David M. Knipe mainly focuses on Virology, Herpes simplex virus, Molecular biology, Virus and Viral replication. Within one scientific family, David M. Knipe focuses on topics pertaining to Innate immune system under Virology, and may sometimes address concerns connected to Proinflammatory cytokine. The concepts of his Herpes simplex virus study are interwoven with issues in Plasmid and Recombinant DNA.
His work carried out in the field of Molecular biology brings together such families of science as Regulation of gene expression, Immediate early protein, Gene expression, ICP8 and Mutant. The subject of his Virus research is within the realm of Genetics. His study looks at the intersection of Viral replication and topics like Immunization with Nasal administration and Virulence.
His primary areas of study are Virology, Herpes simplex virus, Virus, Molecular biology and Viral replication. The Virology study combines topics in areas such as Immunology and Immune system. His work deals with themes such as Immediate early protein, Mutant, Gene, Cell biology and Innate immune system, which intersect with Herpes simplex virus.
His Cell biology study combines topics from a wide range of disciplines, such as Chromatin and Transcription. Virus is frequently linked to Immunogenicity in his study. His studies deal with areas such as Gene expression, ICP8, DNA, Cell nucleus and Single-stranded binding protein as well as Molecular biology.
His primary scientific interests are in Virology, Herpes simplex virus, Cell biology, Virus and Viral replication. His research in Virology tackles topics such as Genome which are related to areas like Peptide sequence. His research in Herpes simplex virus intersects with topics in Chromatin, Gene and Innate immune system.
His biological study spans a wide range of topics, including Death-associated protein 6, Heterochromatin, Gene silencing and Cell. His studies in Virus integrate themes in fields like genomic DNA, Ubiquitin ligase, Sequence analysis and IRF3. His Viral replication research integrates issues from Recombinant virus, Transcription and DNA, DNA replication.
His main research concerns Herpes simplex virus, Virology, Cell biology, Innate immune system and Molecular biology. His study in Herpes simplex virus is interdisciplinary in nature, drawing from both genomic DNA, TLR2, Transfection and IFI16. The Viral replication and Virus research he does as part of his general Virology study is frequently linked to other disciplines of science, such as Synonymous substitution, therefore creating a link between diverse domains of science.
David M. Knipe has researched Cell biology in several fields, including Chromatin, Genetics, Gene silencing and Gene. His Innate immune system research incorporates themes from Nuclear localization sequence and Cell type. His research integrates issues of Conserved sequence, Mutant protein, Mutant and Single-stranded binding protein in his study of Molecular biology.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Herpes simplex virus 1 interaction with Toll-like receptor 2 contributes to lethal encephalitis.
Evelyn A. Kurt-Jones;Melvin Chan;Shenghua Zhou;Jennifer P. Wang.
Proceedings of the National Academy of Sciences of the United States of America (2004)
An siRNA-based microbicide protects mice from lethal herpes simplex virus 2 infection
Deborah Palliser;Dipanjan Chowdhury;Qing Yin Wang;Sandra J. Lee.
Thymidine kinase-negative herpes simplex virus mutants establish latency in mouse trigeminal ganglia but do not reactivate
Donald M. Coen;Magdalena Kosz-Vnenchak;Jennie G. Jacobson;David A. Leib.
Proceedings of the National Academy of Sciences of the United States of America (1989)
Chromatin control of herpes simplex virus lytic and latent infection
David M. Knipe;Anna Cliffe.
Nature Reviews Microbiology (2008)
Functions of Cytoplasmic Fibers in Non-Muscle Cell Motility
Robert D. Goldman;David M. Knipe.
Cold Spring Harbor Symposia on Quantitative Biology (1973)
Immediate-early regulatory gene mutants define different stages in the establishment and reactivation of herpes simplex virus latency.
D A Leib;D M Coen;C L Bogard;K A Hicks.
Journal of Virology (1989)
Vaginal Submucosal Dendritic Cells, but Not Langerhans Cells, Induce Protective Th1 Responses to Herpes Simplex Virus-2
Xinyan Zhao;Eszter Deak;Kelly A. Soderberg;Melissa Linehan.
Journal of Experimental Medicine (2003)
A deletion mutant of the latency-associated transcript of herpes simplex virus type 1 reactivates from the latent state with reduced frequency
D A Leib;C L Bogard;M Kosz-Vnenchak;K A Hicks.
Journal of Virology (1989)
Nuclear IFI16 induction of IRF-3 signaling during herpesviral infection and degradation of IFI16 by the viral ICP0 protein
Megan H. Orzalli;Neal A. DeLuca;David M. Knipe.
Proceedings of the National Academy of Sciences of the United States of America (2012)
The intranuclear location of a herpes simplex virus DNA-binding protein is determined by the status of viral DNA replication
Margaret P. Quinlan;Lan Bo Chen;David M. Knipe.
If you think any of the details on this page are incorrect, let us know.
We appreciate your kind effort to assist us to improve this page, it would be helpful providing us with as much detail as possible in the text box below: