D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Immunology D-index 67 Citations 28,059 157 World Ranking 1753 National Ranking 862

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Immune system
  • Cytokine

The scientist’s investigation covers issues in Immunology, Inflammation, Colitis, Cytokine and Pathogenesis. His biological study spans a wide range of topics, including Lamina propria, Interleukin 12 and Inflammatory bowel disease. His Inflammation study incorporates themes from Missense mutation, Natural killer T cell, Immunopathology, Crohn's disease and Interleukin.

Ivan J. Fuss has researched Colitis in several fields, including Muramyl dipeptide and TLR2. His Cytokine research focuses on subjects like Tumor necrosis factor alpha, which are linked to Interleukin 23. His study looks at the relationship between Pathogenesis and topics such as Ulcerative colitis, which overlap with Oxazolone and Immune system.

His most cited work include:

  • Antibodies to interleukin 12 abrogate established experimental colitis in mice. (1225 citations)
  • The immunology of mucosal models of inflammation. (1180 citations)
  • Disparate CD4+ lamina propria (LP) lymphokine secretion profiles in inflammatory bowel disease. Crohn's disease LP cells manifest increased secretion of IFN-gamma, whereas ulcerative colitis LP cells manifest increased secretion of IL-5. (1087 citations)

What are the main themes of his work throughout his whole career to date?

His scientific interests lie mostly in Immunology, Colitis, Inflammatory bowel disease, Inflammation and Ulcerative colitis. As part of his studies on Immunology, he often connects relevant areas like Lamina propria. His Colitis research incorporates elements of Oxazolone, Molecular biology, Interleukin, Interleukin 12 and FOXP3.

His Inflammatory bowel disease research is multidisciplinary, incorporating perspectives in Fibrosis and Cancer research. His work carried out in the field of Inflammation brings together such families of science as Tumor necrosis factor alpha, Intestinal mucosa, Antigen, NOD2 and Disease. Ivan J. Fuss combines subjects such as Crohn's disease, Gastrointestinal tract, Gastroenterology and Pathogenesis with his study of Ulcerative colitis.

He most often published in these fields:

  • Immunology (65.52%)
  • Colitis (29.89%)
  • Inflammatory bowel disease (20.11%)

What were the highlights of his more recent work (between 2015-2021)?

  • Immunology (65.52%)
  • Cancer research (12.07%)
  • Inflammasome (2.87%)

In recent papers he was focusing on the following fields of study:

His primary areas of investigation include Immunology, Cancer research, Inflammasome, Colitis and Immune system. His Immunology study combines topics in areas such as Enteropathy, Disease and Gene. His study in Disease is interdisciplinary in nature, drawing from both Interleukin 18 and Pathogenesis.

While the research belongs to areas of Cancer research, Ivan J. Fuss spends his time largely on the problem of LRRK2, intersecting his research to questions surrounding Susceptibility gene, Transgene, Lamina propria, Proinflammatory cytokine and Genetically modified mouse. His research in Inflammasome focuses on subjects like Inflammatory bowel disease, which are connected to Kinase, Pyrin domain and B cell. His study explores the link between Colitis and topics such as Signal transduction that cross with problems in NOD2 and NOD1.

Between 2015 and 2021, his most popular works were:

  • Akkermansia muciniphila mediates negative effects of IFNγ on glucose metabolism (110 citations)
  • BACH2 immunodeficiency illustrates an association between super-enhancers and haploinsufficiency (63 citations)
  • The Role of NLRP3 and IL-1β in the Pathogenesis of Inflammatory Bowel Disease. (49 citations)

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Antibodies to interleukin 12 abrogate established experimental colitis in mice.

Markus F. Neurath;Ivan Fuss;Brian L. Kelsall;Eckhard Stüber.
Journal of Experimental Medicine (1995)

2017 Citations

The immunology of mucosal models of inflammation.

Warren Strober;Ivan J. Fuss;Richard S. Blumberg.
Annual Review of Immunology (2002)

1917 Citations

Disparate CD4+ lamina propria (LP) lymphokine secretion profiles in inflammatory bowel disease. Crohn's disease LP cells manifest increased secretion of IFN-gamma, whereas ulcerative colitis LP cells manifest increased secretion of IL-5.

I J Fuss;M Neurath;M Neurath;M Boirivant;J S Klein.
Journal of Immunology (1996)

1765 Citations

The fundamental basis of inflammatory bowel disease

Warren Strober;Ivan Fuss;Peter Mannon.
Journal of Clinical Investigation (2007)

1725 Citations

Proinflammatory cytokines in the pathogenesis of inflammatory bowel diseases.

Warren Strober;Ivan J. Fuss.
Gastroenterology (2011)

1241 Citations

Interleukin-13 Is the Key Effector Th2 Cytokine in Ulcerative Colitis That Affects Epithelial Tight Junctions, Apoptosis, and Cell Restitution

Frank Heller;Peter Florian;Christian Bojarski;Jan Richter.
Gastroenterology (2005)

1217 Citations

Anti-interleukin-12 antibody for active Crohn's disease

Peter J. Mannon;Ivan J. Fuss;Lloyd Mayer;Charles O. Elson.
The New England Journal of Medicine (2004)

1095 Citations

Cutting edge: regulatory T cells induce CD4+CD25-Foxp3- T cells or are self-induced to become Th17 cells in the absence of exogenous TGF-beta.

LiLi Xu;Atsushi Kitani;Ivan Fuss;Warren Strober.
Journal of Immunology (2007)

1000 Citations

TGF-β1 Plays an Important Role in the Mechanism of CD4+CD25+ Regulatory T Cell Activity in Both Humans and Mice

Kazuhiko Nakamura;Kazuhiko Nakamura;Atsushi Kitani;Ivan Fuss;Aasta Pedersen.
Journal of Immunology (2004)

974 Citations

Nonclassical CD1d-restricted NK T cells that produce IL-13 characterize an atypical Th2 response in ulcerative colitis

Ivan J. Fuss;Frank Heller;Monica Boirivant;Francisco Leon.
Journal of Clinical Investigation (2004)

965 Citations

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