The scientist’s investigation covers issues in Immunology, Inflammation, Colitis, Cytokine and Pathogenesis. His biological study spans a wide range of topics, including Lamina propria, Interleukin 12 and Inflammatory bowel disease. His Inflammation study incorporates themes from Missense mutation, Natural killer T cell, Immunopathology, Crohn's disease and Interleukin.
Ivan J. Fuss has researched Colitis in several fields, including Muramyl dipeptide and TLR2. His Cytokine research focuses on subjects like Tumor necrosis factor alpha, which are linked to Interleukin 23. His study looks at the relationship between Pathogenesis and topics such as Ulcerative colitis, which overlap with Oxazolone and Immune system.
His scientific interests lie mostly in Immunology, Colitis, Inflammatory bowel disease, Inflammation and Ulcerative colitis. As part of his studies on Immunology, he often connects relevant areas like Lamina propria. His Colitis research incorporates elements of Oxazolone, Molecular biology, Interleukin, Interleukin 12 and FOXP3.
His Inflammatory bowel disease research is multidisciplinary, incorporating perspectives in Fibrosis and Cancer research. His work carried out in the field of Inflammation brings together such families of science as Tumor necrosis factor alpha, Intestinal mucosa, Antigen, NOD2 and Disease. Ivan J. Fuss combines subjects such as Crohn's disease, Gastrointestinal tract, Gastroenterology and Pathogenesis with his study of Ulcerative colitis.
His primary areas of investigation include Immunology, Cancer research, Inflammasome, Colitis and Immune system. His Immunology study combines topics in areas such as Enteropathy, Disease and Gene. His study in Disease is interdisciplinary in nature, drawing from both Interleukin 18 and Pathogenesis.
While the research belongs to areas of Cancer research, Ivan J. Fuss spends his time largely on the problem of LRRK2, intersecting his research to questions surrounding Susceptibility gene, Transgene, Lamina propria, Proinflammatory cytokine and Genetically modified mouse. His research in Inflammasome focuses on subjects like Inflammatory bowel disease, which are connected to Kinase, Pyrin domain and B cell. His study explores the link between Colitis and topics such as Signal transduction that cross with problems in NOD2 and NOD1.
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Antibodies to interleukin 12 abrogate established experimental colitis in mice.
Markus F. Neurath;Ivan Fuss;Brian L. Kelsall;Eckhard Stüber.
Journal of Experimental Medicine (1995)
The immunology of mucosal models of inflammation.
Warren Strober;Ivan J. Fuss;Richard S. Blumberg.
Annual Review of Immunology (2002)
Disparate CD4+ lamina propria (LP) lymphokine secretion profiles in inflammatory bowel disease. Crohn's disease LP cells manifest increased secretion of IFN-gamma, whereas ulcerative colitis LP cells manifest increased secretion of IL-5.
I J Fuss;M Neurath;M Neurath;M Boirivant;J S Klein.
Journal of Immunology (1996)
The fundamental basis of inflammatory bowel disease
Warren Strober;Ivan Fuss;Peter Mannon.
Journal of Clinical Investigation (2007)
Proinflammatory cytokines in the pathogenesis of inflammatory bowel diseases.
Warren Strober;Ivan J. Fuss.
Interleukin-13 Is the Key Effector Th2 Cytokine in Ulcerative Colitis That Affects Epithelial Tight Junctions, Apoptosis, and Cell Restitution
Frank Heller;Peter Florian;Christian Bojarski;Jan Richter.
Anti-interleukin-12 antibody for active Crohn's disease
Peter J. Mannon;Ivan J. Fuss;Lloyd Mayer;Charles O. Elson.
The New England Journal of Medicine (2004)
Cutting edge: regulatory T cells induce CD4+CD25-Foxp3- T cells or are self-induced to become Th17 cells in the absence of exogenous TGF-beta.
LiLi Xu;Atsushi Kitani;Ivan Fuss;Warren Strober.
Journal of Immunology (2007)
TGF-β1 Plays an Important Role in the Mechanism of CD4+CD25+ Regulatory T Cell Activity in Both Humans and Mice
Kazuhiko Nakamura;Kazuhiko Nakamura;Atsushi Kitani;Ivan Fuss;Aasta Pedersen.
Journal of Immunology (2004)
Nonclassical CD1d-restricted NK T cells that produce IL-13 characterize an atypical Th2 response in ulcerative colitis
Ivan J. Fuss;Frank Heller;Monica Boirivant;Francisco Leon.
Journal of Clinical Investigation (2004)
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