His scientific interests lie mostly in Immunology, Lupus erythematosus, Internal medicine, Systemic lupus erythematosus and Autoimmune disease. Many of his studies involve connections with topics such as Gene expression and Immunology. The concepts of his Lupus erythematosus study are interwoven with issues in T cell, Immune system, Systemic disease, Disease and Autoimmunity.
His studies deal with areas such as Surgery and Pathology as well as Internal medicine. His study in Systemic lupus erythematosus is interdisciplinary in nature, drawing from both Microarray, Regulation of gene expression and Molecular biology. His Autoimmune disease research focuses on Interferon regulatory factors and how it relates to Haplotype and Cancer research.
His primary areas of study are Immunology, Internal medicine, Systemic lupus erythematosus, Interferon and Molecular biology. His studies in Immune system, Autoimmune disease, Autoimmunity, Lupus erythematosus and Autoantibody are all subfields of Immunology research. His biological study spans a wide range of topics, including Endocrinology and Cardiology.
Mary K. Crow frequently studies issues relating to Alpha interferon and Systemic lupus erythematosus. His Interferon study incorporates themes from Lupus nephritis, Innate immune system, Signal transduction and Cytokine. As part of one scientific family, Mary K. Crow deals mainly with the area of Molecular biology, narrowing it down to issues related to the T cell, and often Cytotoxic T cell, B cell, CD40 and Antigen.
The scientist’s investigation covers issues in Internal medicine, Immunology, Rheumatology, Systemic lupus erythematosus and Interferon. His studies deal with areas such as MEDLINE and Outcomes research as well as Internal medicine. His study in Immunology concentrates on Autoimmune disease, Immune system, Autoimmunity, Lymphoma and Cytokine.
Mary K. Crow combines subjects such as Lupus erythematosus, Rheumatism and Family medicine with his study of Rheumatology. The study incorporates disciplines such as Acquired immune system, Lupus nephritis, Exact test and Pathogenesis in addition to Systemic lupus erythematosus. His Interferon research is multidisciplinary, relying on both Autoantibody, Innate immune system, Immunity and Host defence.
Mary K. Crow mainly investigates Immunology, Rheumatology, Internal medicine, Interferon and Systemic lupus erythematosus. His research in Autoimmune disease, Immune system, Immune tolerance, Immunity and Host defence are components of Immunology. He focuses mostly in the field of Rheumatology, narrowing it down to matters related to MEDLINE and, in some cases, Cohort study and Lupus erythematosus.
His Internal medicine research integrates issues from Translational science and Family medicine. The various areas that Mary K. Crow examines in his Interferon study include Epigenomics, Signal transduction, Innate immune system, Autoimmunity and Antigen presentation. Mary K. Crow interconnects Pathogenesis, Inflammation, Scleroderma, Plasmacytoid dendritic cell and Fibrosis in the investigation of issues within Systemic lupus erythematosus.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Prevalence and correlates of accelerated atherosclerosis in systemic lupus erythematosus
Mary J Roman;Beth-Ann Shanker;Adrienne Davis;Michael D Lockshin.
The New England Journal of Medicine (2003)
Toll-like receptor 9-dependent activation by DNA-containing immune complexes is mediated by HMGB1 and RAGE.
Jane Tian;Ana Maria Avalos;Su-Yau Mao;Bo Chen.
Nature Immunology (2007)
2019 European League Against Rheumatism/American College of Rheumatology Classification Criteria for Systemic Lupus Erythematosus
Martin Aringer;Karen Costenbader;David Daikh;Ralph Brinks.
Arthritis & Rheumatism (2019)
Systemic lupus erythematosus.
Arvind Kaul;Caroline Gordon;Mary K Crow;Zahi Touma.
Nature Reviews Disease Primers (2016)
Activation of the interferon‐α pathway identifies a subgroup of systemic lupus erythematosus patients with distinct serologic features and active disease
Kyriakos A. Kirou;Kyriakos A. Kirou;Christina Lee;Sandhya George;Kyriakos Louca.
Arthritis & Rheumatism (2005)
Identification of a central role for complement in osteoarthritis
Qian Wang;Andrew L Rozelle;Christin M Lepus;Carla R Scanzello.
Nature Medicine (2011)
High serum IFN-alpha activity is a heritable risk factor for systemic lupus erythematosus.
T B Niewold;J Hua;T J A Lehman;J B Harley.
Genes and Immunity (2007)
Type I Interferon in the Pathogenesis of Lupus
Mary K. Crow.
Journal of Immunology (2014)
Coordinate overexpression of interferon‐α–induced genes in systemic lupus erythematosus
Kyriakos A. Kirou;Christina Lee;Sandhya George;Kyriakos Louca.
Arthritis & Rheumatism (2004)
Increased expression of CD40 ligand on systemic lupus erythematosus lymphocytes.
M Koshy;D Berger;M K Crow.
Journal of Clinical Investigation (1996)
If you think any of the details on this page are incorrect, let us know.
We appreciate your kind effort to assist us to improve this page, it would be helpful providing us with as much detail as possible in the text box below: