Andras Perl mostly deals with Systemic lupus erythematosus, Immunology, Lupus erythematosus, T cell and Mitochondrion. His work carried out in the field of Systemic lupus erythematosus brings together such families of science as Sirolimus, Cross-sectional study, Bipolar disorder, Psychiatry and Pharmacology. Andras Perl has researched Immunology in several fields, including Mechanistic target of rapamycin, PI3K/AKT/mTOR pathway and Cytotoxic T cell.
His T cell research is multidisciplinary, incorporating perspectives in B cell, Internal medicine, Endocrinology and Cell biology. The study incorporates disciplines such as Hyperpolarization, Intracellular and Apoptosis, Programmed cell death in addition to Mitochondrion. His Programmed cell death research integrates issues from Sequestosome 1, Autophagosome and Physiology.
The scientist’s investigation covers issues in Immunology, Systemic lupus erythematosus, Molecular biology, Internal medicine and T cell. While the research belongs to areas of Immunology, Andras Perl spends his time largely on the problem of PI3K/AKT/mTOR pathway, intersecting his research to questions surrounding Cancer research and Proinflammatory cytokine. His work in Systemic lupus erythematosus covers topics such as Mitochondrion which are related to areas like Apoptosis, Oxidative stress, ATP synthase and Mitophagy.
His biological study spans a wide range of topics, including Concanavalin A, Transaldolase, Peripheral blood mononuclear cell, Cytotoxicity and Antibody. His Internal medicine study combines topics in areas such as Gastroenterology and Endocrinology. His T cell research is multidisciplinary, relying on both T lymphocyte and CD8.
His primary scientific interests are in Immunology, Systemic lupus erythematosus, Mechanistic target of rapamycin, PI3K/AKT/mTOR pathway and Internal medicine. His studies deal with areas such as Arthritis, T cell, CD8, Autoimmunity and Nephritis as well as Systemic lupus erythematosus. His study in PI3K/AKT/mTOR pathway is interdisciplinary in nature, drawing from both Proinflammatory cytokine, Cancer research, Autophagy and Pentose phosphate pathway.
His Autophagy study incorporates themes from Mutant, Computational biology and Programmed cell death. His research in Computational biology intersects with topics in MAP1LC3B, BECN1, Autophagosome and Physiology. The various areas that Andras Perl examines in his Internal medicine study include Gastroenterology, Lupus erythematosus and Endocrinology.
Immunology, Mechanistic target of rapamycin, Systemic lupus erythematosus, PI3K/AKT/mTOR pathway and Lupus erythematosus are his primary areas of study. His Immunology research is multidisciplinary, incorporating elements of Oxidative stress, Reactive oxygen species, Mitochondrion and Molecular biology. His Systemic lupus erythematosus research integrates issues from Sirolimus, T cell, Interleukin 4, Regulation of gene expression and B-cell receptor.
Andras Perl has included themes like Proinflammatory cytokine, Metabolic pathway, Immune system and Pentose phosphate pathway in his PI3K/AKT/mTOR pathway study. In his research, Depression and Endocrinology is intimately related to Internal medicine, which falls under the overarching field of Lupus erythematosus. Andras Perl has researched Autophagy in several fields, including Computational biology, Programmed cell death and Physiology.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)
Daniel J. Klionsky;Amal Kamal Abdel-Aziz;Sara Abdelfatah;Mahmoud Abdellatif.
Autophagy (2021)
Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)
Daniel J. Klionsky;Kotb Abdelmohsen;Akihisa Abe;Joynal Abedin.
Autophagy (2016)
Elevation of Mitochondrial Transmembrane Potential and Reactive Oxygen Intermediate Levels Are Early Events and Occur Independently from Activation of Caspases in Fas Signaling
Katalin Banki;Eliza Hutter;Nick J. Gonchoroff;Andras Perl.
Journal of Immunology (1999)
Mitochondrial hyperpolarization and ATP depletion in patients with systemic lupus erythematosus.
Peter Gergely;Craig Grossman;Brian Niland;Ferenc Puskas.
Arthritis & Rheumatism (2002)
Rapamycin reduces disease activity and normalizes T cell activation–induced calcium fluxing in patients with systemic lupus erythematosus
David Fernandez;Eduardo Bonilla;Naureen Mirza;Brian Niland.
Arthritis & Rheumatism (2006)
N‐acetylcysteine reduces disease activity by blocking mammalian target of rapamycin in T cells from systemic lupus erythematosus patients: A randomized, double‐blind, placebo‐controlled trial
Zhi-Wei Lai;Robert Hanczko;Eduardo Bonilla;Tiffany N. Caza.
Arthritis & Rheumatism (2012)
Oxidative stress in the pathology and treatment of systemic lupus erythematosus.
Andras Perl.
Nature Reviews Rheumatology (2013)
Erratum to: Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition) (Autophagy, 12, 1, 1-222, 10.1080/15548627.2015.1100356
Daniel J. Klionsky;Kotb Abdelmohsen;Akihisa Abe;Joynal Abedin.
Autophagy (2016)
Glutathione Levels and Sensitivity to Apoptosis Are Regulated by Changes in Transaldolase Expression
Katalin Banki;Eliza Hutter;Emanuela Colombo;Nick J. Gonchoroff.
Journal of Biological Chemistry (1996)
Mitochondrial hyperpolarization: a checkpoint of T-cell life, death and autoimmunity.
Andras Perl;Peter Gergely;Gyorgy Nagy;Agnes Koncz.
Trends in Immunology (2004)
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