syndrome. (1222 citations)
  • Cytokines in symptomatic asthma airways. (688 citations)
  • Immunotherapy with a Ragweed–Toll-Like Receptor 9 Agonist Vaccine for Allergic Rhinitis (470 citations)

What are the main themes of his work throughout his whole career to date?

David H. Broide mainly focuses on Immunology, Eosinophil, Inflammation, Asthma and Pathology. Immunology is frequently linked to Airway in his study. His Eosinophil study combines topics in areas such as Cytokine, Interleukin 5, Molecular biology, Cell adhesion molecule and Allergic inflammation.

His work is dedicated to discovering how Allergic inflammation, Immunoglobulin E are connected with Mast cell and other disciplines. His study focuses on the intersection of Immune system and fields such as Antigen with connections in the field of Timothy-grass. His Fibrosis research is multidisciplinary, incorporating perspectives in Respiratory epithelium, Ovalbumin and Eosinophilic esophagitis.

He most often published in these fields:

  • Immunology (71.70%)
  • Eosinophil (25.28%)
  • Inflammation (18.49%)

What were the highlights of his more recent work (between 2014-2021)?

  • Immunology (71.70%)
  • Asthma (14.72%)
  • Innate lymphoid cell (8.68%)

In recent papers he was focusing on the following fields of study:

His main research concerns Immunology, Asthma, Innate lymphoid cell, Cell biology and Airway. Inflammation, Allergy, Antigen, Eosinophilia and Pathogenesis are subfields of Immunology in which his conducts study. In the field of Inflammation, his study on Allergic inflammation overlaps with subjects such as Mucus.

He interconnects T cell and Immune system in the investigation of issues within Allergy. His biological study spans a wide range of topics, including Chromosome, Gene, Follistatin-Related Proteins, Lung and Immunoglobulin E. His research investigates the connection with Prostaglandin D2 and areas like Leukotriene E4 which intersect with concerns in Eosinophil.

Between 2014 and 2021, his most popular works were:

  • Prostaglandin I2 Signaling and Inhibition of Group 2 Innate Lymphoid Cell Responses. (86 citations)
  • miR-23~27~24 clusters control effector T cell differentiation and function (72 citations)
  • Leukotriene C4 Potentiates IL-33-Induced Group 2 Innate Lymphoid Cell Activation and Lung Inflammation. (62 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Immune system
  • Cytokine

Immunology, Inflammation, Innate lymphoid cell, Internal medicine and Asthma are his primary areas of study. David H. Broide regularly links together related areas like Airway in his Immunology studies. His work on Allergic inflammation as part of general Inflammation study is frequently linked to Mucus, therefore connecting diverse disciplines of science.

His research in Innate lymphoid cell intersects with topics in Eosinophilia, Interleukin 33, Cell adhesion molecule, Prostaglandin D2 and VLA-4. His studies deal with areas such as Gastroenterology and Endocrinology as well as Internal medicine. His Asthma research includes elements of In vitro, Transgene, Gene knockdown, Lung and Transforming growth factor.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Mutation of a new gene encoding a putative pyrin-like protein causes familial cold autoinflammatory syndrome and Muckle-Wells syndrome.

Hal M. Hoffman;James L. Mueller;David H. Broide;Alan A. Wanderer.
Nature Genetics (2001)

1593 Citations

Cytokines in symptomatic asthma airways.

David H. Broide;Martin Lotz;Anthony J. Cuomo;David A. Coburn.
The Journal of Allergy and Clinical Immunology (1992)

890 Citations

Immunotherapy with a Ragweed–Toll-Like Receptor 9 Agonist Vaccine for Allergic Rhinitis

Peter S. Creticos;John T. Schroeder;Robert G. Hamilton;Susan L. Balcer-Whaley.
The New England Journal of Medicine (2006)

612 Citations

Cutting Edge: Activation of Toll-Like Receptor 2 Induces a Th2 Immune Response and Promotes Experimental Asthma

Vanessa Redecke;Hans Häcker;Sandip K. Datta;Agnes Fermin.
Journal of Immunology (2004)

557 Citations

Immunostimulatory DNA Sequences Inhibit IL-5, Eosinophilic Inflammation, and Airway Hyperresponsiveness in Mice

D. Broide;J. Schwarze;H. Tighe;T. Gifford.
Journal of Immunology (1998)

543 Citations

IL-5 links adaptive and natural immunity specific for epitopes of oxidized LDL and protects from atherosclerosis

Christoph J. Binder;Karsten Hartvigsen;Mi-Kyung Chang;Marina Miller.
Journal of Clinical Investigation (2004)

518 Citations

Inhibition of airway remodeling in IL-5-deficient mice.

Jae Youn Cho;Marina Miller;Kwang Je Baek;Ji Won Han.
Journal of Clinical Investigation (2004)

446 Citations

Esophageal remodeling in pediatric eosinophilic esophagitis

Seema S. Aceves;Robert O. Newbury;Ranjan Dohil;John F. Bastian.
The Journal of Allergy and Clinical Immunology (2007)

432 Citations

Tobacco Smoke Promotes Lung Tumorigenesis by Triggering IKKβ- and JNK1-Dependent Inflammation

Hiroyuki Takahashi;Hisanobu Ogata;Reiko Nishigaki;David H. Broide.
Cancer Cell (2010)

421 Citations

Inhibition of experimental asthma by indoleamine 2,3-dioxygenase

Tomoko Hayashi;Lucinda Beck;Cyprian Rossetto;Xing Gong.
Journal of Clinical Investigation (2004)

415 Citations

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