Mark S. Cragg mostly deals with Cell biology, Monoclonal antibody, Immunology, Antibody and CD20. He has researched Cell biology in several fields, including Molecular biology and Apoptosis, Programmed cell death, Genetics. His study explores the link between Monoclonal antibody and topics such as Antigen that cross with problems in Cancer research.
His biological study spans a wide range of topics, including Cell surface receptor, Intracellular and In vivo. The various areas that Mark S. Cragg examines in his Antibody study include Receptor and Cytotoxicity. His CD20 research includes themes of Monoclonal, B cell, Rituximab, Chronic lymphocytic leukemia and Immunotherapy.
Mark S. Cragg mainly focuses on Immunology, Monoclonal antibody, Cancer research, Antibody and Cell biology. Rituximab, CD20, B cell, Monoclonal and Antibody-dependent cell-mediated cytotoxicity are among the areas of Immunology where he concentrates his study. The study incorporates disciplines such as T cell, Epitope, Antigen, In vivo and Immunotherapy in addition to Monoclonal antibody.
Mark S. Cragg studied Cancer research and B-cell receptor that intersect with Signal transduction. His Antibody research incorporates elements of Receptor, Immune system and Effector. His work deals with themes such as Autophagy, Cell, Molecular biology and Chromatin, which intersect with Cell biology.
His primary areas of investigation include Antibody, Cancer research, Monoclonal antibody, Immune system and Immunology. His research in Antibody intersects with topics in Inflammation, Pharmacology, In vivo and Cell biology. He interconnects Lymphoma, Rituximab, Cancer immunotherapy, Signal transduction and Chronic lymphocytic leukemia in the investigation of issues within Cancer research.
His research integrates issues of Autophagy, Bcr signaling and Computational biology in his study of Chronic lymphocytic leukemia. His studies deal with areas such as Epitope, Receptor, Effector and Immunotherapy as well as Monoclonal antibody. His work carried out in the field of Immunology brings together such families of science as Nod and Disease.
Mark S. Cragg mainly focuses on Cell biology, Cancer research, Immunotherapy, Antibody and Monoclonal antibody. His Cell biology research is multidisciplinary, incorporating perspectives in FCGR2B, T cell, CD8, Fc receptor and Cytotoxic T cell. The concepts of his Cancer research study are interwoven with issues in Apoptosis, PI3K/AKT/mTOR pathway, Downregulation and upregulation, Lymphoma and Kinase.
His Immunotherapy research is multidisciplinary, incorporating elements of Allergic response, Peanut allergy and Vaccination. His studies in Antibody integrate themes in fields like Immunohistochemistry, Antagonist and Pharmacology. His Monoclonal antibody research is multidisciplinary, relying on both Receptor and Peripheral blood mononuclear cell.
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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)
Daniel J. Klionsky;Kotb Abdelmohsen;Akihisa Abe;Joynal Abedin.
Autophagy (2016)
Characterization of new human CD20 monoclonal antibodies with potent cytolytic activity against non-Hodgkin lymphomas
Jessica L Teeling;Ruth R French;Mark S Cragg;Jeroen van den Brakel.
Blood (2004)
Complement-mediated lysis by anti-CD20 mAb correlates with segregation into lipid rafts
Mark S Cragg;Suzanne M Morgan;H T Claude Chan;B Paul Morgan.
Blood (2003)
Mechanisms of killing by anti-CD20 monoclonal antibodies.
Martin J. Glennie;Ruth R. French;Mark S. Cragg;Ronald P. Taylor.
Molecular Immunology (2007)
Antibody specificity controls in vivo effector mechanisms of anti-CD20 reagents.
Mark S. Cragg;Martin J. Glennie.
Blood (2004)
Coupled Proliferation and Apoptosis Maintain the Rapid Turnover of Microglia in the Adult Brain
Katharine Askew;Kaizhen Li;Adrian Olmos-Alonso;Fernando Garcia-Moreno.
Cell Reports (2017)
The biology of CD20 and its potential as a target for mAb therapy.
Mark S. Cragg;Claire A. Walshe;Andrey O. Ivanov;Martin J. Glennie.
Current directions in autoimmunity (2005)
Bim and Bad mediate imatinib-induced killing of Bcr/Abl+ leukemic cells, and resistance due to their loss is overcome by a BH3 mimetic
Junya Kuroda;Hamsa Puthalakath;Mark S. Cragg;Priscilla N. Kelly;Priscilla N. Kelly.
Proceedings of the National Academy of Sciences of the United States of America (2006)
Gefitinib-Induced Killing of NSCLC Cell Lines Expressing Mutant EGFR Requires BIM and Can Be Enhanced by BH3 Mimetics
Mark S Cragg;Junya Kuroda;Hamsa Puthalakath;David C. S Huang.
PLOS Medicine (2007)
Antigenic modulation limits the efficacy of anti-CD20 antibodies: implications for antibody selection
Stephen A. Beers;Ruth R. French;H. T. Claude Chan;Sean H. Lim.
Blood (2010)
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