His primary areas of study are Endocrinology, Internal medicine, Receptor, Insulin receptor and Molecular biology. In the subject of general Endocrinology, his work in Gonadotropin-releasing hormone, Luteinizing hormone, Hypogonadotropic hypogonadism and Gonadotropic cell is often linked to Amenorrhea, thereby combining diverse domains of study. His Internal medicine study frequently links to adjacent areas such as Regulation of gene expression.
The Receptor study combines topics in areas such as DNA-binding protein, Signal transduction, Insulin receptor substrate and Fungal protein. His Insulin receptor study results in a more complete grasp of Insulin. His Molecular biology research is multidisciplinary, incorporating elements of MBNL1, Transcription factor, CELF1 Protein and Exon.
Nicholas J. G. Webster mostly deals with Internal medicine, Endocrinology, Insulin receptor, Insulin and Cell biology. In general Internal medicine, his work in Insulin resistance, Estrous cycle and Calorie restriction is often linked to Chromogranin A linking many areas of study. His study in Endocrinology is interdisciplinary in nature, drawing from both Receptor and Signal transduction.
His Insulin receptor study combines topics in areas such as Molecular biology, RNA splicing and Alternative splicing, Exon. Nicholas J. G. Webster combines subjects such as Glycogen, 3T3-L1 and Breast cancer with his study of Insulin. His biological study spans a wide range of topics, including IRF4, Phenotype and Biochemistry.
Nicholas J. G. Webster mainly focuses on Endocrinology, Internal medicine, Carbohydrate metabolism, Cell biology and Chromogranin A. His study in Endocrinology concentrates on Insulin, Insulin resistance, Calorie restriction, Stimulation and Adrenal medulla. He interconnects Cancer and Breast cancer in the investigation of issues within Insulin.
In his articles, Nicholas J. G. Webster combines various disciplines, including Internal medicine and Tyrosine hydroxylase. His Carbohydrate metabolism research incorporates elements of Estrous cycle and Impaired glucose tolerance. His biological study deals with issues like IRF4, which deal with fields such as Pattern recognition receptor and Signal transduction.
His primary areas of investigation include Internal medicine, Endocrinology, Carbohydrate metabolism, Chromogranin A and Inflammation. His work on Mutant expands to the thematically related Internal medicine. He has researched Carbohydrate metabolism in several fields, including Calorie restriction, Ovulation, Estrous cycle, Sirtuin 1 and Kisspeptin.
His research integrates issues of Circadian rhythm, Peripheral, Impaired glucose tolerance, Reproductive function and Metabolism in his study of Estrous cycle. His Chromogranin A studies intersect with other subjects such as Cardioprotection, Infiltration, Insulin, Insulin resistance and Type 2 diabetes. Nicholas J. G. Webster interconnects Gene expression, Secretion, Downregulation and upregulation, Monocyte and Mediator in the investigation of issues within Inflammation.
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The human estrogen receptor has two independent nonacidic transcriptional activation functions.
Laszio Tora;John White;Christel Brou;Dlane Tasset.
The hormone-binding domains of the estrogen and glucocorticoid receptors contain an inducible transcription activation function
Nicholas J.G. Webster;Stephen Green;Jia Rui Jin;Pierre Chambon.
The Yeast UASG Is a Transcriptional Enhancer in Human HeLa Cells in the Presence of the GAL4 Trans-Activator
Nicholas Webster;Jia Rui Jin;Stephen Green;Melvyn Hollis.
Brain PPAR-γ promotes obesity and is required for the insulin–sensitizing effect of thiazolidinediones
Min Min Lu;David A. Sarruf;Saswata Talukdar;Shweta Sharma;Shweta Sharma.
Nature Medicine (2011)
GnRH Activates ERK1/2 Leading to the Induction of c-fos and LHβ Protein Expression in LβT2 Cells
Fujun Liu;Darrell A. Austin;Pamela L. Mellon;Jerrold M. Olefsky.
Molecular Endocrinology (2002)
GnRH pulsatility, the pituitary response and reproductive dysfunction.
Rie Tsutsumi;Rie Tsutsumi;Rie Tsutsumi;Nicholas J.G. Webster;Nicholas J.G. Webster.
Endocrine Journal (2009)
Involvement of Both Gq/11 and Gs Proteins in Gonadotropin-releasing Hormone Receptor-mediated Signaling in LβT2 Cells *
Fujun Liu;Isao Usui;Lui Guojing Evans;Darrell A. Austin.
Journal of Biological Chemistry (2002)
Elevated Insulin-like Growth Factor I Receptor Autophosphorylation and Kinase Activity in Human Breast Cancer
J. L. Resnik;D. B. Reichart;Kim Huey;N. J. G. Webster.
Cancer Research (1998)
Curcumin disrupts the Mammalian target of rapamycin-raptor complex
Christopher S. Beevers;Long Chen;Lei Liu;Yan Luo.
Cancer Research (2009)
Repression of the insulin receptor promoter by the tumor suppressor gene product p53: a possible mechanism for receptor overexpression in breast cancer.
N. J. G. Webster;J. L. Resnik;D. B. Reichart;B. Strauss.
Cancer Research (1996)
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